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Croup (Laryngotracheobronchitis)


BASICS


DESCRIPTION


  • Croup is a subacute viral illness characterized by upper airway symptoms such as barking cough, stridor, and fever. "Croup"� is used to refer to viral laryngotracheitis or laryngotracheobronchitis (LTB), although it is sometimes used for LTB with pneumonitis, bacterial tracheitis, or spasmodic croup.
  • Most common cause of upper airway obstruction or stridor in children
  • Spasmodic croup: noninfectious form with sudden resolution
    • No fever or radiographic changes
    • Initially treated as croup
    • Usually self-limiting and resolves with mist therapy at home
    • Often recurs on same night or in 2 to 3 nights
  • System(s) affected: pulmonary, respiratory
  • Synonym(s): infectious croup; viral croup

EPIDEMIOLOGY


  • Predominant age
    • Common among children 7 months to 3 years
    • Most common during the 2nd year of life
    • Rare among those >6 years
  • Predominant sex: male > female (1.5:1)
  • Timing
    • Possible during any time of year but is most common in autumn and winter (with parainfluenza 1 and respiratory syncytial virus [RSV])

Incidence
  • Six cases per year per 100 children <6 years old
  • 1.5-6% of cases require hospitalization.
  • 2-6% of those require intubation.
  • Decreasing incidence in the United States and Canada

ETIOLOGY AND PATHOPHYSIOLOGY


  • Subglottic region/larynx is entirely encircled by the cricoid cartilage.
  • Inflammatory edema and subglottic mucus production decrease airway radius.
  • Small children have small airways with more compliant walls.
  • Negative-pressure inspiration pulls airway walls closer together.
  • Small decrease in airway radius causes significant increase in resistance (Poiseuille law: resistance proportional to 1/radius4).
  • Usually viruses that initially infect oropharyngeal mucosa and then migrate inferiorly
  • Parainfluenza virus
    • Most common pathogen: 75% of cases
    • Type 1 is the most common, causing 18% of all cases of croup.
    • Types 2, 3, and 4 are also common.
    • Type 3 may cause a particularly severe illness.
  • Other viruses: RSV, paramyxovirus, influenza virus type A or B, adenovirus, rhinovirus, enteroviruses (coxsackie and echo), reovirus, measles virus where vaccination not common, and metapneumovirus
  • Haemophilus influenzae type B now rare with routine immunization
  • May have bacterial cause: Mycoplasma pneumoniae has been reported.

RISK FACTORS


  • History of croup
  • Recurrent upper respiratory infections
  • Atopic disease increases the risk of spasmodic croup.

COMMONLY ASSOCIATED CONDITIONS


  • If recurrent (>2 episodes in a year) or during first 90 days of life, consider host factors.
  • Underlying anatomic abnormality (e.g., subglottic stenosis)
    • In one study, found to be present in 59% children with recurrent croup
  • Paradoxical vocal cord dysfunction
  • Gastroesophageal reflux disease
  • Neonatal intubation

DIAGNOSIS


  • Most children who present with acute onset of barky cough, stridor, and chest wall indrawing have croup.
  • Croup is a clinical diagnosis; lab tests and imaging serve only ancillary purposes.
  • Classic "seal-like"� barking, spasmodic cough
  • May have biphasic stridor
  • Low- to moderate-grade fever
  • Upper respiratory infection prodrome lasting 1 to 7 days
  • Severity usually is determined by clinical observation for signs of respiratory effort: nasal flaring, retractions, tripoding, sniffing position, abdominal breathing, and tachypnea. Later symptoms: hypoxia/cyanosis or fatigue
  • Westley croup score (≤2 mild; 3 to 7 moderate; ≥8 severe), most useful for research purposes
    • Level of consciousness: normal, including sleep, 0; disoriented, 5
    • Cyanosis: none, 0; with agitation, 4; at rest, 5
    • Stridor: none, 0; with agitation, 1; at rest, 2
    • Air entry: normal, 0; decreased, 1; markedly decreased, 2
    • Retractions: none, 0; mild, 1; moderate, 2; severe, 2
  • No change in stridor with positioning
  • Nontender larynx
  • Inflamed subglottic region with normal-appearing supraglottic region
  • Differentiate from epiglottitis: non-toxic-appearing, normal voice, no drooling, is coughing (1).

HISTORY


  • 2 to 3 days of nonspecific prodromal syndrome with low-grade fever, coryza, and rhinorrhea
  • Onset and recurrence at night when child is sleeping
  • Symptoms often resolve en route to the hospital, as the child is exposed to cool night air.
  • Lack of prodrome indicates spasmodic croup.

PHYSICAL EXAM


  • Pulse oximetry often is normal because there is no disturbance of alveolar gas exchange.
  • Overall appearance: Is the child comfortable or struggling?
  • Work of breathing: labored or comfortable?
  • Sound of breathing and voice: hoarse, stridor, inspiratory wheezing, short sentences?
  • Observed/subjective tidal volume: sufficient for child's size?

DIFFERENTIAL DIAGNOSIS


  • Epiglottitis: currently rare
  • Foreign body aspiration
  • Subglottic stenosis (congenital or acquired)
  • Bacterial tracheitis
  • Simple upper respiratory infection
  • Retropharyngeal or peritonsillar abscess
  • Trauma
  • Allergic reaction (acute angioneurotic edema)
  • Airway anomalies (e.g., tracheo-/laryngomalacia)
  • Other anatomic obstructions: subglottic hemangioma, subglottic cyst

DIAGNOSTIC TESTS & INTERPRETATION


  • No laboratory abnormality is diagnostic.
  • WBCs may be low, normal, or elevated.
  • Lymphocytosis is expected but not required.
  • Rapid antigen or viral culture tests are available in some centers.
    • Guide isolation precautions, not management.

Initial Tests (lab, imaging)
  • Posteroanterior and lateral neck films show funnel-shaped subglottic region with normal epiglottis: "steeple,"� "hourglass,"� or "pencil point"� sign (present in 40-60% of children with LTB).
  • CT may be more sensitive for defining obstruction in a confusing clinical picture.
  • Patient should be monitored during imaging; airway obstruction may occur rapidly.

Test Interpretation
  • Inflammatory reaction of respiratory mucosa
  • Loss of epithelial cells
  • Thick mucoid secretions

TREATMENT


GENERAL MEASURES


  • Minimize lab tests, imaging, and procedures that upset the child; agitation worsens tachypnea and can be more detrimental than accepting a clinical diagnosis.
  • ECG monitoring and pulse oximetry
    • Frequent checks are more sensitive to worsening disease than is pulse oximetry.

MEDICATION


First Line
  • Well established in the literature; cornerstones of treatment are immediate nebulized epinephrine and dexamethasone.
  • Racemic or L-epinephrine (Equal efficacy and side effect profiles; L-epinephrine is used for most other hospital purposes and is less expensive.) (2)[A]
    • Reserved for more severe cases with stridor at rest
    • Racemic epinephrine: 0.05 mL/kg/dose (max 0.5 mL) of 2.25% solution nebulized in normal saline to total volume of 3 mL
    • L-epinephrine: 0.5 mL/kg/dose (max 5 mL) of a 1:1,000 dilution nebulized
    • Onset in 1 to 5 minutes, duration of 2 hours
    • Repeat as necessary if side effects are tolerated.
    • Observe child for 2 hours to ensure no recurrence after epinephrine wears off.
  • Corticosteroids
    • Dexamethasone (least expensive, easiest), 0.15-0.6 mg/kg; higher doses have been traditional care, but studies have shown 0.15 mg/kg has equal efficacy (3)[B]. Single dose; IV/IM/PO has proven equal efficacy.
    • Randomized controlled trials show this begins to improve symptoms within 30 minutes (4)[A]; full effect by 4 hours
    • Other steroids (betamethasone, budesonide (5)[A], prednisolone) are beneficial; there may be minimal superiority of dexamethasone; also, dexamethasone carries benefit of single-dose administration (6,7)[A].
  • Heliox: a helium-oxygen mixture
    • Smaller, lower mass helium molecule (compared with nitrogen) theoretically maintains laminar flow in narrower airways and serves as bridge therapy to steroids.
    • Minimum of 60% helium must be used; 70% is preferable; 79% if patient has no oxygen requirement.
    • Limited data; a Cochrane review found no benefit in mild cases but likely benefit in moderate to severe croup as a bridge to steroid effect (8)[A].
  • Antibiotics are not indicated in this viral illness.
    • Antecedent or subsequent bacterial infection is possible but uncommon.
  • Oxygen as needed
  • Humidified air shows no clinical benefit.

Second Line
Oseltamivir for influenza A �

SURGERY


  • Intubation rarely is required; tube 0.5 to 1 mm smaller than normal.
    • After trial of medical management, intubation is for fatigue caused by work of breathing or beginning total obstruction; not secondary to low oxygen saturation
    • Extubate in 3 to 5 days when there is an appropriate air leak around the endotracheal tube.
  • Tracheotomy: rarely; maintenance 3 to 7 days

COMPLEMENTARY & ALTERNATIVE MEDICINE


  • Although proven no benefit in hospital, may be helpful at home. Do not use high-temperature misters (e.g., teakettles) because of a risk of burns. A hot shower running in a bathroom is a good steam generator.
  • Some children respond well to cold, dry air.
  • Probiotics may decrease the incidence of upper respiratory tract infections (9)[A].

INPATIENT CONSIDERATIONS


Admission Criteria/Initial Stabilization
  • Minor cases need no visit to a hospital or primary care physician (PCP).
  • Outpatient care in mild cases
  • Admit patients who do not respond to therapy or have recurrent stridor at rest after epinephrine wears off. Also admit those who have oxygen requirement, pneumonia, or congestive heart failure.
  • In most cases, observation in the ED after medical management is sufficient.

Discharge Criteria
  • >2 hours since last epinephrine
  • No stridor at rest, no difficulty breathing
  • Child able to tolerate liquids PO
  • No underlying medical condition
  • Caretakers able to assess changes to clinical picture and reaccess medical care

ONGOING CARE


FOLLOW-UP RECOMMENDATIONS


Patient Monitoring
Most patients will be seen in an ED or PCP office setting. �

DIET


  • NPO and IV fluids for severe cases
  • Frequent small feedings with increased fluids for mild cases

PATIENT EDUCATION


  • Must keep the patient quiet; crying may exacerbate symptoms.
  • Educate parents about when to seek emergency care if mild cases progress.
  • Provide emotional support and reassurance for the patient.

PROGNOSIS


  • Up to ⅓; of patients will have a recurrence.
  • Recovery is usually full and without lasting effects.
  • If multiple recurrences, consider referral to ENT specialist to evaluate for possible anatomic etiology.

COMPLICATIONS


  • Rare
  • Subglottic stenosis in intubated patients
  • Bacterial tracheitis
  • Cardiopulmonary arrest
  • Pneumonia

REFERENCES


11 Tibballs �J, Watson �T. Symptoms and signs differentiating croup and epiglottitis. J Paediatr Child Health.  2011;47(3):77-82.22 Bjornson �C, Russell �KF, Vandermeer �B, et al. Nebulized epinephrine for croup in children. Cochrane Database Syst Rev.  2013;10:CD006619.33 Dobrovoljac �M, Geelhoed �GC. 27 years of croup: an update highlighting the effectiveness of 0.15 mg/kg of dexamethasone. Emerg Med Australas.  2009;21(4):309-314.44 Dobrovoljac �M, Geelhoed �GC. How fast does oral dexamethasone work in mild to moderately severe croup? A randomized double-blinded clinical trial. Emerg Med Australas.  2012;24(1):79-85.55 Cetinkaya �F, T �fek �i �BS, Kutluk �G. A comparison of nebulized budesonide, and intramuscular, and oral dexamethasone for treatment of croup. Int J Pediatr Otorhinolaryngol.  2004;68(4):453-456.66 Russell �KF, Liang �Y, O'Gorman �K, et al. Glucocorticoids for croup. Cochrane Database Syst Rev.  2011;(1):CD001955.77 Garbutt �JM, Conlon �B, Sterkel �R, et al. The comparative effectiveness of prednisolone and dexamethasone for children with croup: a community-based randomized trial. Clin Pediatr (Phila).  2013;52(11):1014-1021.88 Moraa �I, Sturman �N, McGuire �T, et al. Heliox for croup in children. Cochrane Database Syst Rev.  2013;(12):CD006822.99 Hao �Q, Lu �Z, Dong �BR, et al. Probiotics for preventing acute upper respiratory tract infections. Cochrane Database Syst Rev.  2011;(9):CD006895.

ADDITIONAL READING


  • Bjornson �CL, Johnson �DW. Croup. Lancet.  2008;371(9609):329-339.
  • Cherry �JD. Clinical practice. Croup. N Engl J Med.  2008;358(4):384-391.

SEE ALSO


Bronchiolitis; Epiglottitis; Tracheitis, Bacterial �

CODES


ICD10


  • J05.0 Acute obstructive laryngitis [croup]
  • J20.9 Acute bronchitis, unspecified
  • J38.5 Laryngeal spasm
  • J04.2 Acute laryngotracheitis

ICD9


  • 464.4 Croup
  • 466.0 Acute bronchitis
  • 478.75 Laryngeal spasm
  • 464.20 Acute laryngotracheitis without mention of obstruction

SNOMED


  • 71186008 Croup (disorder)
  • 85915003 Laryngotracheobronchitis
  • 406444002 Laryngeal spasm (disorder)
  • 55130001 Laryngotracheitis (disorder)

CLINICAL PEARLS


  • Parainfluenza virus is the most common pathogen but caused by many viruses.
  • Nebulized epinephrine only for stridor at rest
  • Dexamethasone 0.6 mg/kg PO � 1
  • Lateral neck films show funnel-shaped subglottic region with normal epiglottis: "steeple,"� "hourglass,"� or "pencil point"� sign (present in 40-60% of children with LTB).
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