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Epstein-Barr Virus (Infectious Mononucleosis), Pediatric


Basics


Description


A double-stranded DNA virus implicated as a causative agent for infectious mononucleosis by an infected laboratory worker in 1968 �

General Prevention


  • No vaccine is clinically available.
  • Standard precautions should be used in the hospitalized patient.
  • Restriction of intimate contact with immunosuppressed individuals may be advisable.
  • Patients with recent Epstein-Barr virus (EBV) infection, either proven or suspected, should not donate blood or solid organs.

Epidemiology


  • Worldwide distribution
  • Humans are the only known reservoir.
  • Transmission occurs through saliva and, occasionally, via blood transfusions and solid organ transplant (SOT).
  • Incubation period is 4-7 weeks.
  • Antibodies to EBV are almost universally present in adult populations.
  • Areas with a high population density or low socioeconomic status usually become primarily infected within the first 3 years of life.

Incidence
In developed countries, acquisition of EBV is biphasic. �
  • Initial peak in incidence occurs before the age of 5 years.
  • Second peak occurs during adolescence, coinciding with an increased frequency of intimate oral contacts.

Prevalence
90-95% of adults have demonstrable EBV titers. �

Pathophysiology


  • Replicates initially in the oropharyngeal epithelium
  • Selective infection of B lymphocytes occurs.
  • The clinical syndrome of infectious mononucleosis results from proliferation of cells in the tonsils, lymph nodes, and spleen.
  • Nonspecific humoral immune responses include the formation of heterophile antibodies and autoantibodies.
  • Specific antibodies to EBV antigens are produced.
  • Despite humoral responses, cellular immunity is responsible for controlling EBV infection.
  • Latent, lifelong infection of B lymphocytes occurs.
  • Latent virus may be reactivated during periods of immunosuppression.

Commonly Associated Conditions


  • Subclinical infection
    • Most EBV infections in children, and even in adolescents, are clinically inapparent.
    • Mild, nonspecific symptoms may include coryza, diarrhea, and/or fever.
    • Immunologic seroconversion does occur.
  • Infectious mononucleosis ("glandular fever"�): most commonly observed with late primary acquisition of EBV. The classically defined illness is characterized by the following:
    • Fatigue
    • Malaise
    • Fever
    • Tonsillopharyngitis (often exudative)
    • Lymphadenopathy
    • Splenomegaly
    • Usually associated with increased atypical lymphocytes in the peripheral blood
  • Rare illnesses of the nervous system have been reported, including the following:
    • Guillain-Barr � syndrome
    • Bell palsy
    • Aseptic meningitis
    • Meningoencephalitis
    • Peripheral and/or optic neuritis
  • Hematologic complications have been reported in association with EBV.
    • Aplastic anemia
    • Hemolytic anemia
    • Agranulocytosis
    • Hemophagocytic syndrome
  • Other illnesses associated with EBV in case reports include the following:
    • Hemolytic uremic syndrome
    • Hepatitis
    • Pancreatitis
    • Myocarditis
    • Mesenteric adenitis
    • Orchitis
    • Genital ulcerative disease
  • Lymphoproliferative disorders
    • Burkitt lymphoma
    • Nasopharyngeal carcinoma
    • Lymphoma and non-Hodgkin lymphoma (in immunocompromised children)
    • Lymphomatoid granulomatosis
    • Posttransplant lymphoproliferative disorders (PTLD)
    • X-linked lymphoproliferative disease (Duncan disease)

Diagnosis


History


  • A prodrome may occur.
    • Most often, lasts 3-5 days
    • Malaise, fatigue, with or without fever
  • In the acute phase, the following features are common:
    • Fever: begins abruptly, lasts 1-2 weeks
    • Fatigue
    • Malaise
    • Anorexia
    • Sore throat
    • "Swollen glands"�
    • Rash; more common with ampicillin administration
  • Young children are more likely to have rash or abdominal pain.

Physical Exam


  • Tonsillopharyngitis
    • May be exudative and mimic streptococcal pharyngitis
    • Often accompanied by palatal petechiae
  • Lymphadenopathy
    • Occurs in 90%
    • Most prominent in cervical chains
    • May be diffuse
    • Usually nontender, nonerythematous, and discrete
  • Hepatosplenomegaly
    • Splenomegaly occurs in more than half the cases.
    • Even if not palpable, splenomegaly may be demonstrated on ultrasound.
    • Most prominent in 2nd-4th week of illness
    • Hepatomegaly is less common.

Diagnostic Tests & Interpretation


Lab
  • Complete blood count with differential
    • Leukocyte count up to 20,000/mm3
    • Lymphocytosis
    • Atypical lymphocytes often constitute >10% of total leukocyte count.
    • Thrombocytopenia may occur.
    • False positives: Atypical lymphocyte counts >10% of the total leukocyte count also occur with cytomegalovirus and toxoplasmosis infections.
  • Liver enzymes
    • Mild hepatitis is often found.
    • Jaundice is rare.
  • "Monospot"� (mononucleosis rapid slide agglutination test for heterophile antibodies)
    • Detects heterophile antibodies (nonspecific IgM antibodies to unrelated antigens)
    • Appears in first 2 weeks of illness, usually slow decline over 6 months
    • Detects 85% of cases in adolescents/adults
    • False positives: infrequent; heterophile antibodies are also produced in serum sickness and neoplastic processes; heterophile antibodies may persist for months after acute infection and be indicative of past illness.
  • EBV serology
    • Usually reserved for heterophile-negative patients or children <4 years of age when strong clinical suspicion persists
    • Antibodies are detected by indirect immunofluorescence or enzyme-linked immunosorbent assay techniques.
    • Acute or past infection can usually be detected and differentiated.
    • EBV IgM is consistent with acute infection, whereas EBV nuclear antibody (EBNA) is indicative of past infection.
  • Other technology
    • Tissue culture of EBV is difficult and, therefore, not clinically useful.
    • Polymerase chain reaction (PCR) may detect EBV genetic material.
    • Real-time PCR may quantify the amount of EBV genome present, which is useful in patients with PTLD.

Alert
  • Heterophile antibodies may not appear early in the illness.
  • Up to 10% of patients with acute EBV infection may have no heterophile response 3 weeks into the illness.
  • The heterophile response is less common in infants and children and should not be used in children <4 years of age.

Differential Diagnosis


  • Infectious
    • Group A Streptococcus
    • Adenovirus
    • Cytomegalovirus
    • Toxoplasma gondii
    • Human herpesvirus-6
    • Mycoplasma pneumoniae
    • Human immunodeficiency virus
    • Rubella
    • Diphtheria
    • Viral hepatitis (A, B, C)
  • Noninfectious
    • Leukemia/lymphoma

Treatment


Medication


  • Acetaminophen or ibuprofen reduces fever and provides analgesia.
  • Corticosteroids (prednisone 1 mg/kg/24 h PO, maximum of 20 mg/24 h) may reduce swelling of lymphoid tissues (see "FAQ"�)
    • Indicated for patients with impending airway obstruction
    • May be considered for patients with severe tonsillopharyngitis requiring IV hydration
    • May be considered for patients with rare, life-threatening manifestations of EBV infection, such as hepatitis, aplastic anemia, and central nervous system dysfunction
    • 7-day treatment followed by tapering
  • Acyclovir has not been shown to provide clinical benefit; sometimes, used in cases of active replicating EBV in posttransplant situations
  • Patients with PTLD should have immunosuppression reduced.
  • Advise avoidance of contact sports until resolution of symptoms and no further splenomegaly.

Inpatient Considerations


Admission Criteria
  • Respiratory distress secondary to airway obstruction
  • Dehydration secondary to severe pharyngitis and poor oral intake

Discharge Criteria
  • Resolved airway obstruction
  • Good oral intake

Issues for Referral


  • PTLD
  • EBV in immunocompromised host
  • EBV-associated lymphoproliferative disorders
  • Considering steroid use as treatment

Ongoing Care


Follow-up Recommendations


Patient Monitoring
  • Immunocompetent individuals usually recover uneventfully in 1-4 weeks.
  • Recovery is often biphasic, with a worsening of symptoms after a period of improvement.
  • Splenomegaly may persist for weeks after primary infection (see "FAQ"�).
  • Fatigue may persist months after recovery.

Prognosis


  • Most patients with primary EBV infection will recover uneventfully in 1-4 weeks.
  • Long-lasting immunity generally ensues.
  • Prognosis of patients with unusual manifestations of EBV infection depends on the severity of the illness and the organ system involved.
  • Patients with inherited or acquired immunodeficiency are at higher risk of complications and neoplasms.

Complications


  • Dehydration
    • Severe pharyngitis often limits fluid intake.
    • Most common problem requiring hospitalization
  • Antibiotic-induced rash
    • Morbilliform in appearance
    • Most common after administration of ampicillin or amoxicillin
    • Rare association with penicillin
    • Usually benign; resolves with discontinuation of the aminopenicillin
  • Splenic rupture
    • Incidence of ~1 in 1,000 patients
    • More common in males
    • 50% of the cases of splenic rupture are spontaneous; 50% follow blunt trauma.
  • Airway obstruction: may result from massive lymphoid hyperplasia and mucosal edema

Additional Reading


  • Bravender �T. Epstein-Barr virus, cytomegalovirus, and infectious mononucleosis. Adolesc Med State Art Rev.  2010;21(2):251-264. �[View Abstract]
  • Hurt �C, Tammaro �D. Diagnostic evaluation of mononucleosis-like illness. Am J Med.  2007;20(10):911. e1-911. e8. �[View Abstract]
  • Macsween �KF, Crawford �DH. Epstein-Barr virus-recent advances. Lancet Infect Dis.  2003;3(3):131-140. �[View Abstract]
  • Okano �M. Overview and problematic standpoints of severe chronic active Epstein-Barr virus infection syndrome. Crit Rev Oncol Hematol.  2002;44(3):273-282. �[View Abstract]
  • Putukian �M, O'Connor �FG, Stricker �P, et al. Mononucleosis and athletic participation: an evidence-based subject review. Clin J Sport Med.  2008;18(4):309-315. �[View Abstract]

Codes


ICD09


  • 075 Infectious mononucleosis

ICD10


  • B27.90 Infectious mononucleosis, unspecified without complication
  • B27.99 Infectious mononucleosis, unsp with other complication
  • B27.91 Infectious mononucleosis, unspecified with polyneuropathy
  • B27.92 Infectious mononucleosis, unspecified with meningitis

SNOMED


  • 402121009 Epstein-Barr virus infection (disorder)
  • 271558008 Infectious mononucleosis (disorder)

FAQ


  • Q: Should all patients with infectious mononucleosis be given corticosteroids?
  • A: No. Symptomatic EBV infection is most often self-limited. EBV has been linked to certain lymphoproliferative disorders, and theoretic risks to modulating the host immune response with corticosteroids have been proposed.
  • Q: How long after infectious mononucleosis may a patient return to athletic activity?
  • A: More than half of patients with "mono"� will have a boggy, enlarged spleen, which is prone to rupture even if it is not palpable. Athletic activity should be restricted until no evidence exists for a clinically enlarged spleen. Return to contact sports is not advised until 4-6 weeks after resolution of illness. Some experts recommend ultrasound of the spleen before a return to heavy contact sports such as rugby, football, lacrosse, and hockey.
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