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Women with Epilepsy


Basics


Description


  • A seizure is defined as an abrupt alteration in behavior or perception and is often a symptom of underlying CNS or metabolic dysfunction.
  • Epilepsy is a disease characterized by recurrent, unprovoked seizures (2 or more).

Epidemiology


Incidence
  • Seizures:
    • 80/100,000
  • Epilepsy:
    • Average: 45/100,000
    • Highest incidence under age 10 and over age 60 (>70 cases per 100,000)
    • Cumulative lifetime incidence: 3.1% by age 80

Prevalence
  • Seizures:
    • Lifetime prevalence: 9%
  • Epilepsy:
    • Point prevalence: 0.5 " “1.0% (highest in underdeveloped countries)

Risk Factors


History of any of the following: ‚  
  • Perinatal or gestational insults including prematurity
  • Febrile seizures
  • Family history of epilepsy
  • Encephalitis or meningitis
  • Stroke or subarachnoid hemorrhage
  • Head trauma involving loss of consciousness
  • CNS tumor
  • Dementia

Genetics
  • Contribution of genetics unknown in most epilepsy syndromes
  • Some idiopathic epilepsy syndromes linked to defects in ion channels via mendelian or complex inheritance

General Prevention


Avoid conditions that lower seizure threshold: ‚  
  • Sleep deprivation
  • Alcohol intoxication or withdrawal
  • Illicit drugs: Cocaine, amphetamines
  • Prescription drugs: Antipsychoticstricyclic antidepressants, bupropion, SSRIs, demerol, penicillins

Pathophysiology


  • Prolonged depolarization of neuronal cell membranes
  • Many possible mechanisms:
    • Dysfunction of excitatory (glutamate) or inhibitory (GABA) neurotransmitters
    • Defective ion channels

Etiology


  • Idiopathic: 65.5%
  • Vascular: 10.9%
  • Congenital: 8.0%
  • Trauma: 5.5%
  • Neoplastic: 4.1%
  • Degenerative: 3.5%
  • CNS infection: 2.5%
  • Underlying etiology varies by age. Most common identified etiology:
    • <15 years: Congenital abnormalities
    • 5 " “24 years: Head trauma
    • 25 " “44 years: Brain tumor
    • >45 years: Stroke

Diagnosis


History


  • Assess for underlying risk factors
  • Inquire about seizure frequency and duration
  • Ask about catamenial pattern:
    • Increased seizures in periovulatory and perimenstrual period seen in up to1/3 of women
  • Signs and symptoms:
    • Preceding the seizure (aura):
      • Dej ƒ   vu
      • Rising epigastric sensation
      • Olfactory hallucinations
    • During the seizure (ictal):
      • Automatisms: Lip smacking, picking
      • Unresponsiveness or aphasia
      • Focal or generalized clonic movements
      • Oral laceration
      • Urinary incontinence
    • After the seizure (postictal):
      • Confusion, agitation, psychosis
      • Amnesia of the event

Physical Exam


  • Focal neurological findings may provide clues to underlying CNS etiology.
  • Postictal period: Todd 's paralysis, positive Babinski, dilated pupils
  • Nystagmus and ataxia often seen with toxicity from anticonvulsant medications

Tests


Lab
Initial or acute onset seizures: ‚  
  • Blood glucose
  • Electrolytes: Sodium, calcium, magnesium, phosphate
  • CBC
  • Urine drug screen

Imaging
Initial or acute onset seizures: ‚  
  • Head CT without contrast to exclude conditions requiring urgent intervention (hemorrhage, tumor)
  • Brain MRI, performed in follow-up as outpatient to exclude more subtle structural lesions

Surgery
  • EEG:
    • Aids in classification of seizure type and localization of seizure onset
    • Initial EEG normal in up to 50%
    • Normal EEG does not exclude epilepsy.
  • Lumbar puncture:
    • All HIV+ patients with new onset seizure(s)
    • Any patient with fever, elevated WBC, or suspicion of infection

Differential Diagnosis


  • Physiological:
    • Syncope
    • Transient ischemic attack (TIA)
    • Complicated migraine
    • Sleep disorder
    • Movement disorder: Tremor, tics
    • Transient metabolic disturbance
  • Psychiatric:
    • Conversion disorder
    • Panic attacks
    • Attention deficit hyperactivity disorder (ADHD)

Treatment


Medication


  • Traditional antiepileptic drugs (AEDs)
    • Carbamazepine, phenobarbital, phenytoin, primidone, valproate
    • Advantages
      • Once daily dosing available with most preparations (except carbamazepine)
      • Can be rapidly titrated or loaded intravenously (except carbamazepine)
      • Inexpensive/generic available
    • Disadvantages
      • Drug interactions
      • CNS side effects
      • Teratogenicity
      • Long-term effects
      • Serum monitoring required
  • Second-generation AEDs
    • Gabapentin, lacosamide, lamotrigine, levetiracetam, oxcarbazepine, pregabalin, tiagabine, topiramate, zonisamide
    • Advantages
      • Few drug interactions due to predominantly nonhepatic metabolism and low protein binding
      • Fewer side effects
      • Broad-spectrum coverage of all seizure types (lamotrigine, levetiracetam, topiramate, zonisamide)
      • Generic formulations available for most
    • Disadvantages
      • Slow titration rate (except lacosamide, levetiracetam, gabapentin)
      • Only levetiracetam and lacosamide available in IV formulation
      • Degree of teratogenic risk unknown except for lamotrigine (see below)

Additional Treatment


General Measures
2/3 of patients can be controlled with medications ‚  
Issues for Referral
Refer to neurologist or epileptologist if: ‚  
  • Refractory to 1st or 2nd medication trial
  • Suspicion of pseudoseizures
  • Pregnant or considering pregnancy

Complementary and Alternative Medicine


  • Vagus nerve stimulation
    • For patients refractory to medications and not surgical candidates
    • Stimulator implanted subcutaneously in the chest with electrode to the left vagus nerve
    • Provides seizure reduction and shortened seizure duration but rarely complete seizure control

  • Maternal risk
    • Moderately increased risk of cesarean delivery (1)[B]
    • No clear evidence that seizure frequency increases during pregnancy (1)[B]
    • Declining AED levels due to altered pharmacokinetics (lamotrigine, phenytoin and carbamazepine) (2)[B]
  • Fetal risk
    • Major malformations in 4 " “8% (twice the general population) of pregnant women taking AEDs
    • Risks of maternal seizures include injury to fetus, abruption, or miscarriage secondary to maternal trauma.
    • Potential harm of untreated seizure disorder to a pregnant woman and her fetus usually greater than risk of AED use
    • Congenital malformations:
      • Cleft lip/palate, congenital heart defects, neural tube defects, urogenital defects (3)[A]
      • Most strongly associated with use of polytherapy and valproate (3)[A]
      • Data regarding use of other newer agents lacking except lamotrigine associated with possible increased risk of cleft lip/palate
    • Poor cognitive outcome associated with valproate, possibly phenytoin and phenobarbital (3)[B]
    • Increased risk of small for gestational age (3)[B]
    • Some AEDs are transferred into breast milk but no evidence to support adverse effects on newborn (2)[C]
  • Recommendations
    • Monotherapy at the lowest dose needed to control seizures (3)[B]
    • Avoid polytherapy as well as monotherapy with valproate (3)[A]
    • Folic acid supplementation: 1 " “4 mg/day (start prior to conception) (2)[B]
    • Monthly serum drug levels for women taking lamotrigine, phenytoin, and carbamazepine after conception (2)[B]
    • Breastfeeding is not contraindicated but should be monitored (2)[C].
    • Prenatal testing
      • Maternal serum alpha-fetoprotein at 15 " “20 weeks
    • Level II (structural) ultrasound at 16 " “20 weeks

Surgery


  • Focal brain resection in patients with partial onset seizures refractory to trials of 2 or more medications (alone or in combination)
  • Most successful in patients with focal lesions seen on MRI and/or temporal lobe seizures
  • Up to 70% seizure freedom rate

In-Patient Considerations


Admission Criteria
  • Status epilepticus
    • Continuous seizure activity >5 " “10 minutes or
    • ≥2 seizures without return to baseline in between
  • Prolonged postictal state

Ongoing Care


Follow-Up Recommendations


Patient Monitoring
  • CBC and liver function tests
    • All patients taking carbamazepine, phenobarbital, phenytoin, and valproate due to risk of agranulocytosis and hepatotoxicity
  • Sodium
    • Risk of hyponatremia in patients taking carbamazepine and oxcarbazepine, especially elderly and patients on salt-wasting diuretics
  • Serum drug levels
    • Available for all AEDs
    • Aids in monitoring for toxicity, noncompliance

Prognosis


  • Classification into appropriate epilepsy syndrome aids in prognosis
    • Some generalized epilepsy syndromes will remit in childhood (childhood absence, benign rolandic).
    • Juvenile myoclonic epilepsy and adult onset temporal lobe epilepsy least likely to remit

Complications


  • Infertility
    • Number of births decreased by 33 " “66%
    • Anovulatory cycles increased
    • Also occurs in women on no AEDs
  • Decreased bone mineral density
    • Reported with phenobarbital, mysoline, phenytoin, carbamazepine, and valproate
    • Consider screening with DXA scan
    • Supplement calcium and vitamin D to ensure adequate daily intake (vitamin D minimum 1,000 IU/day)
  • Contraception

  • AEDs that decrease the efficacy of hormonal contraception: Carbamazepine, oxcarbazepine, phenobarbital, phenytoin, primidone, topiramate (>200 mg/day)
  • Oral contraceptives may decrease lamotrigine serum levels by up to 50%.
  • Intrauterine devices should be considered to avoid these potentially serious drug interactions.

References


1Harden ‚  CL, Hopp ‚  J, Ting ‚  T. Management issues for women with epilepsy: Obstetrical complications and change in seizure frequency. Epilepsia.  2009;50(5):1229 " “1236. ‚  [View Abstract]2Harden ‚  CL, Pennell ‚  PB, Koppel ‚  BS. Management issues for women with epilepsy: Vitamin K, folic acid, blood levels and breast feeding. Epilepsia.  2009;50(5):1247 " “1255. ‚  [View Abstract]3Harden ‚  CL, Meador ‚  KJ, Pennell ‚  PB. Management issues for women with epilepsy: Teratogenesis and perinatal outcomes. Epilepsia.  2009;50(5):1237 " “1246. ‚  [View Abstract]

Additional Reading


1Crawford ‚  P. Managing epilepsy in women of child bearing age. Drug Safe.  2009;32(4):293 " “307. ‚  [View Abstract]2LaRoche ‚  SM. A new look at the second-generation antiepileptic drugs: A decade of experience. Neurologist.  2007;12(3):133 " “139.

Additional Reading see also


  • Epilepsy Foundation. Website: www.efa.org
  • North American Pregnancy Registry. Website: www.aedpregnancyregistry.org

Codes


ICD9


  • 345.90 Epilepsy, unspecified, without mention of intractable epilepsy
  • 649.40 Epilepsy complicating pregnancy, childbirth, or the puerperium, unspecified as to episode of care or not applicable

ICD10


  • G40.909 Epilepsy, unsp, not intractable, without status epilepticus
  • O99.350 Diseases of the nervous sys comp pregnancy, unsp trimester

SNOMED


  • 84757009 epilepsy (disorder)
  • 199297006 disease of nervous system complicating pregnancy, childbirth and puerperium (disorder)

Clinical Pearls


  • Patients with a single seizure and risk factors for seizure recurrence should be considered for treatment.
  • Second-generation anti-epileptic drugs have similar efficacy but are better tolerated than the traditional agents.
  • Pre-conceptual counseling aids in optimizing treatment to reduce maternal and fetal complications.
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