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Dermatomyositis

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  • Muscles Symmetric proximal extremity muscle weakness (extensors)

    • Lungs

    • Check for bibasilar fine crackles/rales
      • 15-30% develop interstitial lung disease
      • Poor prognosis
    • Risk aspiration pneumonia and respiratory failure due to respiratory muscle weakness

    • Cardiac

    • Conduction defects, myocarditis, congestive heart failure
    • May be under-recognized; major prognostic factor for death

    • Joints

    • Nonerosive arthritis of small joints

    Tests


    Lab
    • Muscle enzymes: Creatine kinase (CK), aldolase may be elevated
      • Early disease or amyopathic disease may have no elevation.
      • CK most sensitive. Others affected: Lactate dehydrogenase, aspartate aminotransferase, alanine aminotransferase
    • Autoantibodies: Help define subtypes but role in pathogenesis unclear. Not useful for diagnostic purposes but helpful for prognosis
      • Antinuclear (ANA) (+1/3 patients)
      • Anti-Jo-1 (anti-histidyl-tRNA synthetase) (+ in 20%)
        • 70% increased risk for interstitial lung disease; associated with anti-synthetase syndrome
      • Anti-Mi-2 (anti-nuclear helicase) (+ in 5-15%)
        • Highly specific for DM

    Surgery
    • Optional
      • Electromyography: If muscle enzymes (-)
      • Muscle biopsy: Gold standard; consider if above negative
      • MRI: If above negative or to help localize place to biopsy
      • Skin biopsy: Lupus will have similar features
    • Screen for other organ involvement
      • Pulmonary function tests with diffusing capacity
      • Baseline EKG
      • Esophageal study (except if amyopathic)
    • Malignancy work-up in women
      • Pelvic, breast, rectal exam
      • CBC, CMP, urinalysis, TSH, stool occult blood test, colonoscopy (if age appropriate), PAP smear, CA-125
      • CT chest/abdomen/pelvis
      • Transvaginal pelvic ultrasound

    Differential Diagnosis


    • Cutaneous findings:
      • Systemic lupus erythematosus, psoriasis, airborne or contact dermatitis, photodrug eruption, cutaneous T-cell lymphoma, atopic dermatitis, scleroderma
    • Muscle findings and elevated creatine phosphokinase:
      • Polymyositis, hypothyroidism, viral myositis, rhabdomyolysis, trichinosis, myasthenia gravis

    Treatment


    • There is a discordance between response of muscle disease and that of dermatologic disease to systemic therapy.
    • Treatment depends on presence of muscle disease and organ involvement.
    • Only FDA-approved drug is intravenous immunoglobulin (IVIG); however, other medications are considered first line.

    Cutaneous Disease


    • First line
      • Sunscreen (broad-spectrum UVB/UVA) (1)[C]
      • High-potency topical steroid (including face) — 2 weeks then switch to topical calcineurin inhibitor (Protopic ®, Elidel ®) (1)[C]
      • Treat pruritus (hydroxyzine, diphenhydramine) (1)[C]
    • Second line (if severe)
      • Hydroxychloroquine (1)[B]

    20% with dermatomyositis develop morbilliform eruption with hydroxychloroquine  
    • Hydroxychloroquine plus quinacrine (1)[C]
    • Methotrexate: Low-dose weekly (1)[B]
      • Third line
    • Mycophenolate mofetil (CellCept ®)(1)[B]
    • IVIG (1)[A]
    • Thalidomide (1)[C]
    • Dapsone (1)[C]

    Muscle Disease


    • First line
      • Oral prednisone: 1-2 mg/kg/day tapered to 50% over 6 months, and to zero over 2-3 years (1)[A]; especially if swallowing problem
        • Option to use pulse, split dose, or alternate day (1)[B]
      • Often with addition of steroid sparing agent:
        • Methotrexate: Low-dose weekly (1)[B]
        • Mycophenolate mofetil (CellCept ®) 2.5 g/day divided (1)[B]
        • Azathioprine: 2-3 mg/kg/day (1)[C]
    • Second line +/- prednisone
      • Azathioprine"‚+ methotrexate (1)[B]
      • IVIG (1)[A]; can combine with any of above
      • Cyclosporine (1)[B]
    • Third line
      • Rituximab (1)[C]
      • TNF-alpha inhibitors (1)[B]

    TNF blockers can also induce dermatomyositis.  
    • Pulmonary disease

    • First line
      • Oral prednisone (2)[A]
    • Second line +/- prednisone
      • Cyclophosphamide (2)[A]
      • Azathioprine (2)[C]
      • Methotrexate (2)[C]

    Additional Treatment


    General Measures
    • Physical therapy to prevent contractures, avoid aggressive weight training.
    • If severe muscle inflammation, bed rest can be of benefit.
    • If dysphagia, elevation of head of bed and avoid eating before bed.

    Issues for Referral
    Multidisciplinary care: Rheumatology, dermatology, neurology, oncology (if cancer-associated), pulmonary  

    Ongoing Care


    Follow-Up Recommendations


    Patient Monitoring
    • Monitor regularly for esophagus, lung, cardiac, joint involvement
    • If amyopathic, monitor CK closely; muscle strength exams at 1-3 month intervals
      • If no muscle involvement at 2 years, can consider "amyopathic,"ť but with continued muscle strength screening
      • If muscle disease confirmed, systemic therapy needed
    • Malignancy screening yearly
      • Highest risk in first 2-5 years, but risk does not return to zero

    Patient Education


    Aggressive sun protection: Sun-protective clothing; broad-spectrum UVB/UVA sunscreen  

    Prognosis


    • Long-term outcomes are variable and range from remission to progressive disease to death.
      • 20% may spontaneously remit, 5% may have a fulminant, progressive course.
      • Death occurs in about 10-20%, with worse prognosis if cancer-associated, pulmonary or cardiac involvement, or elderly.
    • Skin disease presents first in 50% of patients. Myositis typically follows 3-6 months later.

    Complications


    • Treatment-related: Infections and medication side effects
    • Muscle-related difficulty swallowing or chest muscle weakness: Weight loss and malnutrition; aspiration pneumonia; respiratory failure
    • Skin: Calcium deposits; skin breakdown; Raynaud's syndrome

    References


    1Iorizzo  LJ 3rd, Jorizzo  JL. The treatment and prognosis of dermatomyositis: An updated review. J Am Acad Dermatol.  2008;59(1):99-112.  [View Abstract]2Connors  G, Christopher-Stine  L, Oddis  CV. Interstitial lung disease associated with the idiopathic inflammatory myopathies: What progress has been made in the past 35 years? Chest.  2010;138(6):1464-1474.  [View Abstract]

    Additional Reading


    1Goreshi  R, Chock  M, Foering  K. Quality of life in dermatomyositis. J Am Acad Dermatol.  2011;65(6):1107-1116.  [View Abstract]2Madan  V, Chinoy  H, Griffiths  CE. Defining cancer risk in dermatomyositis. Part I. Clin Exp Dermatol.  2009;34(4):451-455.  [View Abstract]

    Codes


    ICD9


    710.3 Dermatomyositis  

    ICD10


    • M33.10 Other dermatopolymyositis, organ involvement unspecified
    • M33.90 Dermatopolymyositis, unsp, organ involvement unspecified
    • M33.91 Dermatopolymyositis, unsp with respiratory involvement
    • M33.99 Dermatopolymyositis, unsp with other organ involvement

    SNOMED


    • 396230008 dermatomyositis (disorder)
    • 238935002 dermatomyositis sine myositis (disorder)
    • 46696008 dilated cardiomyopathy secondary to dermatomyositis (disorder)

    Clinical Pearls


    • Amyopathic dermatomyositis (DM), in addition to classic DM, can be associated with both pulmonary disease and malignancy.
    • Risk of malignancy highest in first 2-5 years, but risk does not return to zero.
    • Pulmonary involvement is not universal, but is an important cause of morbidity and mortality.
    • The mainstay of therapy for classic dermatomyositis (skin and muscle involvement) is long-term systemic steroids, often in conjunction with a steroid-sparing systemic agent.
    • Myopathy usually resolves with therapy. However, skin is often resistant.
    • Quality of life significantly reduced.
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