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Salicylate Poisoning, Emergency Medicine


Basics


Description


  • Respiratory alkalosis and metabolic acidosis:
    • Secondary to inhibition of Krebs cycle and uncoupling of oxidative phosphorylation
  • Dehydration, hyponatremia or hypernatremia, hypokalemia, hypocalcemia:
    • Owing to increased sweating, vomiting, tachypnea
  • Noncardiogenic pulmonary edema:
    • Because of toxic effect of salicylate on pulmonary endothelium resulting in extravasation of fluids
  • Salicylate pharmacokinetics change from first order to zero order in overdose setting; i.e., a small dosage
    increment results in a large increase in salicylate concentration.

  • Greater morbidity
  • Respiratory distress/altered mental status indicative of severe toxicity
  • Diagnosis of salicylate intoxication delayed because underlying disease states mask signs and symptoms; e.g., CHF

  • Children exhibit faster onset and more severe signs and symptoms than adults:
    • Results from salicylate being distributed more quickly into target organs such as brain, kidney, and liver
  • Respiratory alkalosis (hallmark of salicylate poisoning in adults) may not occur in children.
  • Metabolic acidosis occurs more quickly in children than in adults.
  • Hypoglycemia more common than hyperglycemia
  • Ingestion of more than "a taste " ť of oil of wintergreen (98% methyl salicylate) by children <6 yr or >4 mL of oil of wintergreen by patients >6 yr warrants ED assessment.

Etiology


Sources of salicylate: ‚  
  • Aspirin:
    • Ingestion of >150 mg/kg can cause serious toxicity
  • Oil of wintergreen:
    • Any exposure should be considered dangerous.
  • Bismuth subsalicylate
  • Salicylsalicylic acid (salsalate)

Diagnosis


Signs and Symptoms


  • GI:
    • Nausea
    • Vomiting
    • Epigastric pain
    • Hematemesis
  • Pulmonary:
    • Tachypnea
    • Noncardiogenic pulmonary edema
  • CNS:
    • Tinnitus
    • Deafness
    • Delirium
    • Seizures
    • Coma

History
  • Ask if taking aspirin or aspirin products:
    • Many patients do not list aspirin among their regular medications, may not consider aspirin a medication.
  • Patients may not know the difference between aspirin, acetaminophen, and the OTC NSAIDs

Essential Workup


  • Salicylate level:
    • At presentation and then q2h until level begins to decline
    • Verify that units are correct, generally mg/dL.
  • Watch for recurrence of signs of salicylate toxicity and increasing levels even after levels have declined due to intestinal absorption of enteric-coated products and salsalate

Guidelines for Assessing Severity of Salicylate Poisoning ‚  
  • Acute ingestion of:
    • <150 mg/kg or <6.5 g of aspirin equivalent " ”considered nontoxic
    • 150 " “300 mg/kg " ”mild to moderately toxic
    • >300 mg/kg " ”potentially lethal
  • In the chronic overdose setting:
    • Manage patient on clinical findings and not solely on levels
    • Clinical findings are better indication of severity than plasma salicylate levels
    • No valid nomogram exists for salicylate level interpretation
    • Salicylate levels needed to achieve anti-inflammatory effect (20 " “25 mg/dL) approach toxic levels
    • Enteric-coated aspirin absorbed in intestine; peak level delayed

Diagnosis Tests & Interpretation


Lab
  • Arterial blood gas (ABG):
    • Respiratory alkalosis
    • Metabolic acidosis
  • CBC
  • Electrolytes, BUN/creatinine, glucose:
    • Anion-gap metabolic acidosis
    • Hypokalemia
    • Baseline renal function
  • Urinalysis:
    • Urine pH
  • PT/PTT with significant ingestions
  • Ferric chloride test:
    • Purple if salicylate present
    • Positive 30 min postingestion
  • In the presence of salicylate, Phenistix turn brown-purple; may detect concentrations as low as 20 mg/dL

Imaging
  • Abdominal flat-plate radiograph for concretions
  • Chest radiograph for pulmonary edema

Differential Diagnosis


  • Acute salicylate poisoning:
    • Consider with change in mental status, unexplained noncardiogenic pulmonary edema, mixed acid " “base disorder.
    • Methanol
    • Ethylene glycol
    • Conditions causing noncardiogenic pulmonary edema
  • Chronic salicylate poisoning:
    • Impending myocardial infarction
    • Alcohol withdrawal
    • Organic psychoses
    • Sepsis
    • Dementia

Treatment


Pre-Hospital


In suspected overdose settings, medication bottles must be brought in for review ‚  

Initial Stabilization/Therapy


  • Management of airway, breathing, and circulation (ABCs)
  • Naloxone, thiamine, glucose (or Accu-Chek) for altered mental status
  • IV rehydration with 0.9% normal saline (NS) for hypotension

Ed Treatment/Procedures


  • Morbidity from chronic salicylate poisoning may be greater than from acute poisoning.
  • Aggressively manage all salicylate intoxication.

Gastric Decontamination
  • Administer activated charcoal in alert patients.
  • Whole-bowel irrigation of theoretical benefit:
    • For concretions visible on abdominal radiograph
    • For ingestion of sustained-release preparation
    • If salicylate levels continue to increase despite appropriate management
    • Do not use in patients who may develop altered mental status

Enhanced Elimination
  • Alkalinization:
    • Enhances elimination of ionized salicylate
    • Indications:
      • Acidosis
      • Presence of symptoms
      • Elevated salicylate levels
    • 1 or 2 ampules of sodium bicarbonate followed by
      IV D5W 1L with 3 ampules of sodium
      bicarbonate:
      • Goal: Urine pH of 7.5 " “8 at the rate of 3 " “6 mL/kg/h
      • Add 20 " “40 mEq KCl per liter to avoid hypokalemia
      • Avoid fluid overload with CHF or CAD
      • Closely monitor serum potassium
  • Indications for hemodialysis include:
    • CHF
    • Noncardiogenic pulmonary edema
    • CNS depression
    • Seizures
    • Unstable vital signs
    • Severe acid " “base disorder
    • Hepatic compromise
    • Coagulopathy
    • Underlying disease state compromising elimination of salicylate
    • Absolute salicylate level should not be used as sole criterion for deciding to dialyze without considering patients clinical status unless level is >80 " “100 mg/dL in acute ingestion.
  • Threshold to dialyze is lower in patients with chronic overdose.

Medication


  • Activated charcoal slurry: 1 " “2 g/kg up to 90 g PO
  • Dextrose: D50W 1 amp (50 mL or 25 g) (peds: D25W 2 " “4 mL/kg) IV
  • Naloxone (Narcan): 2 mg (peds: 0.1 mg/kg) IV or IM initial dose
  • Thiamine (vitamin B1): 100 mg (peds: 50 mg) IV or IM

Follow-Up


Disposition


Admission Criteria
  • Monitor patients with salicylate levels >25 mg/dL until level drops <25 mg/dL and symptoms abate.
  • Salicylate levels increasing after having trended downward to nontoxic levels:
    • In patients who ingest sustained-release aspirin, enteric-coated aspirin, and any aspirin product with delayed absorption
  • ICU admission for altered mental status, metabolic acidosis, pulmonary edema

Discharge Criteria
Repetitive salicylate levels <25 mg/dL and resolution of symptoms ‚  

Follow-Up Recommendations


  • Psychiatric referral for intentional ingestions
  • Close primary care follow-up for chronic ingestions

Pearls and Pitfalls


  • Patients need to maintain their respiratory drive to reverse acidemia, respiratory acidosis:
    • Do not intubate prematurely.
    • It is extremely difficult to achieve and maintain mechanical hyperventilation in these patients.
  • Salicylate poisoning may result from topical exposure to salicylate-containing lotions or creams, rectal suppositories, oral antidiarrheal preparations.
  • Salicylate levels may trend downward only to begin increasing again due to absorption of product from the intestine or from a salicylate bezoar in the gut.

Additional Reading


  • Kent ‚  K, Ganetsky ‚  M, Cohen ‚  J, et al. Non-fatal ventricular dysrhythmias associated with severe salicylate toxicity. Clin Toxicol (Phila).  2008;46:297 " “299.
  • Stolbach ‚  AI, Hoffman ‚  RS, Nelson ‚  LS. Mechanical ventilation was associated with acidemia in a case series of salicylate-poisoned patients. Acad Emerg Med.  2008;15:866 " “869.
  • West ‚  PL, Horowitz ‚  BZ. Delayed recrudescence to toxic salicylate concentrations after salsalate overdose. J Med Toxicol.  2010;6:150 " “154.

Codes


ICD9


  • 276.2 Acidosis
  • 276.3 Alkalosis
  • 965.1 Poisoning by salicylates

ICD10


  • E87.2 Acidosis
  • E87.3 Alkalosis
  • T39.011A Poisoning by aspirin, accidental (unintentional), init
  • T39.012A Poisoning by aspirin, intentional self-harm, init encntr

SNOMED


  • 7248001 Poisoning by salicylate (disorder)
  • 35528000 Metabolic acidosis due to salicylate (disorder)
  • 111378004 Respiratory alkalosis
  • 290145004 Intentional aspirin poisoning (disorder)
  • 290149005 Salicylic acid salt poisoning of undetermined intent (disorder)
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