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Gingivitis

para>More frequent in this age group (due more to additive effects than to increased susceptibility) á
Pediatric Considerations

Mild cases common in children (most common form of pediatric periodontal disease) and usually require no specific interventions other than improved oral hygiene

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Pregnancy Considerations

  • Very common in pregnant women; hormonal effect

  • Self-limited

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EPIDEMIOLOGY


  • Predominant age: children, teenagers, and young adults. Predominant sex: slightly more males than female
  • Prevalence ~50% of children
  • ~90% of adolescents and adult population
  • ~30-75% of pregnant women

ETIOLOGY AND PATHOPHYSIOLOGY


Inflammation of gingiva. This can progress to deeper, destructive inflammation. If involving supporting bone, will be classified as periodontitis, not gingivitis. á
  • Usually noncontagious
  • Inadequate plaque removal
  • Blood dyscrasias (pregnancy)
  • Oral contraceptives
  • Allergic reactions
  • Nutritional deficiencies
  • Vasoconstriction (nicotine, methamphetamine)
  • Endocrine/hormonal variations
    • Pregnancy
    • Menses
    • Menarche
  • Chronic debilitating disease
  • Vincent disease
    • Synergistic infection with fusiform bacillus (Fusobacterium spp.) and spirochete (Borrelia vincentii)
  • Pathology
    • Acute or chronic inflammation
    • Hyperemic capillaries
    • Polymorphonuclear infiltration
    • Papillary projections in subepithelial tissue
    • Fibroblasts

Genetics
Possible genetic link (up to 30% of population); rare condition called hereditary gingival fibromatosis associated with hirsutism á

RISK FACTORS


  • Poor dental hygiene/plaque formation
  • Pregnancy
  • Uncontrolled diabetes mellitus
  • Malocclusion or dental crowding
  • Smoking
  • Mouth breathing
  • Xerostomia
  • Faulty dental restoration
  • HIV-positive; AIDS
  • Stress
  • Hospitalization (1)[A]
  • Vitamin C deficiency; coenzyme Q10 deficiency
  • Dental appliances (dentures, braces)
  • Eruption of primary or secondary teeth
  • Necrotizing ulcerative gingivitis
    • Stress
    • Lack of sleep
    • Malnutrition
    • Viral illness
    • Typically teens and young adults
  • Bronchial asthma and other respiratory diseases (2,3)[B]
  • Rheumatoid arthritis (4)[B]

GENERAL PREVENTION


  • Good oral hygiene
    • Adults
      • Regular twice-daily brushing with fluoride toothpaste and increased benefit of using circular oscillating electric brush rather than regular brush or sonic/vibration (5,6)[A]
      • Daily "high-quality"Ł flossing (studies show that flossing only helps when it is done correctly) (7)[A]
      • Chlorhexidine with oral hygiene better than other oral rinse agents (8,9)[A]
        • Use in acute phase sparingly (10)[B]
    • Pediatrics
      • Regular twice-daily brushing with fluoride toothpaste under parental supervision until full manual dexterity (~8 years of age)
      • Regular flossing if no spaces between teeth
  • Cleaning by a dentist or hygienist every 6 months or more frequently, if indicated
  • Mouth rinse with essential oils (menthol, thymol, eucalyptol; e.g., Listerine) combined with brushing (11)[B]
    • Caution: Long-term use of alcohol-based mouth rinse may be associated with an increased risk of oral cancer (12)[B].

COMMONLY ASSOCIATED CONDITIONS


  • Periodontitis
  • Glossitis
  • Pedunculated growths (pyogenic granulomata)

DIAGNOSIS


HISTORY


  • Gum erythema, swelling, and edema
  • Gums are tender when touched but otherwise painless.
  • Bleeding of gums when brushing, flossing, or eating
  • Inquire about HIV risk, pregnancy, nutritional deficiencies, diabetes, and other risk factors as indicated (see "Risk Factors"Ł).
  • Smoking history
  • Oral hygiene, dental visit history

PHYSICAL EXAM


  • Normal gums should appear pink, firm, stippled, and scalloped.
  • Gingivitis-marginal gum swelling and edema (usually painless, except to touch)
  • Gum erythema: bright red or red-purple appearance
  • Bleeding with manipulation of gums
  • Change of normal gum contours
  • Plaque (soft) and calculus (not easily removed)
  • Edema of interdental papillae
  • HIV gingivitis
    • Also called linear gingival erythema
    • Narrow band of bright red inflamed gum surrounding neck of tooth
    • Painful
    • Bleeds easily
    • Rapid destruction of gingival tissue and can progress to periodontitis with destruction of underlying support tissues (periodontal ligament, supporting alveolar bone)
  • Vincent disease
    • Ulcers
    • Fever
    • Malaise
    • Regional lymphadenopathy
    • Pain
    • Mouth odor

DIFFERENTIAL DIAGNOSIS


  • Periodontitis (deeper inflammation, causing destruction to connective tissue, ligaments, and alveolar bone)
  • Glossitis
  • Desquamative gingivitis (painful, persistent, usually middle-aged women)
  • Pericoronitis (gum flap traps food and plaque over partially erupted third molar), common in adolescence
  • Gingival ulcers (aphthous, herpetic, malignancy, TB, syphilis)
  • Specific forms of gingivitis: See "Description,"Ł including acute necrotizing ulcerative gingivitis (Vincent disease) and HIV gingivitis (linear gingival erythema).

DIAGNOSTIC TESTS & INTERPRETATION


Initial Tests (lab, imaging)
  • No tests usually needed
  • Possible smear or culture to identify causative agent (HIV gingivitis includes gram-negative anaerobes, enteric strains, and Candida)
  • Labs for contributing conditions (HIV, pregnancy, diabetes, nutritional deficiencies)

TREATMENT


GENERAL MEASURES


  • Stop any contributing medications.
  • Remove irritating factors (plaque, calculus, faulty dental restorations, or partial dentures).
  • Good oral hygiene (see "General Prevention"Ł)
  • Regular dental checkups (for scaling and polishing if plaque and/or tartar are present)
  • Smoking cessation
  • Warm saline rinses BID
  • Special care needs patients: use of tray-applied 10% carbamide peroxide gels (13)[C]

MEDICATION


First Line
  • Chlorhexidine rinses or varnishes may be used (14)[B].
  • Mouth rinses with essential oils (EOMW) may be equally effective to chlorhexidine for reduction of gingival inflammation (while EOMW is not as effective for plaque control) (15)[A].
  • Both chlorhexidine and EOMW rinses are as clinically effective as oral prophylaxis and oral hygiene instruction at 6-month recall (16)[B].
  • Antibiotics indicated only for acute necrotizing ulcerative gingivitis (Vincent disease)
  • Antibiotics
    • Penicillin V: pediatric dose, 25 to 50 mg/kg/day divided q6h; adult dose, 250 to 500 mg q6h, OR
    • Metronidazole: pediatric dose, 30 mg/kg/day PO/IV divided q6h; maximum 4 g/day; adult dose, 500 mg BID or TID for 10 days OR
    • Amoxicillin/clavulanic acid: pediatric dose, 30 mg/kg/day PO divided q12h; info: use 125 mg/ 31.25 mg/5 mL susp; adult dose, 875 mg/ 125 mg PO BID for 10 days
    • Erythromycin: pediatric dose 30 to 40 mg/kg/day divided q6h; adult dose, 250 mg q6h
    • Doxycycline: adult dose, 100 mg BID 1st day, then QD for 10 days
  • Topical corticosteroids
    • Triamcinolone 0.1% in Orabase (spray or ointment), applied locally TID, QID
      • Contraindications
        • Allergy to specific medication
  • Precautions
    • Erythromycin frequently causes GI issues.

Second Line
  • Acetaminophen or ibuprofen for any pain (rare)
  • Other antibiotics or antifungal rinses or systemic according to culture or smear
  • Decapinol oral rinse (surfactant that acts as a physical barrier, making it harder for bacteria to stick to tooth and mucosal surfaces) to reduce bacteria (not recommended for pregnant women or children <12 years); should be used in conjunction with other oral hygiene practices when those practices alone are not enough

ISSUES FOR REFERRAL


  • Dental referral for cleanings and further treatment, as needed
  • If gingivitis becomes periodontitis, deep root scaling, planing, and antibiotics may be indicated.

SURGERY/OTHER PROCEDURES


  • D ębridement for acute necrotizing gingivitis
  • Minor surgery may be necessary to correct tissue overgrowth for gingivitis caused by medicines.

COMPLEMENTARY & ALTERNATIVE MEDICINE


  • Bilberry: potentially helpful in reducing inflammation and stabilizing collagen tissue
  • Coenzyme Q10: topically, to restore coenzyme Q10 deficiency
  • Replace any other deficiencies (e.g., vitamin C).

ONGOING CARE


FOLLOW-UP RECOMMENDATIONS


  • Outpatient
  • No restrictions

Patient Monitoring
Until clear; dental follow-up for continued cleanings and secondary prevention á

DIET


  • Well-balanced diet that includes fruits, vegetables, vitamin C; avoid sugary snacks and drinks, which contribute to plaque formation.
  • Soft foods during flare, if significant inflammation/bleeding

PATIENT EDUCATION


  • Good oral hygiene, including twice-daily brushing with circular oscillating electric brush, fluoridated toothpaste, and daily flossing; regular dental visits
  • Printable and viewable patient information available under "gum diseases"Ł from the American Dental Association at http://www.mouthhealthy.org/en/ and the American Academy of Periodontology under "patient resources"Ł at http://www.perio.org/

PROGNOSIS


  • Usual course: acute, relapsing, intermittent; chronic
  • Prognosis: generally favorable, responds well to appropriate treatment
  • Left untreated, may progress to periodontitis (controversial), which is a major cause of tooth loss

COMPLICATIONS


Severe periodontal disease (which is associated with heart disease, diabetes, and preterm birth) á

REFERENCES


11 Terezakis áE, Needleman áI, Kumar áN, et al. The impact of hospitalization on oral health: a systematic review. J Clin Periodontol.  2011;38(7):628-636.22 Stensson áM, Wendt áLK, Koch áG, et al. Oral health in pre-school children with asthma-followed from 3 to 6 years. Int J Paediatr Dent.  2010;20(3):165-172.33 Widmer áRP. Oral health of children with respiratory diseases. Paediatr Respir Rev.  2010;11(4):226-232.44 Keles áZP, Keles áGC, Avci áB, et al. Analysis of YKL-40 acute-phase protein and interleukin-6 levels in periodontal disease. J Periodontol.  2014;85(9):1240-1246.55 Yaacob áM, Worthington áHV, Deacon áSA, et al. Powered versus manual toothbrushing for oral health. Cochrane Database Syst Rev.  2014;(6):CD002281.66 Klukowska áM, Grender áJM, Conde áE, et al. A 12-week clinical comparison of an oscillating-rotating power brush versus a marketed sonic brush with self-adjusting technology in reducing plaque and gingivitis. J Clin Dent.  2013;24(2):55-61.77 Sambunjak áD, Nickerson áJW, Poklepovic áT, et al. Flossing for the management of periodontal diseases and dental caries in adults. Cochrane Database Syst Rev.  2011;(12):CD008829.88 Van Strydonck áDA, Slot áDE, Van der Velden áU, et al. Effect of a chlorhexidine mouthrinse on plaque, gingival inflammation and staining in gingivitis patients: a systematic review. J Clin Periodontol.  2012;39(11):1042-1055.99 Babu áJP, Garcia-Godoy áF. In vitro comparison of commercial oral rinses on bacterial adhesion and their detachment from biofilm formed on hydroxyapatite disks. Oral Health Prev Dent.  2014;12(4):365-371.1010 Eliot áMN, Michaud áDS, Langevin áSM, et al. Periodontal disease and mouthwash use are risk factors for head and neck squamous cell carcinoma. Cancer Causes Control.  2013;24(7):1315-1322.1111 Cortelli áSC, Cortelli áJR, Shang áH, et al. Gingival health benefits of essential-oil and cetylpyridinium chloride mouthrinses: a 6-month randomized clinical study. Am J Dent.  2014 Jun;27(3):119-126.1212 McCullough áM, Farah áCS. The role of alcohol in oral carcinogenesis with particular reference to alcohol-containing mouthwashes. Aust Dent J.  2008;53(4):302-305.1313 Lazarchik áDA, Haywood áVB. Use of tray-applied 10 percent carbamide peroxide gels for improving oral health in patients with special-care needs. J Am Dent Assoc.  2010;141(6):639-646.1414 Puig Silla áM, Montiel Company áJM, Almerich Silla áJM. Use of chlorhexidine varnishes in preventing and treating periodontal disease. A review of the literature. Med Oral Patol Oral Cir Bucal.  2008;13(4):E257-E260.1515 Van Leeuwen áMP, Slot áDE, Van der Weijden áGA. Essential oils compared to chlorhexidine with respect to plaque and parameters of gingival inflammation: a systematic review. J Periodontol.  2011;82(2):174-194.1616 Osso áD, Kanani áN. Antiseptic mouth rinses: an update on comparative effectiveness, risks and recommendations. J Dent Hyg.  2013;87(1):10-18.

SEE ALSO


  • Dental Infection; Glossitis
  • Algorithm: Bleeding Gums

CODES


ICD10


  • K05.10 Chronic gingivitis, plaque induced
  • K05.11 Chronic gingivitis, non-plaque induced
  • K05.00 Acute gingivitis, plaque induced
  • K05.01 Acute gingivitis, non-plaque induced
  • A69.1 Other Vincent's infections

ICD9


  • 523.10 Chronic gingivitis, plaque induced
  • 523.11 Chronic gingivitis, non-plaque induced
  • 523.00 Acute gingivitis, plaque induced
  • 523.01 Acute gingivitis, non-plaque induced
  • 101 Vincent's angina

SNOMED


  • 66383009 Gingivitis (disorder)
  • 72621003 Chronic gingivitis (disorder)
  • 31642005 Acute gingivitis (disorder)
  • 172697005 Acute ulcerative gingivitis
  • 409865002 Vincent's disease (disorder)

CLINICAL PEARLS


  • Gingivitis may be prevented and treated with regular dental cleanings, good oral hygiene, and use of certain mouth rinses including chlorhexidine.
  • Untreated, gingivitis may progress to periodontitis, a possible contributor to systemic inflammation and its consequences (e.g., coronary artery disease and uncontrolled diabetes).
  • New-onset or difficult-to-treat gingivitis, consider differential of etiology: pregnancy, HIV, diabetes, medications, and vitamin deficiencies.
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