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Fever, Adult, Emergency Medicine


Basics


Description


  • Fever is an elevation of core body temperature caused by an increase in the bodys thermoregulatory set point.
  • Prostaglandin E2 (PGE2) synthesis in the anterior hypothalamus controls the thermostat, and is the target of antipyretics.
  • Core temperature is regulated to 37 °C ± 2 °C.
  • Autonomic discharge from hypothalamus can raise core temperature through shivering and dermal vasoconstriction.
  • Normal circadian variation in core temperature occurs with nadir in early morning and peaks in late afternoon.
  • Fever is not synonymous with hyperthermia or hyperpyrexia.
  • Hyperthermia is an elevated temperature with normal thermostat set point; caused by excessive endogenous heat production or endogenous production (e.g., malignant hyperthermia or heat stroke).
  • Hyperpyrexia is extreme fever >41.5 °C usually from CNS hemorrhages.
  • Both exogenous and endogenous factors can raise the body's set thermoregulatory point:
    • Endogenous pyrogens include PGE2, IL-1, IL-6, TNF, IFN-γ.
    • Exogenous pyrogens include lipopolysaccharide (LPS) endotoxin and other TLR ligands, and toxic shock syndrome toxin (TSST-1) and other MHC II ligands.
  • Patients on anticytokine medications or glucocorticoids have impaired fever response.
  • Fever of unknown origin (FUO):
    • Fever >38.3 °C for at least 3 wk as an outpatient and 3 days of inpatient evaluation or 3 outpatient visits without determining etiology.

Etiology


  • Infectious processes:
    • CNS, chest and lung, gastrointestinal, genitourinary, skin, soft tissue and bone, vascular and endocardial
    • Iatrogenic: Catheters, implants, hardware, recent surgical sites.
  • 1 ° CNS processes such as CVA, trauma, seizures
  • Neoplastic fevers
  • Drug fever:
    • Most drugs can cause elevated temperatures by a wide variety of mechanisms
    • Toxidromes (e.g., adrenergic, anticholinergic, dopaminergic, salicylate overdose, serotonin toxicity)
    • Hypersensitivity:
      • Allergic reaction
      • Serum sickness
    • Jarisch-Herxheimer reaction
    • Local phlebitis from irritant drugs
  • Severe withdrawal:
    • Alcohol
    • Benzodiazepines
  • Systemic rheumatologic and inflammatory diseases (e.g., familial Mediterranean fever, rheumatoid arthritis, sarcoidosis, systemic lupus erythematosus, temporal arteritis)
  • Endocrine:
    • Hyperthyroidism, pheochromocytoma
  • Miscellaneous:
    • Alcoholic cirrhosis
    • Acute inhalation exposures (e.g., metal fume fever)
    • Cotton fever:
      • Febrile reaction from an injected contaminant when IV drug abusers strain drug through cotton
    • Sickle cell disease
    • Hemolytic anemia
    • Pulmonary embolus
  • Common causes of FUO:
    • Infectious:
      • Abdominal and pelvic abscesses
      • Cardiac (endocarditis, pericarditis)
      • Cat scratch disease
      • Cytomegalovirus
      • Epstein-Barr virus
      • TB (miliary, renal, or meningitic)
      • Typhoid enteric fevers
      • Visceral leishmaniasis
    • Neoplastic:
      • Colon adenocarcinoma
      • Hepatocellular carcinoma and metastases
      • Myeloproliferative disorders
      • Leukemia and lymphoma
      • Renal cell carcinoma

Diagnosis


Signs and Symptoms


History
  • Chills, shivering, and rigors:
    • Rigors may suggest bacteremia
  • Weight loss:
    • Suggestive of neoplastic, chronic infectious, or endocrine disorders
  • Night sweats:
    • Suggestive of neoplastic, chronic inflammatory disease, or TB
  • Specific fever patterns:
    • Daily morning temperature spikes:
      • Miliary TB, typhoid fever, polyarteritis nodosa
    • Relapsing fevers: Febrile episode with alternating afebrile intervals:
      • Seen in malaria, Borrelia infections, rat-bite fever, and lymphoma
    • Remittent fever: Temperature falls daily but does not return to normal:
      • Seen in TB and viral diseases
    • Intermittent fevers: Exaggerated circadian rhythm:
      • Seen in systemic infections, malignancy, and drug fever
    • Double quotidian fever:
      • Common pattern of 2 temperature spikes in 24 hr
      • In FUO, consider miliary TB, visceral leishmaniasis, and malarial infections
  • High-risk features:
    • Anticytokine therapy (e.g., TNF-α monoclonal antibodies, calcineurin inhibitors)
    • Glucocorticoid use
    • Immunosuppressed states
    • Incomplete vaccination status
    • IV drug use
    • Pregnancy and peripartum patients
    • Rash
    • Recent chemotherapy
    • Recent travel
    • Splenectomy

Physical Exam
  • Elevated core temperature:
    • Temperature >38 °C (100.4 °F) rectally or 37.5 °C (99.5 °F) orally
    • Lower thresholds in patients older than 65 yr, as the febrile response is not as strong
  • Diaphoresis:
    • Absence of diaphoresis with severe hyperthermia suggests anticholinergic poisoning or heat stroke.
  • Tachycardia:
    • For each degree of elevation in temperature in Fahrenheit, there should be a 10 bpm increase in pulse.
    • Relative bradycardia (Faget sign):
      • Associated with malaria, typhoid fever, CNS disorders, lymphoma, drug fever, brucellosis, ornithosis, Legionnaire disease, Lyme disease, and factitious fevers
  • Muscle rigidity, clonus, and hyper-reflexia:
    • Associated with specific toxidromes and medical conditions
  • Changes in mental status:
    • Toxic-metabolic encephalopathy vs. primary CNS disorder
  • Rash:
    • Lesion type, distribution, and progression can offer important clues to diagnosis.
    • Petechia, purpura, vesicles, mucosal, or palm and sole involvement require special note
  • Signs of hyperthyroidism:
    • Goiter
    • Exophthalmos

Essential Workup


  • Core temperature is most acutely measured rectally.
  • Careful history and physical exam (PE) necessary to determine need for further diagnostic testing:
    • History should elicit any sick contacts, previous infections, occupational exposures, recent travel, medications, animal or tick exposure, and immunization status.

Diagnosis Tests & Interpretation


Lab
  • CBC:
    • Important in determining neutropenia in patients with risk factors
    • Neutrophilia and bandemia suggestive of bacterial infection
    • Lymphocytosis suggestive of typhoid, TB, brucellosis, and viral disease
    • Atypical lymphocytosis seen in mononucleosis, cytomegalovirus, HIV, rubella, varicella, measles, and viral hepatitis
    • Monocytosis suggestive of TB, brucellosis, viral illness, and lymphoma
  • Lactate:
    • Initial and repeat measurements useful for screening for sepsis, risk stratification, and management decisions
  • Urinalysis and urine culture
  • Blood cultures:
    • Obtain for all systemically ill patients, and patients at risk for bacteremia
  • Thick and thin blood smears and malaria antigen testing in at-risk individuals for parasitic and intraerythrocytic infections
  • Stool culture and Clostridium difficile assay for suspected individuals.
  • Heterophile antibody testing in select patients.
  • Erythrocyte sedimentation rate and C-reactive protein generally not useful:
    • Very high values suggestive of endocarditis, osteomyelitis, TB, and rheumatologic conditions.

  • Decreased immunocompetence, increased risk of systemic spread, increased exposure to health care settings, may have comorbid conditions.
  • If institutionalized consider the infectious implications of multiple potential sick contacts.

Imaging
  • CXR:
    • In patients with PE finding of cardiopulmonary disease and patients with unclear fever source
  • CT or MRI may be indicated if lumbar puncture or osteomyelitis is considered, respectively.

Differential Diagnosis


  • The differential diagnosis is very broad as listed above, but is generally categorized as infectious vs. noninfectious, and by immunocompetency.

Treatment


Pre-Hospital


  • No specific field interventions required
  • Monitoring and IV access should be obtained in the field for unstable patients or patients with altered mental status.

Initial Stabilization/Therapy


  • ABCs for unstable patients.
  • Initiate early broad-spectrum antibiotics for patients with suspected sepsis or unstable vital signs, particularly those who are at high risk for serious bacterial infection.

Ed Treatment/Procedures


  • Antipyretics:
    • Generally either acetaminophen or NSAIDs
      • Inhibit the cyclooxygenase enzyme, thereby blocking synthesis of prostaglandins.
  • Empiric antibiotics for neutropenic patients:
    • Combination therapy:
      • Extended spectrum β-lactam (ceftazidime, piperacillin) with an aminoglycoside
    • Monotherapy:
      • Cefepime
      • Ceftazidime
      • Imipenem
  • Empiric antibiotics for asplenic patients for encapsulated bacteria
  • Empiric antiviral therapy for patients with encephalitis and potential disseminated viral infections (e.g., recent organ or bone marrow transplant patients, AIDS patients)
  • External cooling mechanism rarely indicated

Medication


  • Antipyretics:
    • Acetaminophen: 650-1,000 mg PO/PR q4-6h; do not exceed 4 g/24h
    • Aspirin: 650 mg PO q4h; do not exceed 4 g/24h
    • Ibuprofen: 800 mg PO q6h
  • Antibiotics:
    • Cefepime: 2 g IV q12
    • Ceftazidime: 2 g IV q8
    • Gentamicin or tobramycin (D): 2 mg/kg IV load then 1.7 mg/kg q8h + piperacillin/tazobactam (B) 3.375 g IV q4h or ticarcillin/clavulanate (B) 3.1 g IV q4h
    • Imipenem/cilastatin: 500-1,000 mg IV q8h
    • Meropenem (B): 1 g IV q8h
    • Ciprofloxacin: 750 mg PO BID + amoxicillin/clavulanate (B) 875 mg PO BID
  • Antivirals:
    • Herpes simplex virus and varicella-zoster virus (VZV):
    • Acyclovir 10-15 mg/kg IV q8h
    • Influenza A and B:
    • Oseltamivir 75 mg PO q12h

Follow-Up


Disposition


Admission Criteria
  • Patients with unstable vital signs require ICU admission.
  • When identified, the underlying source of the fever usually determines the disposition.
  • Certain high-risk groups who have fever without an identifiable source:
    • Neutropenic patients
    • Immunosuppressed or immunocompromised patients
    • Asplenic patients
    • IV drug abusers
  • Lower thresholds for admission in patients older than 60 yr and diabetics

Discharge Criteria
Immunocompetent patients with stable vital signs and an identified source of fever or a high suspicion of a nonthreatening viral infection may be safely discharged.  
Issues for Referral
The suspected etiology of the fever determines the referral to a primary care physician or a specialist.  

Followup Recommendations


Appropriate outpatient treatment and follow-up for further outpatient assessment of the suspected etiology.  

Pearls and Pitfalls


  • Screening lactates for sepsis.
  • Early, empiric, and broad-spectrum antibiotic coverage for all septic patients.
  • Consider all potential sources of infection.
  • Careful consideration for the immunosuppressed, elderly, and IV drug users.

Additional Reading


  • Cunha  BA. Fever of unknown origin: Focused diagnostic approach based on clinical clues from the history, physical examination, and laboratory tests. Infect Dis Clin North Am.  2007;21:1137-1187.
  • Freifeld  AG, Bow  EJ, Sepkowitz  KA, et al. Clinical practice guideline for the use of antimicrobial agents in neutropenic patients with cancer: 2010 update by the Infectious Diseases Society of America. Clin Infect Dis.  2011;52:e56-e93.
  • Mackowiak  PA, Wasserman  SS, Levine  MM. A critical appraisal of 98.6 degrees F, the upper limit of the normal body temperature, and other legacies of Carl Reinhold August Wunderlich. JAMA.  1992;268(12):1578-1580.

Codes


ICD9


  • 780.60 Fever, unspecified
  • 780.61 Fever presenting with conditions classified elsewhere

ICD10


  • R50.2 Drug induced fever
  • R50.9 Fever, unspecified
  • R50.81 Fever presenting with conditions classified elsewhere

SNOMED


  • 386661006 fever (finding)
  • 7520000 Pyrexia of unknown origin (finding)
  • 95908009 Drug fever (finding)
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