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Central Retinal Artery Occlusion, Emergency Medicine


Basics


Description


  • Obstruction of the central retinal artery associated with sudden painless loss of vision
  • Usually occurs in persons 50-70 yr of age
  • Ophthalmic artery is 1st branch of carotid.
  • Risk factors include HTN, atherosclerotic disease, sickle cell disease, vasculitis, valvular heart disease, lupus, trauma, and coronary artery disease.
  • Incidence of 1-10/100,000
  • Often described as a "stroke of the eye"�

Etiology


  • Embolic:
    • Occlusion by intravascular material from a proximal source:
      • Atherosclerotic disease (majority)
      • Carotid artery stenosis
      • Valvular heart disease (cardiogenic emboli)
      • Atrial myxoma
      • Dissection of the ophthalmic artery
      • Carotid artery dissection
  • Thrombotic:
    • Obstruction of flow from the rupture of a pre-existing intravascular atherosclerotic plaque
    • Hypercoagulable states (sickle cell)
  • Inflammatory:
    • Due to temporal arteritis, lupus, vasculitis
  • Arterial spasm:
    • Associated with migraine headaches
  • Decreased perfusion:
    • Low-flow conditions such as in severe hypotension or high-pressure situations seen in acute angle-closure glaucoma or retrobulbar hemorrhage

Diagnosis


Signs and Symptoms


History
  • Sudden, painless, monocular loss of vision
  • Prior episodes of sudden visual loss:
    • May last a few seconds to minutes (amaurosis fugax)
    • Caused by transient embolic phenomena or decreased ocular blood flow

Physical Exam
  • Significantly decreased visual acuity
  • Afferent pupillary defect usually present
  • Retinal appearance:
    • Emboli visualized within vascular tree of the retina
    • Appears as glinting white or yellow flecks (Hollenhorst plaques) within the vessels
    • Ischemic edema visible within 15-20 min of occlusion
    • "Cherry-red spot"� remains over the fovea (only area where there is very thin retina allowing the vascular choroids to show through).
    • Affected arteries empty or showing dark red stationary or barely pulsatile segmented rouleaux ("box-carring"�)
    • Within 1-2 hr opacification of the usually transparent infarcting retinal nerve layer occurs.
  • Partial field deficits:
    • Occur only if branch of central retinal artery involved

Essential Workup


  • Visual acuity and visual field testing
  • Fundoscopic exam
  • Intraocular pressure measurements
  • Emergent ophthalmologic consultation

Diagnosis Tests & Interpretation


Lab
Directed toward evaluating underlying etiology of occlusion: �
  • CBC with differential and platelet count
  • PT/PTT
  • Electrolytes, BUN/creatinine, glucose
  • Electronic spin resonance for giant cell arteritis (in patients >55 yr old)
  • ANA, RF, CRP, ESR
  • Rapid plasma reagin (RPR)
  • Hemoglobin electrophoresis
  • Serum protein electrophoresis

Imaging
Directed toward evaluating underlying etiology of occlusion: �
  • Carotid artery ultrasound/Doppler
  • Possibly echocardiography
  • Fluorescein angiography or electroretinography to confirm the diagnosis

Differential Diagnosis


  • Acute angle-closure glaucoma
  • Central retinal vein occlusion
  • Giant cell arteritis (temporal arteritis)
  • Optic neuritis
  • Retinal detachment

Treatment


Initiate treatment immediately because irreversible visual loss occurs at 90 min: �
  • Only immediate treatment may help to salvage or restore sight to the affected eye.
  • Goals of therapy include dislodging or dissolving the embolus, arterial dilation to improve forward flow, and reduction of intraocular pressure to improve the perfusion gradient.

Ed Treatment/Procedures


  • Immediate global massage in an attempt to dislodge the embolus:
    • Lay patient flat and apply digital global massage bolus.
    • On closed eyelid, apply constant pressure for 15 sec and remove for 15 sec. Repeat for 5 cycles.
  • Initiate high-flow oxygen via 100% nonrebreather:
    • Consider transfer to a facility capable of providing hyperbaric oxygen (HBO) if <24 hr from symptom onset
    • May use inhaled carbogen (a mixture of carbon dioxide and oxygen gas) if available
  • Administer IV acetazolamide to decrease intraocular pressure.
  • Apply topical timolol maleate to reduce intraocular pressure.
  • Administer aspirin and IV heparin for prevention of clot propagation.
  • Obtain emergent ophthalmology consultation for:
    • Anterior chamber paracentesis to help reduce intraocular pressure
    • Possible intra-arterial fibrinolysis for clot lysis
  • Administer high-dose systemic steroids in suspected cases of inflammatory arteritis.

Medication


First Line
  • Acetazolamide: 500 mg IV or PO
  • Carbogen: Inhalation of 95% oxygen and 5% carbon dioxide mixture
  • Heparin: 80 U/kg IV bolus then 18 U/kg/h continuous infusions (rate adjusted based on PTT level)
  • Timolol maleate 0.5% solution: 1 drop topically to affected eye

Second Line
  • Methylprednisolone: 250 mg IV in suspected cases of inflammatory arteritis
  • Aspirin: 325 mg PO
  • Mannitol
  • Sublingual nitroglycerin

Follow-Up


Disposition


Admission Criteria
Required for workup of proximal cause in acute cases (source of embolism, thrombosis, or inflammatory) �
Discharge Criteria
Chronic retinal artery occlusion with no evidence of active disease can be worked up as an outpatient. �
Issues for Referral
All suspected cases warrant emergent ophthalmology consultation. �

Followup Recommendations


Most cases will require carotid ultrasound to exclude atherosclerotic disease. �

Pearls and Pitfalls


  • Amaurosis fugax (transient, possibly resolved retinal artery occlusion) is a sentinel event and may lead to complete occlusion or stroke. Do not ignore these symptoms and urgent workup is required.
  • Retinal artery occlusion is a medical emergency requiring immediate treatment to prevent loss of the eye.
  • It is important to document a full eye exam including visual acuity and evaluation of the optic fundus.

Additional Reading


  • Arnold �M, Koerner �U, Remonda �L, et al. Comparison of intra-arterial thrombolysis with conventional treatment in patients with acute central retinal artery occlusion. J Neurol Neurosurg Psychiatry.  2005;76(2):196-199.
  • Fraser �SG, Adams �W. Interventions for acute non-arteritic central retinal artery occlusion. Cochrane Database Syst Rev.  2009;(1):CD001989.
  • Hazin �R, Daoud �YJ, Khan �F. Ocular ischemic syndrome: Recent trends in medical management. Curr Opin Ophthalmol.  2009;20:430-433.
  • Murphy-Lavoie �H, Butler �F, Hagan �C. Central retinal artery occlusion treated with oxygen: a literature review and treatment algorithm. Undersea Hyperb Med.  2012;39(5):943-953.
  • Vortmann �M, Schneider �JL. Acute monocular visual loss. Emerg Med Clin North Am.  2008;26(1):73-96.

See Also (Topic, Algorithm, Electronic Media Element)


  • Central Retinal Venous Occlusion
  • Visual Loss

Codes


ICD9


362.31 Central retinal artery occlusion �

ICD10


  • H34.10 Central retinal artery occlusion, unspecified eye
  • H34.11 Central retinal artery occlusion, right eye
  • H34.12 Central retinal artery occlusion, left eye
  • H34.13 Central retinal artery occlusion, bilateral
  • H34.1 Central retinal artery occlusion

SNOMED


  • 38742007 Central retinal artery occlusion
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