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Caustic Ingestion, Emergency Medicine


Basics


Description


  • Alkalis:
    • Dissociate in the presence of H2O to produce hydroxy (OH-) ions, which leads to liquefaction necrosis
    • Postingestion-mainly damages the esophagus:
      • Gastric damage can occur (see "Acids"Ł).
    • Esophageal damage (in the order of increasing damage) consists of:
      • Superficial hyperemia
      • Mucosal edema
      • Superficial blisters
      • Exudative ulcerations
      • Full-thickness necrosis
      • Perforation
      • Fibrosis with resulting esophageal strictures
    • Do not directly produce systemic complications.
  • Acids:
    • Dissociate in the presence of H2O to produce hydrogen (H+) ions, which leads to a coagulation necrosis with eschar formation
    • Postingestion-damages the stomach because of rapid transit time through esophagus:
      • Esophageal damage can occur (see "Alkalis"Ł).
    • Gastric damage (in the order of increasing damage) consists of:
      • Edema
      • Inflammation
      • Immediate or delayed hemorrhage
      • Full-thickness necrosis
      • Perforation
      • Fibrosis with resulting gastric outlet obstruction
    • Well-absorbed and can cause hemolysis of RBCs and a systemic metabolic acidosis

Etiology


  • Direct chemical injuries
  • Injuries occur secondary to acid and alkali exposures.
  • Many caustic agents (acids and alkalis) are found in common household and industrial products.
  • Caustic substances:
    • Ammonia hydroxide
  • Glass cleaners:
    • Formaldehyde:
      • Embalming agent
    • Hydrochloric acid:
      • Toilet bowel cleaners
    • Hydrofluoric acid:
      • Glass etching industry
      • Microchip industry
      • Rust removers
    • Iodine:
      • Antiseptics
    • Phenol:
      • Antiseptics
    • Sodium hydroxide:
      • Drain cleaners
      • Drain openers
      • Oven cleaners
    • Sodium borates, carbonates, phosphates, and silicates:
      • Detergents
      • Dishwasher preparations
      • Sodium hypochlorite
      • Bleaches
    • Sulfuric acid:
      • Car batteries
      • Button batteries

Diagnosis


Signs and Symptoms


  • Oropharyngeal:
    • Pain
    • Erythema
    • Burns
    • Erosions
    • Ulcers
    • Drooling
    • Hoarseness
    • Stridor
    • Aphonia
    • Absence of visible lesions in the oropharynx does not exclude visceral injuries.
  • Pulmonary:
    • Tachypnea
    • Cough
    • Pneumonitis if aspirated
  • GI:
    • Pain
    • Emesis or hematemesis
    • Melena, dysphagia
    • Odynophagia
    • Esophageal or gastric perforation
    • Peritonitis owing to perforation
  • Cardiovascular:
    • Tachycardia
    • Hypotension
    • Orthostatic changes
  • Hematologic:
    • Acid ingestion can cause RBC hemolysis.
  • Dermatologic:
    • Pain
    • Erythema
    • 1st-, 2nd-, or 3rd-degree burns
  • Ocular:
    • Pain
    • Erythema
    • Injection
    • Corneal burns
    • Full-thickness corneal damage
  • Metabolic:
    • Metabolic acidosis

Essential Workup


  • History of or signs and symptoms of an exposure
  • Absence of oropharyngeal lesions does not exclude visceral injury.

Diagnosis Tests & Interpretation


Lab
  • CBC
  • Electrolytes, BUN, creatinine, glucose
  • Arterial blood gas
  • Blood cultures:
    • If mediastinitis or peritonitis suspected
  • Type and cross-match

Imaging
Chest and abdominal radiographs for: á
  • Esophageal or gastric perforation

Diagnostic Procedures/Surgery
  • Esophageal and gastric endoscopy:
    • For symptomatic patients to determine the extent of injury
    • Perform within the 1st 12-24 hr after ingestion.
    • Not recommended in the presence of respiratory distress without proper airway management
    • Not recommended in the presence of severe pharyngeal damage
  • Radiographic oral contrast imaging not recommended acutely:
    • May be used in follow-up for assessment for strictures

Differential Diagnosis


  • Chemical injuries from corrosives, acids, alkalis, desiccants, vesicants, and oxidizing and reducing agents
  • Foreign body ingestion
  • Upper airway infection or angioedema

Treatment


Pre-Hospital


  • For oral burns or symptoms: Rinse mouth liberally with water or milk.
  • Water or milk can be given to following patients:
    • Able to drink
    • Not complaining of significant abdominal pain
    • Do not have airway compromise or vomiting
  • Copious irrigation for ocular or dermal exposure

Initial Stabilization/Therapy


  • ABCs:
    • Prophylactic intubation if there is any evidence of respiratory compromise
    • Blind nasotracheal intubation contraindicated
  • Treat hypotension with 0.9% NS IV fluid resuscitation.

Ed Treatment/Procedures


  • Decontamination:
    • Dermal or ocular exposure:
      • Immediate and thorough irrigation with water or 0.9% NS until physiologic pH attained
      • Alkalis typically require more irrigation than acids.
    • Ipecac, activated charcoal, gastroesophageal lavage (large-bore or an NG tube), and a neutralizing acid or base are all contraindicated with caustic ingestions.
  • Dilution:
    • Water or milk in the 1st 30 min of ingestion:
      • Especially useful for solid caustic alkali ingestions
      • Excessive intake may induce vomiting and worsen esophageal damage.
    • If respiratory distress, intubate before dilution.
    • Contraindicated if esophageal or gastric perforation suspected
  • Keep patient NPO if oral exposure.
  • Broad-spectrum antibiotics if mediastinitis or peritonitis suspected
  • Antiemetics for nausea and vomiting
  • Treat dermal exposures according to standard burn recommendations.
  • Detailed exam for ocular exposures
  • IV proton pump inhibitors or H2 blockers for symptomatic relief
  • Gastroenterology and surgical consultation
  • Benefit of corticosteroids following esophageal damage is controversial:
    • May prevent the formation of esophageal stricture
    • May promote bacterial invasion, immune suppression, and tissue softening
    • The decision to initiate corticosteroids requires input from entire team caring for patient.
    • Initiate broad-spectrum antibiotics if corticosteroids are given.
  • Laparoscopy or laparotomy for perforation and full-thickness necrosis
  • Topical hydrofluoric acid exposure (options depend on severity and location):
    • IM injection of 5% calcium gluconate (0.5 mL/cm2 of skin with 30G needle)
    • Intra-arterial infusion of 10 mL of 10% calcium gluconate in 40 mL D5W over 4 hr

Medication


  • Methylprednisolone: 40 mg q8h IV (peds: 2 mg/kg/d IV); the course of therapy is 14-21 days followed by a corticosteroid taper.
  • Ondansetron: 4 mg (peds: 0.1-0.15 mg/kg) IV
  • Pantoprazole: 40 mg IV
  • Prochlorperazine (Compazine): 5-10 mg IV (peds: 0.13 mg/kg per dose IM)
  • Ranitidine (Zantac): 50 mg IV q6-8h

Follow-Up


Disposition


Admission Criteria
  • All symptomatic patients
  • Nonaccidental ingestion

Discharge Criteria
  • Asymptomatic patients who accidentally ingested and are able to swallow without difficulty
  • Minimal oropharyngeal pain with a corresponding visible lesion; no drooling; no respiratory compromise; no deep throat, chest, or abdominal pain; and able to swallow without difficulty

Followup Recommendations


Psychiatric referral for intentional ingestion á

Pearls and Pitfalls


  • Dilute with milk or water at home or in the ED within the 1st 30 min.
  • Perform copious irrigation of ocular or dermal exposure:
    • Alkalis require more irrigation than acids.

Additional Reading


  • Lupa áM, Magne áJ, Guarisco áL, et al. Update on the diagnosis and treatment of caustic ingestions. Ochsner J.  2009;9:54-59.
  • Riffat áF, Cheng áA. Pediatric caustic ingestion: 50 consecutive cases and a review of the literature. Dis Esophagus.  2009;22:89-94.
  • Salzman áM, O'Malley áRN. Updates on the evaluation and management of caustic exposures. Emerg Med Clin North Am.  2007;25(2):459-476.

Codes


ICD9


  • 947.0 Burn of mouth and pharynx
  • 947.2 Burn of esophagus
  • 947.3 Burn of gastrointestinal tract
  • 947.1 Burn of larynx, trachea, and lung
  • 947.8 Burn of other specified sites of internal organs
  • 947.9 Burn of internal organs, unspecified site

ICD10


  • T28.5XXA Corrosion of mouth and pharynx, initial encounter
  • T28.6XXA Corrosion of esophagus, initial encounter
  • T28.7XXA Corrosion of other parts of alimentary tract, init encntr
  • T27.5XXA Corrosion involving larynx and trachea w lung, init encntr
  • T28.90XA Corrosions of unspecified internal organs, initial encounter
  • T28.99XA Corrosions of other internal organs, initial encounter

SNOMED


  • 23509002 Caustic esophageal injury (disorder)
  • 37693008 Caustic injury gastritis (disorder)
  • 235021001 Chemical burn of oral mucosa (disorder)
  • 212039008 Corrosion involving larynx and trachea with lung (disorder)
  • 219171000 Suicide and selfinflicted injury by caustic substances, excluding poisoning (navigational concept)
  • 219362003 Injury undetermined whether accidentally or purposely inflicted, by caustic substances, excluding poisoning (disorder)
  • 269737002 Injury of unknown intent by local effect of caustic substance (disorder)
  • 418409002 Poisoning of undetermined intent by corrosive, acid or caustic alkali (disorder)
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