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Vomiting, Pediatric, Emergency Medicine


Basics


Description


  • Forceful, coordinated act of expelling gastric contents through the mouth; characterized by nausea, retching, and emesis; no gastric
    contents are expelled during retching.
  • Emesis results from sustained contraction of abdominal muscles and diaphragm; at the same time, the pylorus and antrum contract.

Etiology


Mechanism: ‚  
  • GI/mechanical: Gastroesophageal reflux (GER), meconium ileus, necrotizing enterocolitis, hypertrophic pyloric stenosis, intussusception, malrotation with midgut volvulus, Hirschsprung disease, congenital obstructions (atresias, stenoses, and webs), hernia, foreign body/bezoar, pancreatitis, appendicitis, paralytic ileus
  • Metabolic/endocrine: Inborn errors of metabolism (amino acidurias, fatty acid oxidation disorders, urea cycle defects), uremia, diabetic ketoacidosis, congenital adrenal hyperplasia, kernicterus
  • Neurologic: CNS bleeding (often due to trauma), tumor, hydrocephalus
  • Infectious: Otitis media, UTI, pneumonia, sepsis, gastroenteritis, meningitis/encephalitis
  • Feeding problems: Chalasia, improper technique (overfeeding, improper position), milk allergy
  • Other: Toxicologic, nonaccidental trauma, pregnancy

Diagnosis


Signs and Symptoms


  • General:
    • Appearance variable depending on the underlying cause
    • Signs of dehydration, including tachycardia, tachypnea, pallor, decreased perfusion, and shock
    • Altered mental status may occur secondary to shock, hypoglycemia, or extra-abdominal conditions (sepsis, inborn error of metabolism, increased intracranial pressure, toxicologic poisoning).
  • Vomiting characteristics:
    • Assess color, composition, onset, progression, and relationship to intake and position.
    • Nonbilious emesis is caused by a lesion proximal to the pylorus.
    • Bilious (green) emesis indicates obstruction below the duodenal ampulla of Vater; in infants, bilious emesis is associated with a more serious underlying condition (malrotation, volvulus, intussusception, bowel obstruction); may also be due to adynamic ileus or sepsis.
    • Bloody emesis (hematemesis) involves a lesion proximal to the ligament of Treitz; bright red bloody emesis has little or no contact with gastric juices due to an active bleeding site at or above cardia.
    • "Coffee-grounds "  emesis results from reduction of heme by gastric secretions.
    • Feculent odor suggests lower obstruction or peritonitis.
    • Undigested food in emesis suggests an esophageal lesion or one at or above the cardia.
    • GER: Begins shortly after birth, remains relatively constant, usually with normal weight gain.
    • Hypertrophic pyloric stenosis: Begins insidiously at 2 " “6 wk of age and progresses, becoming increasingly forceful (projectile) after feedings.
    • Obstruction and/or ischemic bowel (malrotation with midgut volvulus, intussusception, necrotizing enterocolitis): Sudden onset associated with rapid progression to appearing ill out of proportion to the duration of illness; abdomen distended and tender.
  • Abdominal:
    • Distention suggests obstruction.
    • Peritoneal signs suggest inflammation and possible perforation.
  • Complications:
    • Aspiration
    • Mallory " “Weiss tear
    • Boerhaave syndrome

History
  • Constitutional:
    • Fever
  • Vomiting characteristics:
    • Timing, duration
    • Bilious?
    • Bloody?
  • Associated symptoms:
    • Diarrhea
    • Anorexia
    • Abdominal pain
    • Dysuria
    • Inguinal swelling
  • PMHx:
    • History of similar
    • Past surgical history

Physical Exam
  • General:
    • General appearance, vital signs, hydration status
  • Cardiovascular:
    • Quality heart tones
    • Pulses, perfusion
  • Abdominal:
    • Tenderness, distention, mass
    • Bowel sounds
  • Genitourinary:
    • Scrotal swelling, tenderness, mass
  • Rectal:
    • Presence of blood, mass, tenderness

Essential Workup


Exclude life-threatening causes of vomiting. ‚  

Diagnosis Tests & Interpretation


Lab
  • As indicated by history and physical exam and consideration of differential:
    • Metabolic assessment (glucose, electrolytes)
    • Infection assessment (CBC, culture " ”urine)
    • Pregnancy tests for females of childbearing age

Imaging
  • As indicated by differential considerations
  • Abdominal radiographs (flat plate, upright, and decubitus) helpful for evaluation of obstruction or perforation
  • Pelvic and abdominal US for evaluation of hypertrophic pyloric stenosis, intussusception, appendicitis as well as pelvic or scrotal pathology
  • Abdominal CT scan helpful for evaluation of appendicitis, mass/tumor often requiring contrast

Diagnostic Procedures/Surgery
Nasogastric tube: ‚  
  • Location, character, and severity of gastric bleeding

Differential Diagnosis


  • Neonate/infant:
    • GI/mechanical: GER, meconium ileus, necrotizing enterocolitis, hypertrophic pyloric stenosis, intussusception, malrotation with midgut volvulus, Hirschsprung disease, congenital obstructions (atresias, duplications, imperforate anus. stenoses, and webs), hernia, foreign body/bezoar, paralytic ileus
    • Metabolic/endocrine: Inborn errors of metabolism (amino acidurias, fatty acid oxidation disorders, urea cycle defects), uremia, congenital adrenal hyperplasia, kernicterus
    • Neurologic: CNS bleeding (often due to trauma), tumor, hydrocephalus
    • Infectious: Otitis media, UTI, pneumonia, sepsis, pertussis, meningitis/encephalitis
    • Feeding problems: Chalasia, improper technique (overfeeding, improper position), milk allergy
    • Other: Toxicologic, nonaccidental trauma
  • Child/adolescent:
    • GI: Gastroenteritis, obstruction (hernia, adhesions, intussusception, foreign body, bezoar), pancreatitis, appendicitis, peptic ulcer, peritonitis, paralytic ileus, trauma (duodenal hematoma)
    • Metabolic/endocrine: Diabetic ketoacidosis, uremia, adrenal insufficiency
    • Infectious: Gastroenteritis, UTI, sinusitis, upper respiratory infection, sepsis, meningitis, encephalitis, pneumonia, hepatitis
    • Neurologic: CNS mass/tumor, CNS bleeding (often due to trauma), cerebral edema, concussion, migraine
    • Other: Toxicologic, (nonaccidental) trauma, pregnancy, bulimia

Treatment


Pre-Hospital


Not applicable ‚  

Initial Stabilization/Therapy


  • Fluid resuscitation with 0.9% NS IV; caution if concern about increased intracranial pressure.
  • Determine bedside fingerstick glucose.

Ed Treatment/Procedures


  • Continue fluid resuscitation and correction of electrolyte imbalance if present.
  • Decompress stomach with nasogastric or orogastric tube if abdomen distended or vomiting persistent.
  • Continue evaluation for underlying cause.
  • Consider antiemetic medications.
  • Surgical consultation if acute abdomen; antibiotics if peritonitis or other systemic infection present

Medication


Antiemetics may be helpful once the underlying cause of vomiting has been determined. ‚  
First Line
Ondansetron: 4 " “8 mg (peds: 0.1 mg/kg per dose) IV or PO q6h ‚  
Second Line
  • Metoclopramide: 10 mg (peds: 0.1 mg/kg per dose) PO q6h
  • Prochlorperazine: 2.5 " “5 mg (peds: 0.1 mg/kg per dose) IV, IM, or PR q6h
  • Promethazine: 12.5 " “25 mg (peds: 0.25 mg/kg per dose) PO, PR, or IM q6h

Follow-Up


Disposition


Admission Criteria
  • Unstable vital signs, including persistent tachycardia or other evidence of hypovolemia
  • Serious etiologic condition or inability to exclude serious etiologic conditions
  • Intractable vomiting or inability to take oral fluids
  • Inadequate social situation or follow-up

Discharge Criteria
  • Stable; able to tolerate oral fluids
  • Benign etiology considered most likely and serious or potentially important etiologies excluded
  • Parental understanding of instructions to advance clear liquids slowly and return for continued vomiting, abdominal distention, decreased urination, fever, lethargy, or unusual behavior

Issues for Referral
  • Chronic or recurrent episodes of vomiting or abdominal pain:
    • Pediatric gastroenterology

Followup Recommendations


PCP in 1 " “2 days ‚  

Pearls and Pitfalls


  • Determine presence or absence of bile or blood in emesis.
  • Bilious vomiting in the neonate is an important anatomic abnormality such as malrotation until proven otherwise.
  • Consider causes of vomiting other than just GI (see Differential Diagnosis).

Additional Reading


  • Chandran ‚  L, Chitkara ‚  M. Vomiting in children: Reassurance, red flag, or referral? Pediatr Rev.  2008;29(6):183 " “192.
  • Claudius ‚  I, Kou ‚  M, Place ‚  R, et al. An evidence based review of neonatal emergencies. Pediatric Emergency Med Practice.  2010;7(6):1 " “22.
  • Hostetler ‚  MA. Gastrointestinal disorders. In: Marx ‚  JA, Hockerberger ‚  RS, Walls ‚  RM, et al., eds. Emergency Medicine: Concepts and Clinical Practice. 7th ed. St. Louis: Mosby; 2010:2168 " “2187.
  • Pepper ‚  VK, Stanfill ‚  AB, Pearl ‚  RH. Diagnosis and management of pediatric appendicitis, intussusception and Meckel diverticulum. Surg Clin North Am.  2012;92(3):505 " “526.

Codes


ICD9


  • 530.81 Esophageal reflux
  • 787.03 Vomiting alone
  • 787.04 Bilious emesis
  • 578.0 Hematemesis

ICD10


  • K21.9 Gastro-esophageal reflux disease without esophagitis
  • R11.10 Vomiting, unspecified
  • R11.14 Bilious vomiting
  • K92.0 Hematemesis

SNOMED


  • 422400008 Vomiting (disorder)
  • 71419002 Bilious vomiting
  • 235595009 Gastroesophageal reflux disease (disorder)
  • 8765009 Hematemesis (disorder)
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