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Secretion of Inappropriate Antidiuretic Hormone (SIADH)

para>Resulting abnormal urinary free water retention leads to dilutional hyponatremia (total body sodium levels may be normal or near normal, but the patient 's total body water is increased).
  • Often secondary to medications but may be associated with an underlying disorder, such as neoplasm, pulmonary disorder, or CNS system disease

  • Synonym(s): syndrome of inappropriate secretion of ADH

  • Epidemiology


    Incidence
    • Often found in the hospital setting, where incidence can be as high as 35%
    • Predominant age: elderly
    • Predominate sex: females > males

    Etiology and Pathophysiology


    • Drugs:
      • Antidepressants (e.g., monoamine oxidase inhibitors [MAOIs], tricyclics, SSRIs)

      • Oral hypoglycemics (e.g., chlorpropamide, metformin)

      • Antineoplastic drugs (e.g., vincristine, vinblastine, cisplatin, cyclophosphamide)

      • Antipsychotic agents (e.g., phenothiazines, thioridazine, haloperidol)

      • Analgesics (e.g., NSAIDs)

      • Antiepileptics (e.g., carbamazepine, oxcarbazepine, valproic acid)

      • Others (e.g., vasopressin, DDAVP, ecstasy, oxytocin,α-interferon)

    • Neoplasms (ectopic ADH production):
      • Small cell carcinoma of the lung

      • Oat cell carcinoma of the lung

      • Hodgkin disease

      • Pancreatic carcinoma

      • Thymoma

      • Mesothelioma

      • Bronchogenic carcinoma

    • Infectious diseases:
      • Meningitis

      • Encephalitis

      • Pneumonia

      • Pulmonary tuberculosis (TB)

      • Rocky Mountain spotted fever

      • HIV infection

    • Miscellaneous cardiopulmonary conditions:
      • Asthma

      • Atelectasis

      • Myocardial infarction

      • Vascular diseases

    • Other:
      • CNS injury

      • Mechanical ventilation

      • Multiple sclerosis

      • Guillain-Barre syndrome

      • Lupus erythematosus

      • Porphyria

      • Hypothyroidism, myxedema

    • Idiopathic

    Genetics
    No known genetic pattern ‚  

    Risk Factors


    • Use of predisposing drugs
    • Advanced age
    • Postoperative status
    • Institutionalization

    General Prevention


    • Search for cause, if unknown.
    • Administration of 0.9% sodium chloride in adults during the perioperative period is recommended (if hypernatremia is not present).
    • Monitor electrolytes in postoperative patients to determine if fluid intake needs restriction.
    • Reduce/change medications, if drug-induced.
    • Lifelong restriction of fluid intake

    Commonly Associated Conditions


    See "Etiology. "  ‚  

    Diagnosis


    History


    Early symptoms: ‚  
    • Fatigue
    • Anorexia
    • Nausea
    • Vomiting
    • Diarrhea
    • Headaches
    • Myalgias
    • Increased thirst

    Physical Exam


    Late/severe hyponatremia (serum Na <100 " “115 mEq/L) ‚  
    • Altered mental status
    • Confusion
    • Lethargy
    • Seizures
    • Psychosis
    • Coma
    • Death

    Differential Diagnosis


    • Postoperative complications:
      • Usually after major abdominal/thoracic surgery

      • Caused by nonosmotic release of ADH, probably mediated by pain afferents

      • ADH increased by pain and narcotics

    • Postprostatectomy syndrome
      • Irrigating solution must be nonconducting (i.e., electrolyte-free).

      • D5W absorbed

    • Psychogenic polydipsia
      • Active therapy rarely is needed.

      • Diuresis occurs when intake is stopped.

      • Intake usually >10 L/day

      • Interaction with other psychotropic drugs

    • Acute (usually in children)
      • Swallowing water during swimming

      • Diluted formula

      • Tap water enemas

    • Endocrine
      • Addison disease

      • Hypothyroidism

    • Spurious hyponatremia: caused by increased serum glucose, cholesterol, or proteins
    • Appropriate ADH secretion and hyponatremia with decreased effective arterial blood volume (e.g., congestive heart failure [CHF], nephrotic syndrome, cirrhosis)
    • Cerebral salt-wasting syndrome (hyponatremia, extracellular fluid depletion, CNS insult)

    Diagnostic Tests & Interpretation


    • Serum Na level: low
    • Serum osmolality: low
    • Urine osmolality: high
    • Urinary Na concentration: high (losing sodium, rather than retaining)
    • Serum ADH level: high
    • Not usually required for diagnosis but to assess for other concerns:
      • Uric acid

      • Serum glucose; BUN; creatinine

      • Thyroid function

      • Morning cortisol


    Treatment


    General Measures


    • Fluid restriction (800 " “1,000 mL/day) is the main form of treatment (1)[C].
    • Mildly symptomatic (serum Na >125 mEq/L [>125 mmol/L]): Restrict fluid to 800 " “1,000 mL/day.
    • Acute (<48 hours duration) or symptomatic (altered mental status, seizure, coma)
      • Hypertonic saline (3% normal saline) bolus

      • Diuresis with loop diuretics

      • Decrease oral free water to maintenance.

      • Increase oral salt.

      • Correct serum Na deficit (mEq Na deficit = [desired Na ’ ˆ ’ actual Na] ƒ — 0.5 ƒ — body weight [kg]).

      • Increase serum Na slowly with hypertonic saline by 4 " “6 mEq/L over 4 " “6 hours (not to exceed 9 mEq/L in a 24-hour period) (2,3).


    Medication


    • Diuretics: furosemide (Lasix) + hourly NaCl and KCl replacement
      • Requires frequent monitoring (see "Patient Monitoring " )

      • Treatment of choice for acute management

    • IV saline to increase serum Na cautiously (4) [B]:
      • Goal sodium increase is <9 mEq/L in any 24-period.

      • If serum Na <120 mEq/L or severe neurologic symptoms; consider use of hypertonic saline to increase serum Na by 4 " “6 mEq/L over the first 4 " “6 hours.

    • Contraindications: Avoid fluids in CHF, nephrotic syndrome, or cirrhosis.
    • Precautions: Overly rapid correction (>9 " “10 mEq/L/day) can increase risk for the following:
      • CHF

      • Subdural and intracerebral hemorrhage

      • Permanent CNS damage, especially with serum Na <120 mEq/L (<120 mmol/L)

      • Demyelination syndrome

    • Vasopressin-2 receptor antagonist (the vaptans: tolvaptan, conivaptan) (4),(5)[C]
      • Good efficacy and safety profiles in the treatment of mild to moderate hyponatremia due to SIADH

      • Avoid tolvaptan in patients with liver disease.

    • Demeclocycline (second line) (6)[C]
      • Blocks ADH at renal tubule; produces nephrogenic diabetes insipidus

      • Dosage for long-term management: 300 " “600 mg PO BID

      • Onset of action within 1 week; therefore, not best for acute management.


    Alert
    Increase Na levels slowly, no more than 9 mEq/L/24 hr, to prevent complications such as central pontine myelinosis (CPM) (7)[C]. ‚  

    Ongoing Care


    Follow-up Recommendations


    Patient Monitoring
    • Careful continuous clinical and laboratory monitoring of hyponatremic state during acute phase:
      • Hourly urine output

      • Urine Na

      • Serum Na and potassium (K)

    • Chronic management: Monitor underlying cause, as needed.

    Diet


    May need increased salt/decreased water intake, depending on cause ‚  

    Patient Education


    Diet and fluid restrictions ‚  

    Prognosis


    • Depends on underlying cause; in general, higher morbidity and mortality in hospitalized patients with hyponatremia
    • If symptomatic (seizure, coma): high mortality due to cerebral edema if serum Na <120 mEq/L (<120 mmol/L)

    Complications


    • Osmotic demyelination: central pontine and extrapontine irreversible myelinolysis (2,7)
    • Chronic hyponatremia: usually <120 mEq/L (<120 mmol/L)
    • Complications of overly rapid correction (see "Treatment, "  precautions)
    • Chronic hyponatremia is associated with osteoporosis (8)[C].

    References


    1.Ellison ‚  DH, Berl ‚  T. Clinical practice. The syndrome of inappropriate antidiuresis. N Engl J Med.  2007;356(20):2064 " “2072. ‚  [View Abstract]2.Adrogue ‚  HJ, Madias ‚  NE. The challenge of hyponatremia. J Am Soc Nephrol.  2012;23(7):1140 " “1148. ‚  [View Abstract]3.Sterns ‚  RH, Nigwekar ‚  SU, Hix ‚  JK. The treatment of hyponatremia. Semin Nephrol.  2009;29(3):282 " “299. ‚  [View Abstract]4.Esposito ‚  P, Piotti ‚  G, Bianzina ‚  S, et al. The syndrome of inappropriate antidiuresis: pathophysiology, clinical management and new therapeutic options. Nephron Clin Pract.  2011;119(1):c62 " “c73. ‚  [View Abstract]5.Friedman ‚  B, Cirulli ‚  J. Hyponatremia in critical care patients: frequency, outcome, characteristics, and treatment with the vasopressin V(2)-receptor antagonist tolvaptan. J Crit Care.  2013;28(2):219.e1 " “e12. ‚  [View Abstract]6.Sherlock ‚  M, Thompson ‚  CJ. The syndrome of inappropriate antidiuretic hormone: current and future management options. Eur J Endocrinol.  2010;162(Suppl 1):S13 " “S18. ‚  [View Abstract]7.Fleming ‚  JD, Babu ‚  S. Images in clinical medicine. Central pontine myelinolysis. N Engl J Med.  2008;359(23):e29. ‚  [View Abstract]8.Verbalis ‚  JG, Barsony ‚  J, Sugimura ‚  Y, et al. Hyponatremia-induced osteoporosis. J Bone Miner Res.  2010;25(3):554 " “563. ‚  [View Abstract]

    Additional Reading


    See Also


    Hyponatremia ‚  

    Codes


    ICD10


    • E22.2 Syndrome of inappropriate secretion of antidiuretic hormone

    ICD09


    • 253.6 Other disorders of neurohypophysis

    SNOMED


    • 55004003 Syndrome of inappropriate vasopressin secretion (disorder)

    Clinical Pearls


    • Fluid restriction to 600 " “800 mL/day ƒ — 2 " “3 days will result in weight loss and correction of hyponatremia and salt wasting in SIADH. Fluid restriction fails to correct hyponatremia and Na wasting in salt-losing renal disease.
    • Cerebral salt wasting is a controversial disease entity and is similar to SIADH. However, patients with SIADH are euvolemic, whereas patients with cerebral salt wasting are hypovolemic. The only real way to establish the diagnosis is through fluid restriction. Serum urate and fractional excretion of urate will be corrected with fluid restriction in SIADH, but will not correct in cerebral salt wasting.
    • CPM is a cerebral demyelination syndrome that causes quadriplegia, pseudobulbar palsy, seizures, coma, and death. It is caused by an overly rapid rate of Na correction.
    • Safe correction of hyponatremia is important. Online calculators are available: www.medcalc.com/sodium.html.
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