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Snake Envenomation, Emergency Medicine


Basics


Description


  • Pit viper venom:
    • Mixture of proteolytic enzymes and thrombin-like esterases:
      • Enzymes cause local muscle and subcutaneous tissue necrosis.
      • Esterases have defibrinating anticoagulant effect, leading to venom-induced consumption coagulopathy (VICC) in severe envenomations.
  • Bite location:
    • Extremity bites most common
    • Head, neck, or trunk bites more severe than bite on extremities
  • Severe envenomation:
    • Direct bite into artery or vein
    • Neurotoxic envenomations
  • Bite mark significance:
    • Pit viper bite: Classically includes 1 or 2 puncture marks
    • Nonvenomous snakes and elapids: Horseshoe-shaped row of multiple teeth marks
  • 25% of all pit viper bites are dry and do not result in envenomation.

Etiology


Venomous Snakes Indigenous to US
  • Pit vipers (Crotalinae):
    • Account for 95% of all envenomations
    • Rattlesnakes, cottonmouths, and copperheads
  • Coral snakes (Elapidae):
    • Neurotoxic
    • Western coral snakes, found in Arizona and New Mexico
    • More venomous eastern coral snakes, found in Carolinas and Gulf states

International Exotic Venomous Snakes
Occur in zoos or in owners of exotic snakes ‚  
  • 30% of all snakebites involve patients younger than 20 yr. 12% of all snakebites are 9 yr or younger.
  • Because of their low body weight, smaller children and infants are more vulnerable to severe envenomation with systemic symptoms.

Diagnosis


Signs and Symptoms


  • Local (Crotaline):
    • Classic skin changes:
      • 1 or 2 puncture wounds
      • Pain and swelling at site
    • Swelling and edema of involved extremity:
      • Within 1 hr in severe envenomations
      • Tender proximal lymph nodes
    • Ecchymosis, petechiae, and hemorrhagic vesicles develop within several hours.
  • Systemic (Crotaline):
    • Weakness, dizziness
    • Diaphoresis
    • Nausea
    • Scalp paresthesias
    • Periorbital fasciculations
    • Metallic taste
    • Severe bites can lead to:
      • Coagulopathy (VICC)
      • Hypotension
      • Pulmonary edema
      • Hematuria
      • Rhabdomyolysis
      • Renal failure
      • Cardiac dysfunction
    • Potential elevated compartment pressure in involved extremity
  • Symptoms (Crotaline):
    • Primarily neurotoxic, leading to weakness, diplopia, confusion, delayed respiratory depression:
      • Local effects may be deceivingly minimal.

History
  • Description of snake
  • Geographic location of bite

Physical Exam
Search for manifestations of bites as described above. ‚  

Essential Workup


  • Careful exam of wound site and involved extremity:
    • Essential in judging severity of envenomation
    • Mark wound margins to follow progression
  • Assess for anaphylactic reactions

Diagnosis Tests & Interpretation


Lab
  • CBC
  • Coags
  • Fibrinogen, d-dimer
  • Electrolytes, BUN/creatinine, glucose
  • Creatine phosphokinase (CPK)
  • UA
  • Type and cross-match with moderate to severe envenomation.

Imaging
Plain radiographs if foreign body suspected ‚  

Differential Diagnosis


  • Nonvenomous snakes (in the US):
    • Narrow head
    • Round pupils
    • No rattles
  • Pit vipers:
    • Triangular- or arrow-shaped head
    • Vertical or elliptical pupils
    • Heat-sensing pits just behind the nostrils and in front of eyes
    • ‚ ± Rattles
  • Coral snakes (applies only in US, not internationally):
    • "Red on yellow " ”kill a fellow " 
    • "Red on black " ”venom lack " 

Treatment


Pre-Hospital


  • Retreat well beyond striking range of snake.
  • Immobilize extremity in functional position at the level of heart.
  • Keep physical activity minimal.
  • Remove rings, watches, and all constrictive clothing.
  • It is ill-advised to transport a snake to a health care facility for identification purposes:
    • If you are close enough to get a good picture with a camera/phone, you are too close to a potentially venomous snake.
    • Even severed head can envenomate.
  • Controversies:
    • Pre-hospital local wound maneuvers are NOT recommended because they cause worse local tissue damage and increase the risk of infection. These include:
      • Incision and drainage
      • Mechanical suction devices
      • Oral suction
      • Tourniquets
      • Cryotherapy
      • Electrocution
      • Pressure immobilization
      • Incision attempts by inexperienced can lead to severe tendon, nerve, and vascular damage.

  • Envenomation more likely to be severe.
  • Severity due to relatively low body weight of small child with same volume of venom.

Initial Stabilization/Therapy


  • Airway, breathing, and circulation management (ABCs)
  • Maintain euvolemia with 0.9% normal saline (NS) to maintain renal blood flow
  • Wound monitoring
  • Immobilize bitten extremity

Ed Treatment/Procedures


  • Supportive care
  • Monitor for compartment syndrome:
    • Repeated measurements of extremity circumference every 15 " “20 min until local progression/swelling subsides.
    • A true compartment syndrome is unlikely following rattlesnake envenomation.
    • Elevated compartment pressures are treated with more antivenom, as surgical intervention with fasciotomy causes more damage to the area.
    • Surgical therapy considered only in incredibly rare cases and should only be considered in consultation with a regional poison center and medical toxicologist
  • Analgesia with IV opioids
  • Tetanus prophylaxis if needed
  • Broad-spectrum antibiotics not routinely indicated
  • Steroids not indicated except for reactions to antivenom (see below)
  • Routine use of blood products not indicated
  • Wound severity:
    • Minimal:
      • Local swelling and tenderness
    • Moderate:
      • Extremity swelling
      • Evidence of systemic toxicity
    • Severe:
      • Obvious toxicity
      • Unstable vital signs
      • Coagulopathy
      • Elapid envenomation
      • Lab abnormalities

Antivenom
  • Indications for Crotalid antivenom therapy:
    • More than minimal extremity swelling
    • Extremity swelling that is progressing
    • Clinical signs of systemic toxicity
    • Unstable vital signs
    • Coagulopathy (low platelets or fibrinogen, elevated PT)
  • CroFab:
    • Fundamental treatment for North American pit viper envenomation
    • High-affinity purified ovine Fab antibody fragment antivenom
    • CroFab causes less frequent hypersensitivity reactions than older polyvalent antivenom
    • Pediatric antivenom dose = adult antivenom dose
    • Dosing: 4 " “6 vials initially
    • Reconstitute each CroFab vial with 25 mL sterile water. Dilute in 250 mL 0.9% NaCl and infuse over 1 hr.
    • If hypotensive or with serious active bleeding, initial dose is 8 " “12 vials
    • Evaluate for envenomation control 1 hr after antivenom bolus infusion. Control is defined by stable wound appearance, improving coagulation studies, and hemodynamic stability.
    • If envenomation control achieved after 1st bolus of antivenom, may need maintenance antivenom therapy at 2 vials q6h ƒ — 3 doses.
    • If envenomation control not achieved after 1st bolus of antivenom, repeat initial bolus and reassess. Discuss with regional poison center or medical toxicologist.
  • Victims of envenomation who develop an allergic reaction to antivenom:
    • Stop infusion of antivenom
    • Administer antihistamines, corticosteroids, and fluids. Consider epinephrine for severe reactions.
    • Discussion of risks/benefits of restarting antivenom should take place with regional poison center or medical toxicologist
  • Coral snake antivenom:
    • No longer being manufactured, but stockpile exists in geographically appropriate locales.
    • Effective against more toxic eastern coral snake but not against western coral snakes
    • After proper skin testing, 3 " “5 vials of antivenin recommended.
    • Treatment complications include anaphylaxis and serum sickness.
    • Coral snake venom is neurotoxic; watch for respiratory depression, control airway
  • International exotic venomous snakes:
    • Specific antivenoms may be available at local zoos or through the Antivenom Index.

  • Proportionally more antivenin per body weight
  • Standard adult doses required

  • If mother has systemic signs of envenomation toxicity, fetus is also at risk; timely antivenom therapy is still indicated.
  • Consult obstetrician

Treatment Assistance
  • Contact local poison center 800-222-1222, medical toxicologist, local zoo, or regional herpetologist.
  • Call Antivenom Index at 602-626-6016 in Tucson, Arizona, for assistance in treatment of exotic snakes not indigenous to US

Follow-Up


Disposition


Admission Criteria
  • 24-hr observation after control of envenomation progression for patients requiring antivenom administration after pit viper bites.
  • 24-hr observation for asymptomatic patients with elapid bites.
  • ICU admission for:
    • Patients receiving antivenom
    • Evidence of moderate to severe envenomation, especially in children
    • All victims of elapid bites and for symptomatic exotic snake envenomations

Discharge Criteria
Suspicious bite that shows no signs or symptoms of envenomation for 6 " “8 hr and has normal lab panel: ‚  
  • Dry bites may be observed for 8 hr and discharged if there is no development in local toxicity and if lab studies normal.
  • Minor envenomations should be observed for 12 " “24 hr and have labs repeated 6 hr after presentation, then again before discharge.
  • Discharge with follow-up in 24 hr.

Followup Recommendations


PCP or toxicology follow-up 1 wk after antivenom therapy to assess for possible serum sickness or envenomation wound infection. ‚  

Pearls and Pitfalls


  • Avoid overly aggressive pre-hospital care interventions. It is best to rapidly transport to closest medical center.
  • Be sure to administer proper dose of antivenom in a timely fashion when clinically indicated.

Additional Reading


  • American College of Medical Toxicology, American Academy of Clinical Toxicology, American Association of Poison Control Centers, et al. Pressure immobilization after North American Crotalinae snake envenomation. Clin Toxicol (Phila).  2011;49:881 " “882.
  • Corneille ‚  MG, Larson ‚  S, Stewart ‚  RM, et al. A large single-center experience with treatment of patients with crotalid envenomations: Outcomes with and evolution of antivenin therapy. Am J Surg.  2006;192:848 " “852.
  • Cox ‚  MR, Reeves ‚  JK, Smith ‚  KM. Concepts in Crotaline snake envenomation management. Orthopedics.  2006;29(12):1083 " “1087.
  • Cumpston ‚  KL. Is there a role for fasciotomy in Crotalinae envenomations in North America? Clin Toxicol (Phila).  2011;49:351 " “365.
  • Lavonas ‚  EJ, Ruha ‚  AM, Banner ‚  W, et al. Unified treatment algorithm for the management of crotaline snakebite in the United States: Results of an evidence-informed consensus workshop. BMC Emerg Med  2011;11:2.

Codes


ICD9


989.5 Toxic effect of venom ‚  

ICD10


  • T63.001A Toxic effect of unsp snake venom, accidental, init
  • T63.011A Toxic effect of rattlesnake venom, accidental, init
  • T63.021A Toxic effect of coral snake venom, accidental, init
  • T63.061A Toxic effect of venom of N & S American snake, acc, init
  • T63.091A Toxic effect of venom of snake, accidental, init

SNOMED


  • 238457002 Venomous snake bite (finding)
  • 217659000 poisoning due to rattlesnake venom (disorder)
  • 217653004 poisoning due to coral snake venom (disorder)
  • 217652009 poisoning due to copperhead snake venom (disorder)
  • 217661009 poisoning due to viper venom (disorder)
  • 217662002 Poisoning due to water moccasin venom (disorder)
  • 241811004 poisoning due to bite of unidentified snake (disorder)
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