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Sepsis

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  • Continuum from sepsis to multisystem organ failure (MSOF) is affected by age-specific physiologic variables in children.

  • SIRS in pediatric population requires that abnormality in temperature or WBC be present (2)[C].

  • Drug metabolism is reduced in children with severe sepsis. Monitor drug toxicity labs to prevent adverse effects.

‚  
Geriatric Considerations

Often more difficult to diagnose; change in mental status/behavior may be the only early manifestation.

‚  

EPIDEMIOLOGY


Incidence
  • 3/1,000 population
  • Increasing incidence; 750,000 new cases annually in the United States
  • 2% of hospitalized patients

ETIOLOGY AND PATHOPHYSIOLOGY


  • Multifactorial: Widespread endothelial dysfunction, dysregulation of nitric oxide production, and activation of coagulation cascade leads to maldistribution of blood flow, tissue hypoperfusion, and resulting to organ dysfunction.
  • An imbalance between proinflammatory and anti-inflammatory mediators leads to systemic tissue damage and significant immunosuppression.
  • Causative organisms:
    • Gram-positive bacteria (most common): Staphylococcus sp., Streptococcus sp., Enterococcus sp.
    • Gram-negative: Escherichia coli, Klebsiella sp., Proteus sp., Pseudomonas sp. and anaerobic bacteria
    • Fungi: Candida sp. (incidence increased 207% from 1976 to 2000)
    • Causative organism not identified in 50 " “75% of cases
  • Common sites of infection: respiratory tract (most common), urinary tract, gastrointestinal tract, skin/soft tissue, CNS, bacteremia

RISK FACTORS


  • Extremes of age (very young or age >60 years)
  • Ethnicity: African American
  • Comorbidities: COPD, congestive heart failure (CHF), cancer, diabetes, and renal insufficiency/failure
  • Immunosuppression
  • Bacteremia
  • Community-acquired pneumonia
  • Complicated labor and delivery: premature labor and/or premature rupture of membranes, untreated maternal group B strep colonization
  • Nosocomial factors: surgical site infections, indwelling catheters

GENERAL PREVENTION


  • Vaccination: pneumococcal vaccine in children and also adults who are ≥65 years or with comorbidities placing them at high risk for disease. Haemophilus influenzae type B (infants, young children), influenza (H1N1 in pregnant women), meningococcal vaccine
  • Ž ³-Globulin for hypo- or agammaglobulinemia
  • Treat group B strep carriers during labor.
  • Regular hand washing, sterile technique for catheter placement, appropriate glove use
  • Antibiotic prophylaxis for recommended surgical procedures

COMMONLY ASSOCIATED CONDITIONS


  • Immunologic: neutropenia, HIV, hypo/agammaglobulinemia, complement deficiency, splenectomy, immunosuppressants (corticosteroids, chemotherapy, TNF-α antagonists)
  • Diabetes, alcoholism, malignancy, cirrhosis, burns, multiple trauma, IV drug abuse, malnutrition

DIAGNOSIS


HISTORY


  • Past medical, surgical, social, occupational, and travel history to identify risk factors and potential source
  • General symptoms
    • Fever, chills, rigors, myalgias
    • Mental status changes: restlessness, agitation, confusion, delirium, lethargy, stupor, coma
  • Specific symptoms (relative to primary source)
    • Respiratory: cough, sputum production, dyspnea, pleuritic chest pain
    • Urinary: dysuria, frequency, urgency, flank pain
    • Intra-abdominal: nausea, vomiting, diarrhea, constipation, abdominal pain
    • CNS: stiff neck, headache, photophobia, focal neurologic signs

PHYSICAL EXAM


  • Assess vital signs: hyper/hypothermia, tachycardia, tachypnea, hypotension
  • Signs of poor perfusion: central venous hypoxia, delayed capillary refill, cyanosis, mottled skin
  • Signs of target organ involvement: jaundice, skin lesions (erythema, petechiae, embolic lesions, purpura)

DIFFERENTIAL DIAGNOSIS


  • Bacteremia without sepsis
  • Viral, rickettsial, spirochetal, and protozoal diseases
  • Collagen vascular diseases, vasculitides, pancreatitis, myocardial infarction, CHF, pulmonary embolism, thrombotic thrombocytopenic purpura (TTP), hemolytic uremic syndrome (HUS), thyrotoxicosis, adrenal insufficiency, poisoning, drug reaction

DIAGNOSTIC TESTS & INTERPRETATION


Initial Tests (lab, imaging)
  • Two sets of blood cultures to document presence of bacteremia (not required for diagnosis; 50% of blood cultures are negative in severe sepsis/septic shock). Obtain cultures before antibiotic therapy. Do not delay the start of antibiotic therapy (1)[B].
  • Cultures/Gram stains from potential sites of infection (sputum, urine, pleural fluid, ascites, CSF, wound)
  • CBC with differential, CRP, coagulation profile, comprehensive metabolic profile, lactic acid level, arterial blood gas, urinalysis
  • Radiographic studies (plain films, ultrasound, CT, or MRI) to confirm source of infection

Diagnostic Procedures/Other
  • Aspiration, biopsy, and/or drainage of potentially infected body sites (pleural cavity, peritoneal cavity, biliary tree, CSF, abscess). Interventional radiology techniques to aid in specimen collection as needed
  • Echocardiogram if concern for endocarditis

Test Interpretation
  • Common findings: leukocytosis, bandemia (>10%), hyperglycemia, metabolic acidosis, mild hyperbiliru-binemia, hypoxemia, respiratory alkalosis, proteinuria
  • Less common findings (more severe disease): leukopenia, anemia, thrombocytopenia, coagulopathy, azotemia, hypoglycemia, acidosis

TREATMENT


GENERAL MEASURES


  • Assess oxygenation and supplement as needed. Intubate for respiratory failure.
  • Invasive monitoring for management of hemodynamic instability using goal-directed therapy.
  • Adequate volume resuscitation (30 mL/kg crystalloid for hypotension) within 3 hours of presentation and vasopressors for refractory hypotension (within 6 hours of presentation). Targets are:
    • Central venous pressure (CVP) 8 to 12 mm Hg, MAP ≥65 mm Hg, urine output ≥0.5 mL/kg/hr (1)[A]
    • Central/mixed venous O2 saturation ≥70%/65%
      • If goals not met with fluid resuscitation and/or vasopressor, transfuse PRBCs to achieve hematocrit >30%; if still <70%, add dobutamine (1)[C].
  • Identify source of infection and remove septic foci:
    • Early surgical consultation for acute abdomen, empyema, necrotizing fasciitis
    • Interventional radiology consultation for guided drainage
  • Transfuse PRBCs, platelets, and/or fresh frozen plasma for coagulopathic complications or in association with planned procedures.
    • Transfuse PRBC if Hgb <7.0 to target Hgb of 7.0 to 9.9 g/dL in absence of tissue hypoperfusion, ischemic coronary artery disease, or acute hemorrhage (1)[B]
  • Stress ulcer and DVT prophylaxis (1)[B]
  • Glucose control ( ≥180 mg/dL) (1)[A]

MEDICATION


First Line
  • Fluid management
    • Initial therapy: 30 mL/kg of crystalloids (1)[B]
    • Consider adding albumin for patients requiring large volumes of crystalloid (1)[C],(3)[B].
  • Vasopressors
    • Norepinephrine 0.01 to 3 Ž ¼g/kg/min (1,4)[B].
    • Low-dose dopamine for renal protection is not recommended (1)[C].
  • Initiate broad-spectrum antibiotic within an hour of recognizing severe sepsis or septic shock (1,4)[A]:
    • Adult (Pseudomonas not suspected): vancomycin (loading dose 25 mg/kg) + Gram-negative coverage (3rd-generation cephalosporin, Ž ²-lactam/ Ž ²-lactamase inhibitor, or carbapenem)
    • Adult (Pseudomonas suspected): vancomycin + two agents aimed at resistant Gram-negative bacteria ceftazidime/cefepime/carbapenem/piperacillin " “tazobactam + either fluoroquinolone (ciprofloxacin) or aminoglycoside (gentamicin/amikacin) depending on local hospital sensitivities
    • Nonimmunocompromised child: 3rd-generation cephalosporin
    • Neonate (<7 days old): ampicillin and gentamicin (5)[B]
    • Consider narrowing antibiotic regimen at 48 to 72 hours based on culture results. Anticipate antibiotic course to last 7 to 10 days (1)[B].
  • Antifungals if fungal infection suspected: long-term antibiotic use, TPN, GI surgery
    • Adjuvant intravenous immunoglobulins (IVIGs) may reduce mortality in adults (6)[B].

ALERT

  • Possible interactions

    • Aminoglycosides: increase in nephrotoxicity with enflurane, cisplatin, and possibly vancomycin; ototoxicity with loop diuretics; paralysis with neuromuscular-blocking agents

    • Adjust doses for renal and/or hepatic impairment.

  • The European Society of Intensive Care Medicine statement in critically ill patients

    • Hydroxyethyl starches (HESs) with MW ≥200 kDA and/or degree of substitution >0.4 are not recommended in severe septic patients (1)[C].

    • Hyperoncotic solutions should not be used in fluid resuscitation.

‚  
Pregnancy Considerations

Ž ²-Lactam antibiotics, macrolides, and metronidazole generally are considered safe.

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Second Line
  • Vasopressors: epinephrine, vasopressin
  • Consider IV hydrocortisone 200 mg per day if the patient is poorly responsive to both IV fluid resuscitation and vasopressors (1)[C].
  • Inotropic therapy with dobutamine may benefit patients with myocardial dysfunction or ongoing hypoperfusion despite adequate intravascular volume and MAP (1)[C].

ADDITIONAL THERAPIES


Intermittent hemodialysis or continuous veno-venous hemofiltration for renal failure ‚  

SURGERY/OTHER PROCEDURES


Debride necrotic tissues: Drain or remove abscesses or other foci of infection. ‚  

INPATIENT CONSIDERATIONS


Admission Criteria/Initial Stabilization
ICU care ‚  
IV Fluids
Aggressive fluid resuscitation with normal saline can result in nongap metabolic acidosis. Not likely with lactated Ringer solution. ‚  

ONGOING CARE


FOLLOW-UP RECOMMENDATIONS


Patient Monitoring
  • Place arterial line and central venous catheter in unstable patients
  • Chem " “7 and CBC daily; lactate, mixed venous oxygen saturation q2 " “4h in initial resuscitation period, and daily INR/PTT if DIC is a concern.
  • Follow antibiotic drug levels (vancomycin, gentamicin).
  • Foley catheter placement to monitor urine output

DIET


NPO if intubation considered; otherwise, enteral feeds are preferred to preserve GI mucosal integrity (1)[C]. ‚  

PATIENT EDUCATION


www.nlm.nih.gov/medlineplus/sepsis.html ‚  

PROGNOSIS


Mortality is 10 " “50% overall. Poor prognostic factors include the inability to mount a fever (>40 ‚ °C), nonurinary source of infection, nosocomial infection, inappropriate antibiotic coverage, and certain comorbidities (AIDS, cancer, immunosuppression). ‚  

COMPLICATIONS


GI hemorrhage, DIC, hyperglycemia, de novo embolic foci of infection, ARDS, multiorgan failure, death ‚  

REFERENCES


11 Dellinger ‚  RP, Levy ‚  MM, Rhodes ‚  A, et al. Surviving sepsis campaign: international guidelines for management of severe sepsis and septic shock: 2012. Crit Care Med.  2013;41(2):580 " “637.22 Goldstein ‚  B, Giroir ‚  B, Randolph ‚  A. International pediatric sepsis consensus conference: definitions for sepsis and organ dysfunction in pediatrics. Pediatric Critical Care Medicine.  2005;6(1):2 " “8.33 Raghunathan ‚  K, Shaw ‚  A, Nathanson ‚  B, et al. Association between the choice of IV crystalloid and in-hospital mortality among critically ill adults with sepsis. Crit Care Med.  2014;42(7):1585 " “1591.44 De Backer ‚  D, Biston ‚  P, Devriendt ‚  J, et al. Comparison of dopamine and norepinephrine in the treatment of shock. N Engl J Med.  2010;362(9):779 " “789.55 Paul ‚  M, Lador ‚  A, Grozinsky-Glasberg ‚  S, et al. Beta lactam antibiotic monotherapy versus beta lactam-aminoglycoside antibiotic combination therapy for sepsis. Cochrane Database Syst Rev.  2014;(1):CD003344.66 Stockmann ‚  C, Spigarelli ‚  MG, Campbell ‚  SC, et al. Considerations in the pharmacologic treatment and prevention of neonatal sepsis. Paediatr Drugs.  2014;16(1):67 " “81.

ADDITIONAL READING


  • Alejandria ‚  MM, Lansang ‚  MA, Dans ‚  LF, et al. Intravenous immunoglobulin for treating sepsis, severe sepsis and septic shock. Cochrane Database Syst Rev.  2013;(9):CD001090.
  • Levy ‚  MM, Fink ‚  MP, Marshall ‚  JC, et al. 2001 SCCM/ESICM/ACCP/ATS/SIS International Sepsis Definitions Conference. Crit Care Med.  2003;31(4):1250 " “1256.
  • Venkataraman ‚  R, Kellum ‚  JA. Sepsis: update in the management. Adv Chronic Kidney Dis.  2013;20(1):6 " “13.
  • Wiener ‚  RS, Wiener ‚  DC, Larson ‚  RJ. Benefits and risks of tight glucose control in critically ill adults: a meta-analysis. JAMA.  2008;300(8):933 " “944.

CODES


ICD10


  • A41.9 Sepsis, unspecified organism
  • R65.10 SIRS of non-infectious origin w/o acute organ dysfunction
  • R65.20 Severe sepsis without septic shock
  • R65.21 Severe sepsis with septic shock
  • A41.59 Other Gram-negative sepsis
  • A41.51 Sepsis due to Escherichia coli [E. coli]
  • A41.52 Sepsis due to Pseudomonas
  • A41.2 Sepsis due to unspecified staphylococcus
  • A41.81 Sepsis due to Enterococcus
  • R65.11 SIRS of non-infectious origin w acute organ dysfunction

ICD9


  • 995.91 Sepsis
  • 995.90 Systemic inflammatory response syndrome, unspecified
  • 038.9 Unspecified septicemia
  • 785.52 Septic shock
  • 995.92 Severe sepsis
  • 038.10 Staphylococcal septicemia, unspecified
  • 038.42 Septicemia due to escherichia coli [E. coli]
  • 038.0 Streptococcal septicemia
  • 038.49 Other septicemia due to gram-negative organisms
  • 038.2 Pneumococcal septicemia [Streptococcus pneumoniae septicemia]
  • 038.43 Septicemia due to pseudomonas

SNOMED


  • 91302008 Sepsis (disorder)
  • 238149007 systemic inflammatory response syndrome (disorder)
  • 10001005 Bacterial septicemia (disorder)
  • 76571007 Septic shock (disorder)
  • 447894003 Sepsis due to Staphylococcus (disorder)
  • 310669007 Septicemia due to enterococcus (disorder)
  • 448418006 Sepsis due to Streptococcus (disorder)

CLINICAL PEARLS


  • Treat sepsis aggressively with fluid support (with hemodynamic monitoring and pressor support if necessary) early use of broad-spectrum antimicrobial therapy and removal/drainage of foci of infection.
  • Despite aggressive treatment, overall mortality is high (10 " “50%) in patients with severe sepsis/septic shock.
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