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Theophylline Poisoning, Emergency Medicine


Basics


Description


  • Theophylline causes:
    • Release of endogenous catecholamines resulting in stimulation of ˇ ²1- and ˇ ²2-receptors
    • Adenosine antagonism
    • Inhibition of phosphodiesterase (at supratherapeutic levels)
  • Available in immediate- and sustained-release formulations
  • Peak absorption is 60 " “90 min with immediate-release and 6 " “10 hr with sustained-release formulations
  • Acute overdose:
    • Ingestion within an 8-hr interval in a patient with no prior theophylline use
  • Acute-on-chronic overdose:
    • Single excessive dose in a patient previously receiving usual therapeutic doses for ≥24 hr
  • Chronic intoxication:
    • Accumulation of theophylline >20 ˇ ¼g/mL associated with prior therapeutic use for ≥24 hr secondary to:
      • Drug " “drug, drug " “diet, or drug " “disease interactions
    • Use of serial excessive doses

Etiology


  • Acute ingestions require larger concentrations to achieve specific toxic effects compared with acute-on-chronic or chronic overdoses.
  • Drug " “drug interactions:
    • Inhibiting theophylline metabolism (leads to toxicity when started):
      • H2-receptor antagonists
      • Macrolide antibiotics
      • Fluoroquinolones
      • Allopurinol
      • Influenza vaccine
      • Interferons
    • Enhances theophylline metabolism (leads to toxicity when discontinued):
      • Carbamazepine
      • Barbiturates
      • Smoking
      • Rifampin
  • Chronic theophylline accumulation:
    • Uncontrolled CHF
    • Liver disease (cirrhosis or severe hepatitis)
    • Acute viral infections

Diagnosis


Signs and Symptoms


  • Cardiovascular:
    • Sinus, atrial, and ventricular tachycardias:
      • Multifocal atrial tachycardia
      • Atrial fibrillation
      • Premature ventricular contractions
      • Ventricular tachycardia
      • Due to ˇ ²1-receptor stimulation and adenosine antagonism
    • Hypotension:
      • Associated with theophylline >100 ˇ ¼g/mL (acute ingestion)
      • Due to vasodilatation induced by ˇ ²2-receptor stimulation
      • May be refractory to fluids, positioning, and conventional vasopressors
  • CNS:
    • Tremor
    • Mental status changes
    • Seizures:
      • 14% of chronic intoxications
      • 5% of acute intoxications
  • GI:
    • Nausea, vomiting:
      • Protracted and may be refractory to antiemetics at usual doses
      • 75% of acute intoxications
      • 30% of chronic intoxications
    • Abdominal pain
    • Pharmacobezoar:
      • From sustained-release preparations in acute ingestions
      • Delays peak concentrations
  • Metabolic:
    • Hypokalemia:
      • Typically decreases approximately to 3 mEq/L
      • Due to ˇ ²-receptor stimulation
    • Hyperglycemia
    • Leukocytosis
    • Hypophosphatemia and hypomagnesemia
    • Metabolic acidosis with increased serum lactate levels

Essential Workup


  • Serum theophylline concentration:
    • Finding of ≥20 ˇ ¼g/mL confirms diagnosis.
  • ECG and cardiac monitoring
  • Detailed history to differentiate acute from acute-on-chronic from chronic intoxication

Diagnosis Tests & Interpretation


Lab
  • Serum theophylline level:
    • Repeat every 2 hr until decreasing to confirm immediate absorption is complete and peak value has occurred.
    • Serious morbidity in acute overdose if ≥100 ˇ ¼g/mL
  • CBC
  • Electrolytes

Imaging
  • KUB (kidneys, ureters, bladder):
    • Undissolved sustained-release tablets or pharmacobezoars may appear as radiopacities.
    • Bead-filled capsules may appear as radiolucencies.
  • US of stomach may detect intact sustained-release dosage forms.

Differential Diagnosis


  • Caffeine/ ˇ ²-agonist bronchodilator overdose
  • Amphetamines
  • Sympathomimetics
  • Anticholinergic agents
  • Drug withdrawal syndromes
  • Pheochromocytoma
  • Thyrotoxicosis

Treatment


Pre-Hospital


Bring pill bottles/pill samples in suspected overdose. ‚  

Initial Stabilization/Therapy


  • ABCs:
    • Cardiac monitor
    • Isotonic crystalloids as needed for hypotension
  • Naloxone, thiamine, and dextrose (D50W) as indicated for altered mental status
  • Cardiovascular:
    • Initiate ˇ ²-blockers or calcium channel blockers for rate control with supraventricular tachyarrhythmias (SVT).
    • Adenosine is antagonized by theophylline and may not be effective to treat SVT.
    • Administer isotonic crystalloid IV fluid resuscitation for hypotension:
      • With treatment failure, consider ˇ ²-blocker to reverse theophylline-induced ˇ ²2-receptor " “stimulated vasodilation.
      • If vasopressors are needed, choose vasopressor that is not a ˇ ²-agonist, such as phenylephrine.
    • Treat ventricular dysrhythmias conventionally.
  • Seizures:
    • Administer benzodiazepines.
    • Phenytoin is contraindicated; it is usually ineffective and may paradoxically worsen seizures in theophylline intoxications.

Ed Treatment/Procedures


Decontamination
  • Administer activated charcoal
  • Multidose activated charcoal:
    • Especially with sustained-release products
    • Binds theophylline, which back-diffuses in to the small intestine
    • For mild to moderate toxicity
    • 25 g q2h until theophylline level ≤20 ˇ ¼g/mL
  • Initiate whole-bowel irrigation with sustained-release products:
    • Administer 1 " “2 L/hr of polyethylene glycol until a clear, colorless rectal effluent or theophylline level ≤20 ˇ ¼g/mL
  • Treat protracted vomiting with metoclopramide or 5-HT3-receptor antagonists.
  • Avoid syrup of ipecac.

Electrolyte Disturbances
  • Treat hypokalemia in acute ingestions cautiously:
    • Relative hypokalemia owing to ˇ ²-receptor " “mediated intracellular shift of extracellular potassium.
    • Aggressive correction leads to potentially serious hyperkalemia as theophylline concentrations decrease.
  • Most electrolyte imbalances respond to ˇ ²-blocker therapy:
    • Generally not indicated; however, because of absence of associated morbidity and potential for ˇ ²-blocker " “induced bronchospasm in pulmonary patients

Extracorporeal Elimination
Initiate hemodialysis or hemoperfusion if theophylline level: ‚  
  • ≥90 ˇ ¼g/mL and symptomatic in acute ingestions
  • ≥40 ˇ ¼g/mL and:
    • Seizures or
    • HTN unresponsive to IV fluid or
    • Ventricular dysrhythmias

Medication


  • Activated charcoal: 1 g/kg PO, if dose ingested is known, 10 g/1 g theophylline ingested, max. dose 100 g
    • Multidose-activated charcoal 25 g q2h until theophylline level ≤20 ˇ ¼g/mL
  • Diazepam: 0.1 mg/kg IV q5 " “10min until seizures controlled, up to 30 mg
  • Diltiazem: 0.25 mg/kg IV bolus; may repeat after 15 min, then 5 " “15 mg/h infusion for control of heart rate in patients with contraindication to ˇ ²-blockade
  • Esmolol: 500 ˇ ¼g/kg IV bolus, followed by 50 ˇ ¼g/kg/min infusion; increase by 50 ˇ ¼g/kg/min increments to max. of 200 ˇ ¼g/kg/min
  • Metoclopramide: 10 mg IV bolus; may repeat to max. of 1 mg/kg
  • Ondansetron: 0.15 mg/kg IV bolus up to max. of 16 mg total
  • Polyethylene glycol (high molecular weight): 1 " “2 L/h via nasogastric tube

Follow-Up


Disposition


Admission Criteria
ICU: ‚  
  • Acute overdoses with serum theophylline concentrations ≥100 ˇ ¼g/mL
  • Acute-on-chronic or chronic theophylline with either serum concentration ≥60 ˇ ¼g/mL or patient >60 yr old
  • Seizures or hypotension refractory to fluids and vasopressors in a patient with serum theophylline concentration ≥40 ˇ ¼g/mL

Discharge Criteria
  • 2 consecutive ( ≥2 hr apart) decreasing serum theophylline concentrations with most recent concentration <30 ˇ ¼g/mL
  • Mildly symptomatic or asymptomatic patient meeting above criterion and no evidence of suicidal intention

Follow-Up Recommendations


  • Follow up with medical toxicologist or primary care doctor
  • If patient is on chronic theophylline, dosing regimen may have to be adjusted.

Pearls and Pitfalls


  • Seizures are a major complication.
  • Tachydysrhythmias are common in overdose.
  • Multi-dose activated charcoal is beneficial in theophylline overdose.

A special thanks to Dr. Harry Karydes who contributed to the previous edition. ‚  

Additional Reading


  • Henderson ‚  A, Wright ‚  DM, Pond ‚  SM. Management of theophylline overdose patients in the intensive care unit. Anaesth Intensive Care.  1992;20:56 " “62.
  • Hoffman ‚  RJ. Methylxanthines and selective ˇ ²2-adrenergic agonists. In: Flomenbaum ‚  NE, Goldfrank ‚  LR, Hoffman ‚  RS, et al., eds. Goldfrankss Toxicologic Emergencies. 9th ed. New York, NY: McGraw-Hill Medical; 2011.
  • Shannon ‚  M. Life-threatening events after theophylline overdose: A 10-year prospective analysis. Arch Intern Med.  1999;159:989 " “994.
  • Shannon ‚  MW. Comparative efficacy of hemodialysis and hemoperfusion in severe theophylline intoxication. Acad Emerg Med.  1997;4:674 " “678.

Codes


ICD9


975.7 Poisoning by antiasthmatics ‚  

ICD10


  • T48.6X1A Poisoning by antiasthmatics, accidental, init
  • T48.6X5A Adverse effect of antiasthmatics, initial encounter

SNOMED


  • 64808005 Poisoning by theophylline
  • 291363004 Accidental theophylline poisoning
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