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Torticollis

para>Congenital muscular torticollis (CMT): seen at birth or in early infancy " ”accounts for 80% of torticollis cases presenting in infancy. Results from unilateral fibrosis and shortening of the sternocleidomastoid (SCM) muscle
  • Acquired torticollis

  • Adult disorders include the following:
    • Acquired torticollis (also known as "wryneck " ) " ”usually self-limited.

    • Spasmodic torticollis, (cervical dystonia) caused by recurrent involuntary muscular contractions

    • Other less common forms of torticollis include oculogyric, gastroesophageal reflux " “induced, arthritis-related, scoliosis-related, and hysterical torticollis.

  • System(s) affected: musculoskeletal; nervous
  • Synonym(s): acute wryneck; idiopathic generalized torticollis; SCM torticollis; neonatal torticollis; idiopathic cervical dystonia; focal dystonia; nuchal dystonia

  • Epidemiology


    • ’ ˆ Ό90% of cases occur in individuals aged 31 " “60 years.
    • Predominant age: CMT: newborn and infants; pediatric acquired torticollis, <10 years; adult acquired torticollis, 30 " “60 years; spasmodic torticollis, 30 " “50 years (mean age 40 " “43 years)
    • Predominant sex: spasmodic torticollis, female > male (1.6:1); CMT, male > female (3:2)

    Incidence
    Congenital: up to 1/250 births; spasmodic: 1/100,000; overall incidence for torticollis is 24/1 million persons. ‚  
    Prevalence
    All focal dystonias combined: 295/1 million persons; no reliable data for acquired torticollis. ‚  

    Etiology and Pathophysiology


    • CMT
      • Intrauterine malpositioning may lead to foreshortening and fibrosis of the SCM.

      • Birth trauma (e.g., clavicular fracture) can lead to CMT as well.

    • Pediatric acquired torticollis
      • CNS disorders, increased intracranial pressure

      • Ocular disorders " ”abnormal head/neck positioning as visual compensation

      • Bony abnormalities, soft tissue pathologies

      • Gastrointestinal disease

      • Drug induced

      • Conversion disorder

    • Adult acquired torticollis
      • Emotional stress, postural factors (e.g., work, sleep, lying while reading or watching TV, prolonged unusual positioning of neck), or exposure to cold. Many cases are idiopathic.

      • Medication reactions (most commonly anticholinergics, amphetamines, and certain anesthetic agents)

    • Spasmodic torticollis
      • Muscular spasm secondary to traumatic, infectious or inflammatory insult

      • Cervical spine injuries and spondylosis, ocular disorders, organic CNS disorders, psychogenic causes, certain tumors and vestibular dysfunction can all contribute to cervical dystonia.


    Genetics
    Spasmotic torticollis may also have a genetic basis. ‚  

    Risk Factors


    • CMT: intrauterine crowding, breech presentation, ischemia, birth injury
    • Pediatric acquired torticollis: soft tissue inflammation or infection, neurologic disease, visual disturbances, trauma
    • Adult acquired torticollis: stress, unusual neck position (particularly when sleeping), exposure to cold, medications, trauma, infection
    • Spasmodic: family history of dystonia, soft tissue inflammation or infection, neurologic conditions, visual disturbances, trauma

    Pediatric Considerations
    CMT: without treatment becomes a fibrous cord and may be associated with persistent craniofacial deformities and persistent torticollis ‚  

    Commonly Associated Conditions


    • >80% of infants with CMT present with craniofacial asymmetry, deformational plagiocephaly, and developmental dysplasia of the hip (DDH).
    • CMT and pediatric acquired torticollis: Consider Klippel-Feil syndrome (congenital fusion of cervical vertebrae).
    • Pediatric and adult acquired torticollis: Consider spinal abnormalities, tumors.
    • Spasmodic torticollis often accompanied by behavioral health conditions

    Diagnosis


    History


    • Abnormal head posture, neck pain, headache, neck muscle stiffness, restricted neck range of motion, neck mass or swelling
    • Birth history in children
    • Family history of dystonias
    • Medication history
    • Recent cervical spine trauma

    Physical Exam


    • Normal ROM: flexion 60 degrees; extension 75 degrees; rotation 90 degrees; side-bending 45 degrees
    • Torticollis presents as rotational (twisting), anterocollis (flexion), laterocollis (side bending), and retrocollis (extension) with the head tilting to the affected side (80%, to right side) and the chin rotating to the opposite side.
    • Spasm of trapezius, SCM, and other neck muscles
    • Tenderness over the affected SCM
    • Neck mass, lymphadenopathy
    • Craniofacial asymmetry (plagiocephaly) consistent with congenital or chronic torticollis
    • Phasic jerking or tremor of antagonist muscles
    • Physical gesturing (geste antagoniste) such as touching face or chin to reduce dystonia (pathognomonic for spasmodic torticollis)
    • Ocular irregularities (e.g., diplopia)
    • Spinal abnormality: Short neck with low posterior hairline may indicate occipitocervical synostosis.
    • Structural abnormalities of the hips or feet
    • Common physical findings in different types of torticollis
      • CMT: Firm, nontender, palpable enlargement of the SCM may appear from birth or develop over weeks.

      • Pediatric and adult acquired torticollis: unilateral neck stiffness, pain, spasm, or decreased ROM without trauma

      • Spasmodic torticollis may initially present with neck stiffness progressing to pain, head jerking, and neck spasms.


    Differential Diagnosis


    • Osseous
      • Atlantoaxial rotatory subluxation

      • Atlanto-occipital subluxation

      • Posttraumatic fracture or dislocation

      • Cervical disc disease

      • Congenital scoliosis

      • Klippel-Feil syndrome

      • Occipitocervical synostosis

      • Grisel syndrome

      • Syringomyelia

      • Arnold-Chiari malformation

    • Nonosseous
      • Myositis involving cervical muscles

      • Soft tissue trauma

      • Neoplastic: spinal cord tumor, acoustic neuroma, osteoblastoma, orbital tumor, fibromatosis, metastasis

      • Infection: upper respiratory infection, cervical lymph node abscess, epidural abscess, retropharyngeal abscess, vertebral osteomyelitis

      • Vestibular disorders

      • Essential head tremor

      • Basal ganglion diseases

      • Cranial nerve palsy

      • Psychiatric disorders

      • Drugs or toxins

      • Down syndrome

      • Sandifer syndrome

      • Myasthenia gravis


    Diagnostic Tests & Interpretation


    Lab studies are only needed to exclude underlying disease. ‚  
    • Radiographs should be taken to rule out spinal pathology in traumatic and congenital cases.
    • Consider MRI or CT scan of cervical spine for patients with neurologic deficits.
    • CMT can be confirmed with US of involved SCM.

    Follow-Up Tests & Special Considerations
    Infants with CMT should be screened for DDH by physical exam and subsequent US if not clearly normal (1)[C]. All pediatric patients should have an eye exam. ‚  

    Treatment


    General Measures


    • CMT
      • >90% of children achieve good outcome with conservative treatment if initiated by age 1 year.

      • Conservative treatment includes positioning, environmental adaptations, passive and active stretching of the tight SCM muscle, strengthening of weak neck and trunk muscles, and movement therapy.

      • Physical therapy (PT) and stretching should be started before ages 3 " “6 months (2)[B].

      • Place toys on opposite side of bed from rotational deformity to encourage use of affected muscles.

      • Surgery is indicated for refractory cases, a tight muscular band, or SCM mass.

    • Pediatric acquired torticollis
      • Place a television on opposite side of bed from rotational deformity.

      • Some patients respond well to low-dose levodopa (100 " “300 mg) with carbidopa.

    • Adult and pediatric acquired torticollis
      • Conservative management includes soft cervical collar, intermittent heat or ice, bed rest, and analgesics for pain relief.

    • Spasmodic
      • Conservative: soft cervical collar, heat or ice, bed rest

      • Selective peripheral nerve denervation

      • Pallidal deep brain stimulation can achieve lasting improvements in medication-resistant cases (3)[B].


    Medication


    First Line
    • Treatment for spasms
      • Botulinum toxin injections may be more effective than anticholinergic drugs for cervical dystonia (4)[B],(5)[A]. Major predictors of treatment success are correct dosing and identification of affected muscles. Note that the recommended doses are general guidelines and must be individualized to the patient. EMG (6)[A] or PET-CT (7)[B] may help guide the injection site. Immunoresistance can develop but is rare (8)[A]:

        • Botulinum toxin types A and B have been found effective and safe for treating adults ( ≥16) with cervical dystonia (9)[A].

        • CMT: botulinum toxin type A was found effective in a case series (2)[C].

      • Botox: botulinum toxin type A

        • Adult: initial 1.25 " “2.5 units (0.05 " “0.1 mL) IM into most active neck muscles; repeat every 3 " “4 months; not to exceed 200 units cumulative dose in 1-month period

        • Pediatric: <12 years of age, not established; >12 years, administer as for adults

        • Xeomin (botulinum toxin type A free from complexing proteins) has shown comparable safety and efficacy to Botox (10)[B].

      • Myobloc: botulinum toxin type B

        • Adult: 2,500 " “5,000 units IM divided among affected muscles in patients treated previously with any type of botulinum toxin; use lower dose in untreated patients.

        • Pediatric: not established

      • Diazepam

        • Adult: 2 " “10 mg PO TID/QID

        • Pediatric: 1 " “2.5 mg PO TID/QID; increase gradually as needed or tolerated.

      • Diphenhydramine or diazepam can be used for torticollis caused by medications (4)[C]:

        • Diphenhydramine

          • Adult: 25 " “50 mg PO q6 " “8h PRN, not to exceed 400 mg/day; 10 " “50 mg IV/IM q6 " “8h PRN, not to exceed 400 mg/day

          • Pediatric: 12.5 " “25 mg PO TID/QID or 5 mg/kg/day or 150 mg/m2/day divided TID/QID. PRN, not to exceed 300 mg/day; 5 mg/kg/day IV/IM or 150 mg/m2/day, divided QID. PRN, not to exceed 300 mg/day

      • Anticholinergics may relieve acute muscle spasms (4)[C], but high doses are usually necessary and benefit is often delayed.

      • Carbidopa-levodopa (4)[C] for focal dystonia of unknown cause: Trial might be warranted although some non " “dopa-responsive dystonias improve, equal numbers have symptoms worsen.

    • Treatment for pain
      • NSAIDs and acetaminophen; consider opiates for only the most severe pain.


    Issues for Referral


    • Refer prolonged symptoms of idiopathic spasmodic torticollis to a neurologist.
    • Fixed deformities in children may require surgical consultation.
    • CMT: Refer if visual dysfunction (ophthalmology), failed hip screen (orthopedics), abnormal neurologic exam (neurology), plagiocephaly (plastic surgery), or patient presents with bony prominence (orthopedics).

    Additional Therapies


    • Osteopathic manipulation may be useful:
      • Direct myofascial stretching of cervical region with attention to the SCM

      • Occipital-atlantal release

      • V-spread of the occipitomastoid suture on the side of restriction

      • Muscle energy and/or functional positional release at the cervical region

    • PT may be beneficial for acquired childhood and adult torticollis (11)[B].
    • If detected early, 90% of CMT responds to stretching exercises.

    Surgery/Other Procedures


    CMT: Surgical release if PT is unsuccessful by age 1 year ‚  

    Complementary & Alternative Medicine


    Acupuncture may also provide benefit. ‚  

    Ongoing Care


    Follow-up Recommendations


    • Monitor infants with CMT every 2 " “4 weeks until resolved.
    • Depression is common in protracted cases, screen accordingly.

    Prognosis


    • CMT: good for correctable pathologies
      • 50 " “70% resolve spontaneously by 1st year

      • >90% of children achieve good to excellent outcomes with conservative treatment when therapy is instituted by age 1 year.

    • Pediatric acquired torticollis: good when the underlying pathology is discovered and treated
    • Adult acquired torticollis: excellent; generally resolves in a few days to weeks
    • Spasmodic: may wax and wane for years, even with treatment

    Complications


    • Facial asymmetry in CMT; possibly an increased risk of developmental delay/disorders
    • Dental malocclusion
    • Degenerative osteoarthritis of the cervical spine, hypertrophy of the SCM muscle, and paresthesia due to compressed nerve roots
    • Depression

    References


    1.Joiner ‚  ERA, Andras ‚  LM, Skaggs ‚  DL. Screening for hip dysplasia in congenital muscular torticollis: is physical exam enough? J Child Orthop.  2014;8(2):115 " “119. ‚  [View Abstract]2.Do ‚  TT. Congenital muscular torticollis: current concepts and review of treatment. Curr Opin Pediatr.  2006;18(1):26 " “29. ‚  [View Abstract]3.Walsh ‚  RA, Sidiropoulos ‚  C, Lozano ‚  AM, et al. Bilateral pallidal stimulation in cervical dystonia: blinded evidence of benefit beyond 5 years. Brain.  2013;136(Pt 3):761 " “769. ‚  [View Abstract]4.Tarsy ‚  D, Simon ‚  DK. Dystonia. N Engl J Med.  2006;355(8):818 " “829. ‚  [View Abstract]5.Patel ‚  S, Martino ‚  D. Cervical dystonia: from pathophysiology to pharmacotherapy. Behav Neurol.  2013;26(4):275 " “282. ‚  [View Abstract]6.Nijmeijer ‚  SW, Koelman ‚  JH, Kamphuis ‚  DJ, et al. Muscle selection for treatment of cervical dystonia with botulinum toxin " ”a systematic review. Parkinsonism Relat Disord.  2012;18(6):731 " “736. ‚  [View Abstract]7.Lee ‚  HB, An ‚  YS, Lee ‚  HY, et al. Usefulness of (18)f-fluorodeoxyglucose positron emission tomography/computed tomography in management of cervical dystonia. Ann Rehabil Med.  2012;36(6):745 " “755. ‚  [View Abstract]8.Coleman ‚  C, Hubble ‚  J, Schwab ‚  J, et al. Immunoresistance in cervical dystonia patients after treatment with abotulinum toxin A. Int J Neurosci.  2012;122(7):358 " “362. ‚  [View Abstract]9.Simpson ‚  DM, Blitzer ‚  A, Brashear ‚  A, et al. Assessment: botulinum neurotoxin for the treatment of movement disorders (an evidence-based review): report of the Therapeutics and Technology Assessment Subcommittee of the American Academy of Neurology. Neurology.  2008;70(19):1699 " “1706. ‚  [View Abstract]10.Dressler ‚  D, Paus ‚  S, Seitzinger ‚  A, et al. Long-term efficacy and safety of incobotulinumtoxinA injections in patients with cervical dystonia. J Neurol Neurosurg Psychiatry.  2013;84(9):1014 " “1019. ‚  [View Abstract]11.Queiroz ‚  MA, Chien ‚  HF, Sekeff-Salem ‚  FA, et al. Physical therapy program for cervical dystonia: a study of 20 cases. Funct Neurol.  2012;27(3):187 " “192. ‚  [View Abstract]

    Additional Reading


    • Consky ‚  EA. Clinical assessments of patients with cervical dystonia. In: Jancovic ‚  J, Hallett ‚  M, eds. Therapy with Botulinum Toxin. New York, NY: Marcel Dekker; 1994:211 " “237.
    • Jankovic ‚  J. Treatment of cervical dystonia with botulinum toxin. Mov Disord.  2004;19(Suppl 8):S109 " “S115. ‚  [View Abstract]
    • Tomczak ‚  KK, Rosman ‚  NP. Torticollis. J Child Neurol.  2013;28(3):365 " “378. ‚  [View Abstract]

    Codes


    ICD10


    • M43.6 Torticollis
    • Q68.0 Congenital deformity of sternocleidomastoid muscle
    • G24.3 Spasmodic torticollis
    • S13.4XXA Sprain of ligaments of cervical spine, initial encounter

    ICD09


    • 723.5 Torticollis, unspecified
    • 754.1 Congenital musculoskeletal deformities of sternocleidomastoid muscle
    • 333.83 Spasmodic torticollis
    • 847.0 Sprain of neck

    SNOMED


    • 70070008 torticollis (disorder)
    • 268240006 Congenital torticollis
    • 74333002 Spasmodic torticollis (disorder)
    • 360444002 Traumatic torticollis

    Clinical Pearls


    • Suspect torticollis in patients with head tilt, chin lift, and restricted movement of the neck.
    • Consider secondary causes, and rule out as necessary based on history and physical exam.
    • Most adult cases are self-limiting and resolve within days to weeks.
    • In congenital and acquired pediatric cases, placing items of interest to the child (e.g., TV, toys) on the opposite side of the bed helps to speed resolution.
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