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Bell Palsy

para>Steroids should be used cautiously during pregnancy; consult with an obstetrician.  

ISSUES FOR REFERRAL


Patients may need to be referred to an ear, nose, and throat specialist or a neurologist.  

ADDITIONAL THERAPIES


  • Physical therapy: strong evidence that physical therapy combined with drug treatment has positive effect on grade and time of recovery compared with drug treatment only (7,8 and 9)[A].
  • Electrostimulation and mirror biofeedback rehabilitation have limited evidence of effect (10)[C].
  • Acupuncture with strong stimulation has shown some therapeutic promise.
  • Routine use of eye-protective measures for patients with incomplete eye closure is recommended (6)[A].

SURGERY/OTHER PROCEDURES


  • Surgical treatment of Bell palsy remains controversial and is reserved for intractable cases.
  • There is insufficient evidence to decide whether surgical intervention is beneficial or harmful in the management of Bell palsy (11)[B].
  • In those cases where surgical intervention is performed, cranial nerve XII is surgically decompressed at the entrance to the meatal foramen where the labyrinthine segment and geniculate ganglion reside.
  • Decompression surgery should not be performed >14 days after the onset of paralysis because severe degeneration of the facial nerve is likely irreversible after 2 to 3 weeks.
  • A routine surgical decompression is not recommended (6)[B].

ONGOING CARE


FOLLOW-UP RECOMMENDATIONS


Patient Monitoring
  • Patients should start steroid treatment immediately and be followed for 12 months.
  • Patients who do not recover complete facial nerve function should be referred to an ophthalmologist for tarsorrhaphy.

PATIENT EDUCATION


American Academy of Family Physicians: http://www.aafp.org/afp/2007/1001/p1004.html  

PROGNOSIS


  • Most patients achieve complete spontaneous recovery within 2 weeks. >80% recover within 3 months.
  • 85% of untreated patients will experience the first signs of recovery within 3 weeks of onset.
  • 16% are left with a partial palsy, motor synkinesis, and autonomic synkinesis.
  • 5% experience severe sequelae, and a small number of patients experience permanent facial weakness and dysfunction.
  • Poor prognostic factors include the following:
    • Age >60 years
    • Complete facial weakness
    • Hypertension
    • Ramsay-Hunt syndrome
  • The Sunnybrook and House-Brackmann facial grading systems are clinical prognostic models that identify Bell palsy patients at risk for nonrecovery at 12 months.
  • Treatment with prednisolone or no prednisolone and the Sunnybrook score are significant factors for predicting nonrecovery at 1 month (12)[C].
  • Patients with no improvement or progression of symptoms should be referred to ENT (6)[A] and may require neuroimaging to rule out neoplasms (6)[A].

COMPLICATIONS


  • Corneal abrasion or ulceration
  • Steroid-induced psychological disturbances; avascular necrosis of the hips, knees, and/or shoulders
  • Steroid use can unmask subclinical infection (e.g., TB).

REFERENCES


11 Katz  A, Sergienko  R, Dior  U, et al. Bell's palsy during pregnancy: is it associated with adverse perinatal outcome? Laryngoscope.  2011;121(7):1395-1398.22 Peng  KP, Chen  YT, Fuh  JL, et al. Increased risk of Bell palsy in patients with migraine: a nationwide cohort study. Neurology.  2015;84(2):116-124.33 Thaera  GM, Wellik  KE, Barrs  DM, et al. Are corticosteroid and antiviral treatments effective for bell palsy? A critically appraised topic. Neurologist.  2010;16(12):138-140.44 Worster  A, Keim  SM, Sahsi  R, et al. Do either corticosteroids or antiviral agents reduce the risk of long-term facial paresis in patients with new-onset Bell's palsy? J Emerg Med.  2010;38(4):518-523.55 Madhok  V, Falk  G, Fahey  T, et al. Prescribe prednisolone alone for Bell's palsy diagnosed within 72 hours of symptom onset. BMJ.  2009;338:b255.66 de Almeida  JR, Guyatt  GH, Sud  S, et al. Management of Bell palsy: clinical practice guideline. CMAJ.  2014;186(12):917-922.77 Teixeira  LJ, Valbuza  JS, Prado  GF. Physical therapy for Bell's palsy (idiopathic facial paralysis). Cochrane Database Syst Rev.  2011;(12):CD006283.88 Ferreira  M, Marques  EE, Duarte  JA, et al. Physical therapy with drug treatment in Bell palsy: a focused review. Am J Phys Med Rehabil.  2015;94(4):331-340.99 Pourmomeny  AA, Asadi  S. Management of synkinesis and asymmetry in facial nerve palsy: a review article. Iran J Otorhinolaryngol.  2014;26(77):251-256.1010 Alakram  P, Puckree  T. Effects of electrical stimulation on House-Brackmann scores in early Bell's palsy. Physiother Theory Pract.  2010;26(3):160-166.1111 Axelsson  S, Berg  T, Jonsson  L, et al. Prednisolone in Bell's palsy related to treatment start and age. Otol Neurotol.  2011;32(1):141-146.1212 Marsk  E, Bylund  N, Jonsson  L, et al. Prediction of nonrecovery in Bell's palsy using Sunnybrook grading. Laryngoscope.  2012;122(4):901-906.

SEE ALSO


Amyloidosis; Diabetes Mellitus, Type 1; Diabetes Mellitus, Type 2; Herpes Simplex; Herpes Zoster (Shingles); Lyme Disease; Sarcoidosis; Sj ¶gren Syndrome  

CODES


ICD10


G51.0 Bell's palsy  

ICD9


351.0 Bell's palsy  

SNOMED


  • 193093009 Bell's palsy (disorder)
  • 12239621000119103 Bells palsy of left side of face (disorder)
  • 12239661000119108 Bells palsy of right side of face (disorder)

CLINICAL PEARLS


  • Initiate steroids immediately following the onset of symptoms.
  • Look closely at the voluntary movement on the upper part of the face on the affected side; in Bell palsy, all of the muscles are involved (weak or paralyzed); whereas in a stroke, the upper muscles are spared (because of bilateral innervation).
  • Protect the affected eye with lubrication and taping.
  • In areas with endemic Lyme disease, Bell palsy should be considered to be Lyme disease until proven otherwise.
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