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Staphylococcal Scalded Skin Syndrome, Emergency Medicine


Basics


Description


  • Results from the actions of a soluble epidermolytic exotoxin produced by Staphylococcus aureus:
    • Produced at a distant site of infection or colonization
    • Disseminates hematogenously
    • Lyses desmosomes of granular cells in the superficial epidermis
    • Results in generalized intradermal exfoliation
  • Typically affects infants and children <6 yr of age:
    • Adults have specific staph antibodies allowing them to localize, metabolize, and excrete the staph toxins.
    • Infants and children are unable to metabolize and excrete toxin efficiently.
    • Immunocompromised adults and those with severe renal dysfunction are also susceptible
  • Presentation determined by age and extent of rash:
    • Classic staphylococcal scalded skin syndrome
    • Pemphigus neonatorum
    • Bullous impetigo
    • Generalized in the newborn: Ritter disease
  • Typically, coagulase-positive phage group II Staphylococcus:
    • Phage groups I and III also implicated

Etiology


  • Colonization often without overt infection
  • Concurrent infection or break of skin barrier:
    • Nasopharynx
    • Urinary tract
    • Minor skin abrasions
    • Circumcision site
    • Conjunctivitis
    • Umbilicus/omphalitis
    • Impetigo
    • Endocarditis and septicemia
  • Often no focus identified

Diagnosis


Signs and Symptoms


  • Constitutional symptoms:
    • Malaise
    • Fever
    • Irritability
    • Child may appear well, ill, or overtly toxic
    • Abrupt onset
  • Scarlatiniform erythematous rash (sandpaper like) resembling a "sunburn " ¯ " ”erythroderma
  • Exquisitely tender skin
  • Areas of prominence:
    • Around the flexor areas of the neck
    • Intertriginous areas, especially axilla and groin
    • Near the eyes and mouth
    • Increased erythema in skin creases
  • Facial edema with radial crusting fissures around the eyes, nose, and mouth
  • Flaccid bullae:
    • Within 1 " “3 days after onset of rash
    • Initially over flexures (axillae, groin, body orifices)
    • Bullae migrate through epidermis with light lateral pressure; epidermis separates with minor pressure (Nikolsky sign).
    • Rupture within hours
    • Epidermis separates with minor trauma.
    • Epidermis is shed in sheets.
    • Denuded areas are moist, sensitive, and painful.
    • Complete healing within 2 wk, no scarring
  • Purulent conjunctivitis
  • Mucous membranes not affected
  • Complications rare:
    • Hypothermia
    • Fluid and electrolyte imbalance
    • Secondary infection
    • Pneumonia
    • Septicemia
    • Cellulitis
    • Osteomyelitis

Essential Workup


  • Clinical presentation is diagnostic.
  • Determine location/source of toxin producing Staphylococcus.
  • Assess systemic nature of infection.

Diagnosis Tests & Interpretation


Lab
  • CBC and urinalysis:
    • Assess for sepsis if source not obvious.
  • Electrolytes:
    • Indicated if signs of dehydration or extensive rash
  • Blood cultures (rarely positive)

Imaging
Indicated as need to determine location/source of infection ‚  
Diagnostic Procedures/Surgery
  • Fluid aspirated from bullae:
    • Sterile in staphylococcal scalded skin syndrome
    • Consistent with hematogenous dissemination of the toxin
  • Isolation of staphylococci from a site other than the blisters:
    • Commonly conjunctivae, nasopharynx, or blood
  • Skin biopsy or frozen histologic section:
    • Determine level of epidermal/dermal separation (cleavage is in granular layer of dermis).
    • Indicated for children on medications, those >6 yr, and in cases of mixed presentation

Differential Diagnosis


  • Infection:
    • Scarlet fever:
      • Involves the mucous membranes
      • Strawberry tongue
      • Painful desquamation does not occur
  • Bullous impetigo:
    • Turbid or cloudy bullous fluid
  • Bullous varicella:
    • Tzanck prep or viral base reveals giant cells.
    • 5 days after the onset of varicella
  • Toxic shock syndrome:
    • Rapid development of clinical signs and symptoms
    • Mucous membrane and multiorgan involvement
  • Toxic epidermal necrolysis or drug eruption:
    • Much more common in adults
    • Severely afflicted mucous membranes
    • Full-thickness epidermal necrosis
  • Dermatologic:
    • Erythema multiforme
    • Epidermolysis hyperkeratosis
    • Epidermolysis bullosa
    • Pemphigus vulgaris
  • Scald injury
  • Secondary rash of an underlying disorder:
    • Lymphoma
    • Aspergillosis
    • Irradiation
    • Graft-versus-host reaction
    • Kawasaki disease

Treatment


Pre-Hospital


  • 9% NS fluid bolus if dehydration present
  • Initial burn treatment

Initial Stabilization/Therapy


  • Management is similar to an extensive 2nd-degree burn:
    • Involvement of large body surface area will require IV fluids.
  • Provide adequate analgesia.
  • Undress and place child on sterile linen.
  • Limit handling of child.
  • Apply moist sterile dressings.
  • Avoid excess heat loss.

Ed Treatment/Procedures


  • Topical burn creams are of no proven benefit.
  • Steroids are contraindicated.
  • IV antibiotics effective against penicillinase-resistant S. aureus:
    • Cefazolin
    • Nafcillin
    • Vancomycin if methicillin-resistant S. aureus (MRSA) suspected
  • Oral antibiotics for mild involvement:
    • Dicloxacillin
    • Erythromycin
    • Cephalexin

Medication


  • Cefazolin: 50 " “100 mg/kg/24 h IV div. QID
  • Cephalexin: 25 " “100 mg/kg/24 h PO div. QID
  • Dicloxacillin: 12 " “25 mg/kg/24 h PO div. QID
  • Erythromycin: 30 " “50 mg/kg/24 h PO div. QID
  • Nafcillin: 1 " “2 g IV q6h (peds: Newborns, 50 " “100 mg/kg/24 h IV div. q6h; children, 100 " “200 mg/kg/24 h IV div. q6h)
  • Vancomycin: 40 mg/kg/24h IV q 6 hrs

Follow-Up


Disposition


Admission Criteria
  • Children <1 yr
  • All toxic-appearing children
  • Widespread skin involvement
  • Dehydration and/or electrolyte derangement

Discharge Criteria
  • Older, well-appearing children with mild involvement
  • Oral antibiotics for 7 days
  • Follow-up within 48 hr

Issues for Referral
  • Infectious disease consultant
  • Surgeon if source needs excision/drainage

Additional Reading


  • Blyth ‚  M, Estela ‚  C, Young ‚  AE. Severe staphylococcal scalded skin syndrome in children. Burns.  2008;34:98 " “103.
  • Freedberg ‚  IM, Eisen ‚  AZ, Wolff ‚  K, et al. Fitzpatricks Dermatology in General Medicine. 6th ed. New York, NY: McGraw-Hill; 2003:195.
  • Ladhani ‚  S. Recent developments in staphylococcal scalded skin syndrome. Clin Microbiol Infect.  2001;7(6):301 " “307.
  • Patel ‚  GK, Finlay ‚  AY. Staphylococcal scalded skin-syndrome: Diagnosis and management. Am J Clin Dermatol.  2003;4:165 " “175.
  • Stanley ‚  JR, Amagai ‚  M. Pemphigus, bullous impetigo, and the staphylococcal scalded-skin syndrome. N Engl J Med.  2006;355(17):1800 " “1810.

Codes


ICD9


695.81 Ritters disease ‚  

ICD10


L00 Staphylococcal scalded skin syndrome ‚  

SNOMED


  • 200946001 Staphylococcal scalded skin syndrome (disorder)
  • 402967005 Neonatal staphylococcal scalded skin syndrome (disorder)
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