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Congenital Megacolon (Hirschsprung Disease)

para>Some infants have only mild or intermittent constipation with intervening bouts of diarrhea. These patients typically have aganglionosis of a short segment of distal rectum. These cases may not be diagnosed until later in infancy or childhood.  

EPIDEMIOLOGY


  • Predominant age: infancy
  • Predominant sex: males > females for short segment (4:1); males > females for long segment (5:4)

Incidence
  • 1/2,000 to 5,000 births in the United States (white 91%, black 8%, Asian 0.5%)
  • 2.8/10,000 live births in Asians (1)[B]

ETIOLOGY AND PATHOPHYSIOLOGY


  • Congenital absence of Auerbach and Meissner autonomic plexuses in bowel wall (usually limited to the colon)
  • Aganglionosis results in the lack of propagation of propulsive waves of muscle contraction and absence of relaxation of the internal anal sphincter.
  • Obstruction may begin at anus and extend proximally to varying portions of the colon and small bowel.

Genetics
  • Familial inheritance: 50 times base rate
  • Associated with Down syndrome
  • Dominant mutations in the RET gene are found in 50% of familial patients with Hirschsprung disease and in 15-35% of patients with sporadic Hirschsprung disease (10q11.2 locus) (1)[B].

RISK FACTORS


  • Family history of Hirschsprung disease
  • Offspring risk if parent has short segment is 2%; if parent has long segment, risk is up to 50%.
  • Sibling risk if male affected: Female has 0.6% risk (short segment).
  • Sibling risk if female affected: Male has 18% risk (long segment).

COMMONLY ASSOCIATED CONDITIONS


  • Associated neurologic, cardiovascular, urologic, and gastrointestinal (GI) anomalies are found in 11-30% of patients (2)[C].
  • Secondary aganglionic megacolon may be a late complication of Chagas disease.
  • Down syndrome (four times expected incidence of Down syndrome in the normal population)
  • Cardiovascular defects (6%)
  • Urogenital anomalies (11%)
  • Dandy-Walker; Waardenburg; Ondine; Sipple syndromes
  • Cleft palate
  • Congenital small or large bowel atresia
  • Multiple endocrine neoplasia (MEN)-2B syndrome
  • Prematurity

DIAGNOSIS


HISTORY


  • Onset early in infancy; Newborn fails to pass meconium in 24 to 48 hours after birth (60-90% of infants with Hirschsprung disease).
  • Obstipation
  • May have diarrhea (usually intermittent)
  • Constipation (should be thoroughly investigated in any child if present since birth)
    • Diagnosis missed in older children and adults because it is considered a neonatal disease.
  • Poor feeding habits
  • Lack of physiologic urge to defecate (older children)
  • Vomiting: bilious vomiting in 19-37% of children
  • Abdominal distension
  • 5-44% of children present with Hirschsprung-associated enterocolitis (3)[B]:
    • Foul-smelling diarrhea, fever, and abdominal distension
    • May progress to fatal toxic megacolon if untreated

PHYSICAL EXAM


  • Early infancy
    • Marked abdominal distention (63-91% of neonates with Hirschsprung)
    • Visible colonic peristalsis
    • Palpable fecal mass
    • Growth retardation (possible)
  • Older infants
    • Failure to thrive
    • Empty rectum on digital examination
    • Palpable colon
    • Visible peristalsis
  • Explosive diarrhea with rectal exam
  • 5-44% of children may present with Hirschsprung-associated enterocolitis (3)[B].

DIFFERENTIAL DIAGNOSIS


  • Secondary megacolon (e.g., Chagas disease)
  • Functional/acquired megacolon
  • Functional constipation
  • Hypoganglionosis
  • Meconium plug syndrome
  • Small left colon syndrome
  • Meconium ileus
  • Neuronal intestinal dysplasia

DIAGNOSTIC TESTS & INTERPRETATION


Initial Tests (lab, imaging)
  • Serum electrolytes
  • Albumin
  • CBC
  • Thyroid function
  • Plain abdominal radiograph
    • Large ovoid fecal mass mottled by small, irregular gas shadows
    • Fluid levels within proximal bowel loops
    • Diaphragm may be elevated.
  • Contrast enema, water soluble (unprepped): Identification of transition zone is useful in planning definitive operative procedure (4)[B]
    • Dilation of sigmoid colon above narrowed distal sigmoid or rectum
    • Narrowed portion of colon rippled or segmented
    • Typical transition zone between narrowed distal colon and dilated proximal bowel
    • Enema may be therapeutic for other diagnoses (meconium ileus/plug, small left colon syndrome).

Diagnostic Procedures/Other
  • Suction aspiration biopsy of bowel wall (2 cm above dentate line): can be done without anesthesia at bedside (absence of ganglia in wall of narrowed rectum)
  • Full-thickness biopsy may be required if suction biopsy is equivocal (most commonly needed in older infants/children who have thicker mucosa).
  • Proctosigmoidoscopy: ampulla empty of feces (not often of value without biopsy)
  • Laparoscopy: normal proximal colon dilated (not helpful without biopsy)
  • Anorectal manometry: high baseline resting pressure and absent rectoanal inhibitory reflex (3)[B]
    • Rectosphincteric reflex may not be present until after 14 days of age.

Test Interpretation
  • Dilatation and hypertrophy of all layers proximal to involved colon (transition zone represents the transition from narrowed distal colon to dilated proximal bowel)
  • Aganglionosis of involved bowel with submucosal hypertrophied nerve bundles on biopsy
  • Calretinin and acetylcholinesterase stains may be helpful.

TREATMENT


GENERAL MEASURES


  • Treatment may be symptomatic or definitive.
  • Correct fluid and electrolyte imbalances.
  • Abdominal decompression with a nasogastric tube
  • Removal of fecal accumulation: retention enemas of 3 to 4 oz (90 to 120 mL) of mineral oil followed by repeated colonic irrigations with isotonic saline solution. Avoid use of other solutions (e.g., water, soap-sud enemas).
  • Manual disimpaction for short-segment disease may be necessary.

MEDICATION


First Line
  • Broad-spectrum IV antibiotics for patients presenting with enterocolitis (4)[A]
  • Bowel prep prior to surgery

Second Line
Metronidazole (Flagyl) for bowel preparation  

SURGERY/OTHER PROCEDURES


  • Inpatient surgery
    • Proximal colostomy and resection of aganglionic bowel is the gold standard (necessary when there is significant proximal dilatation).
    • Definitive pull-through procedure (Duhamel, Soave, or Swenson) when dilatation has resolved (usually performed at 3 to 6 months) (2)[C].
    • Single-stage procedure may be possible in infants.
    • Transanal endorectal pull-through in infants if proximal bowel not too dilated (5)[C]; usually done without a protective colostomy.
    • Transanal endorectal pull-through is associated with fewer complications and fewer episodes of enterocolitis when compared to transabdominal pull-through procedures (6,7)[A]:
      • Laparoscopic technique may be used alone or combined with transanal endorectal pull-through.
  • Confirmation of normal ganglion cells mandatory at colostomy and proximal resection sites prior to anastomosis
  • Total-colon Hirschsprung disease may require other procedures, including Martin modification of Duhamel pull-through (5,8)[C]:
    • Cecal patches may be used to facilitate water absorption in pulled-through small bowel.
  • Total-bowel Hirschsprung disease (68 reported cases in world literature through 2009) (9)[B]
    • Family history of Hirschsprung disease in 14.7%, RET gene mutation was identified in 71.4%, associated anomalies were found in 20.6%; 66.2% died before age 8 years
    • Ultimately requires small bowel transplantation.
    • Long (30 to 40 cm) myomectomy of proximal bowel and creation of proximal small bowel stoma (at 30 to 40 cm from ligament of Treitz) may allow small-volume feeding in some infants.

INPATIENT CONSIDERATIONS


Admission Criteria/Initial Stabilization
Early workup (ambulatory or in hospital, depending on the patient's condition)  

ONGOING CARE


FOLLOW-UP RECOMMENDATIONS


Patient Monitoring
  • Postsurgical monitoring for fluid/electrolyte balance, protein-calorie nutrition and growth
  • Long-term monitoring for continence and constipation

DIET


  • Diet does not control obstipation.
  • Postoperative diet: standard for age

PATIENT EDUCATION


  • After surgery, instruct parents to detect and report dehydration, decreased urinary output, sunken eyes, poor skin turgor, vomiting, or fever.
  • Parental bonding through maximal participation in their child's care, including colostomy care
  • Request enterostomal therapist to teach family appropriate colostomy care.
  • Daily anorectal dilations are frequently required following Soave or transanal endorectal pull-through procedures to prevent anastomotic stricture:
    • May be required for several months

PROGNOSIS


  • Favorable
    • Mortality decreased over last 25 years, now estimated at ≤3% (2)[C]
  • Requires long-term follow-up.
  • Aggressive management of suspected enterocolitis:
    • Rectal irrigation
    • IV antibiotics
    • Nasogastric decompression

COMPLICATIONS


  • Toxic enterocolitis, possibly fatal
    • The reported mortality from enterocolitis has decreased to <1% over the last 20 years (10)[B].
    • Enterocolitis may occur before or after definitive pull-through:
      • 5-42% of postoperative patients
      • The rate of enterocolitis may be less after transanal pull-through compared with other procedures (10)[B].
    • Enterocolitis may be rapidly progressive.
    • Diagnostic features are abdominal distension, lethargy, bilious emesis, and diarrhea.
    • Treated with nasogastric decompression, IV hydration, IV antibiotics, and colonic washouts
  • Bleeding and/or perforation
  • Anal stenosis at anastomotic site (0-35%)
    • Stricture is associated with higher risk of postoperative enterocolitis (3)[B].
  • Anastomotic leak in 1-10%
  • Cuff abscess in 5%
  • Perineal excoriation is common (usually improves within 2 to 3 months).
  • Wound infection in 4% and dehiscence in 1%
  • Fecal incontinence in 1-39% (3)[B]
  • Enuresis in 5-26%
  • Late mortality in 0.7-5% (3)[B]
  • Obstructive complications (9)[C]
    • Mechanical obstruction from twisting of pulled-through bowel and intra-abdominal adhesions
    • Secondary aganglionosis related to ischemia of pulled-through bowel
    • Motility disorder involving the proximal bowel (containing ganglion cells)
    • Internal sphincter achalasia (all children with Hirschsprung disease lack the normal rectoanal inhibitory reflex): The inability to relax the internal anal sphincter which typically improves over time.

REFERENCES


11 Tam  PK, Garcia-Barcel ³  M. Genetic basis of Hirschsprung's disease. Pediatr Surg Int.  2009;25(7):543-558.22 Suita  S, Taguchi  T, Ieiri  S, et al. Hirschsprung's disease in Japan: analysis of 3852 patients based on a nationwide survey in 30 years. J Pediatr Surg.  2005;40(1):197-201; discussion 201-202.33 Haricharan  RN, Georgeson  KE. Hirschsprung disease. Semin Pediatr Surg.  2008;17(4):266-275.44 Langer  JC. Hirschsprung disease. Curr Opin Pediatr.  2013;25(3):368-374.55 Wildhaber  BE, Teitelbaum  DH, Coran  AG. Total colonic Hirschsprung's disease: a 28-year experience. J Pediatr Surg.  2005;40(1):203-206; discussion 206-207.66 Kim  AC, Langer  JC, Pastor  AC, et al. Endorectal pull-through for Hirschsprung's disease-a multicenter, long-term comparison of results: transanal vs transabdominal approach. J Pediatr Surg.  2010;45(6):1213-1220.77 Gosemann  JH, Friedmacher  F, Ure  B, et al. Open versus transanal pull-through for Hirschsprung disease: a systematic review of long-term outcome. Eur J Pediatr Surg.  2013;23(2):94-102.88 Laughlin  DM, Friedmacher  F, Puri  P. Total colonic aganglionosis: a systematic review and meta-analysis of long-term clinical outcome. Pediatr Surg Int.  2012;28(8):773-779.99 Ruttenstock  E, Puri  P. A meta-analysis of clinical outcome in patients with total intestinal aganglionosis. Pediatr Surg Int.  2009;25(10):833-839.1010 Ruttenstock  E, Puri  P. Systematic review and meta-analysis of enterocolitis after one-stage transanal pull-through procedure for Hirschsprung's disease. Pediatr Surg Int.  2010;26(11):1101-1105.

ADDITIONAL READING


De La Torre  L, Langer  JC. Transanal endorectal pull-through for Hirschsprung disease: technique, controversies, pearls, pitfalls, and an organized approach to the management of postoperative obstructive symptoms. Semin Pediatr Surg.  2010;19(2):96-106.  

SEE ALSO


Constipation  

CODES


ICD10


Q43.1 Hirschsprungs disease  

ICD9


751.3 Hirschsprungs disease and other congenital functional disorders of colon  

SNOMED


  • Hirschsprungs disease (disorder)
  • Congenital dilatation of colon (disorder)
  • Long segment Hirschsprung's disease (disorder)
  • Short segment Hirschsprung's disease (disorder)

CLINICAL PEARLS


  • All infants with primary constipation must be evaluated for Hirschsprung disease.
  • Early diagnosis helps prevent toxic enterocolitis.
  • An unprepped barium enema followed by a suction rectal biopsy confirms the diagnosis of Hirschsprung disease.
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