Home

helps physicians and healthcare professionals

Erectile Dysfunction

helps physicians and healthcare professionals

Doctor123.org

helps physicians and healthcare professionals

Brugada Syndrome, Emergency Medicine


Basics


Description


  • Inherited heart disease due to mutations of cardiac Na+ channels without structural abnormalities
  • Very high risk of sudden cardiac death in the form of ventricular fibrillation
  • 2-yr mortality ~30%
  • Suspected in 40-60% of what was previously known as idiopathic ventricular fibrillation
  • Higher prevalence in men of Southeast Asian descent, but all ages, genders, races can be affected

Etiology


  • Inherited:
    • Autosomal dominant in 50%
    • Variable penetration
  • Cardiac Na+ channel:
    • >70 described mutations
    • Variable penetrance
    • SCN5A mutations account for 20%

Diagnosis


Signs and Symptoms


  • Most commonly presents as episodes of sudden death (in ventricular fibrillation) or as syncope or near-syncope in self-terminating episodes of polymorphic ventricular tachycardia

History
  • HPI:
    • Episodes of syncope or near-syncope
    • Palpitations
    • Cardiac arrest
    • Concomitant illness, fever, metabolic, or electrolyte disorders
    • Cocaine use
    • TCA and psychotropic drugs
    • Nocturnal agonal respirations
  • Family history:
    • History of drowning due to syncope or dysrhythmias while submerged
    • History of early and/or sudden cardiac death
    • Known relatives with Brugada syndrome

Physical Exam
  • Complete physical exam, with special attention to other causes of syncope or dysrhythmia:
    • Abnormal heart sounds
    • Pectus excavatum (normal variant EKG changes)
    • Athletes
    • Pacemaker in situ

Essential Workup


  • A 12-lead EKG is imperative
  • Detailed HPI and family history
  • Toxicology screen

Diagnosis Tests & Interpretation


EKG Diagnostic Criteria
  • Basic:
    • Right bundle branch block (RBBB) or Incomplete right bundle branch block (IRBBB) with ST-segment elevation in the right precordial leads only
  • Morphology of QRS-T in V1-V3
    • ST-elevation
    • Sometimes only in V1 and very rarely in V3
  • Type 1 (coved pattern):
    • Initial ST-elevation ≥2 mm, slowly descending, concave with respect to the isoelectric line
    • Negative symmetric T-wave
    • No clear r"�
    • QRS duration mismatch between V1 and V6
  • Type 2 (saddle back pattern):
    • High r"� take-off is ≥2 mm with respect to the isoelectric line
    • Followed by ST-elevation - convex with respect to the isolectric line
    • QRS duration mismatch between V1 and V6

Lab
  • Serum:
    • Chemistries to rule out underlying electrolyte causes of dysrhythmia or syncope
    • Cardiac biomarkers (troponin, CK-MB) for ischemia
    • D-dimer in the appropriate population (Wells, PERC) if considering pulmonary embolism
    • CBC for evaluation of syncope

Imaging
  • CXR:
    • Evaluate for cardiomegaly
  • CT-angiogram of the chest:
    • If considering pulmonary embolism as a cause

Diagnostic Procedures/Surgery
  • Electrophysiology lab
    • Drug challenge with sodium channel blockers (type 1a and 1c)
  • AICD placement
    • Mortality reduced to 0% in this group

Differential Diagnosis


  • Syncope:
    • Primary cardiogenic
    • Vasovagal
    • Neurogenic
    • Hypovolemia
    • Pregnancy
  • Dysrhythmias:
    • Paroxysmal atrial fibrillation
    • Atrial fibrillation with rapid ventricular response
    • Wolff-Parkinson-White syndrome
    • Lown-Ganong-Levine syndrome
    • Ventricular tachycardia
    • Multifocal atrial tachycardia
    • Spontaneously terminating ventricular fibrillation
    • Symptomatic bradycardia
    • High-grade heart blocks
    • Long QT syndromes
    • Overdose especially TCA
  • EKG mimics:
    • Isolated RBBB
    • Athletes
    • Septal hypertrophy
    • Pectus excavatum
    • Arrhythmogenic right ventricular dysplasia
    • STEMI
  • Other systemic illness:
    • Electrolyte disturbances
    • Pericarditis
    • Myocarditis
    • Myopericarditis
    • Pulmonary embolism

Treatment


Pre-Hospital


  • Airway, breathing, and circulation management
  • ACLS protocol for arrest/dysrhythmias

Initial Stabilization/Therapy


  • Airway, breathing, and circulation management
  • Start or continue ACLS algorithms

Ed Treatment/Procedures


  • Cardiac monitoring at all times
  • Cardiology consult:
    • For electrophysiology evaluation
  • Defibrillator/pacing pads
  • Correct underlying disease processes:
    • Replete electrolytes
    • Correct metabolic derangements
    • Asymptomatic patients' management controversial. Current consensus states EP evaluation but not supported in literature.

Medication


  • ACLS medications per protocol
  • Antiarrhythmics usually not helpful

  • PALS/defibrillation
  • Appropriate weight-based medication and energy (joule) adjustments

Follow-Up


Disposition


Admission Criteria
  • EKG findings concerning for Brugada in the appropriate clinical setting
  • Unexplained syncope
  • Inability to obtain rapid cardiology follow-up
  • Ongoing dysrhythmias even if they are spontaneously terminating

Discharge Criteria
  • Hemodynamically stable
  • Asymptomatic
  • Cardiology clearance
  • Appropriate AICD intervention after an event
    • After interrogation of AICD

Follow-Up Recommendations


  • All patients with concerning EKG findings and history should be referred to EP for additional evaluation

Pearls and Pitfalls


  • Consider in any episodes of sudden cardiac death or syncope, especially in the setting of family history of the same
  • The EKG is diagnostic showing RBBB or IRBBB with ST-segment elevation in the right precordial leads only
  • Beware of EKG mimics which can have similar presentation - typically mimics will have concordant QRS duration in V1 and V6 whereas Brugada QRS changes should be isolated to V1-V3
  • Have a low threshold for cardiology consultation given high risk of death
  • AICD implantation is definitive treatment, almost eliminating risk of sudden cardiac death
  • Antiarrhythmic agents have not been found to be helpful
  • The Brugada pattern may be "unmasked"� in systemic illness, even if resolution of the EKG occurs, the patient should still have EP follow-up

Additional Reading


  • Bay �s de Luna �A, Brugada �J, Baranchuk �A, et al. Current electrocardiographic criteria for diagnosis of Brugada pattern: A consensus report. J Electrocardiol.  2012;45(5):433-442.
  • Brady �WJ. ST segment and T wave abnormalities not caused by acute coronary syndromes. Emerg Med Clin North Am.  2006;24(1):91-111, vi. Review.
  • Brugada �P, Brugada �J. Right bundle branch block, persistent ST segment elevation and sudden cardiac death: A distinct clinical and electrocardiographic syndrome. J Am Coll Cardiol.  1992;20:1391-1396.
  • Mattu �A, Rogers �RL, Kim �H, et al. The Brugada syndrome. Am J Emerg Med.  2003;21(2):146-151.

Codes


ICD9


746.89 Other specified congenital anomalies of heart �

ICD10


  • I49.8 Other specified cardiac arrhythmias
  • Q24.8 Other specified congenital malformations of heart

SNOMED


  • 418818005 brugada syndrome (disorder)
Copyright © 2016 - 2017
Doctor123.org | Disclaimer