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Polycythemia, Emergency Medicine


Basics


Description


  • Increase in hemoglobin (Hgb) above the normal range:
    • Men: Hgb >17.5 g/dL, hematocrit (Hct) >52%
    • Women: Hgb >16 g/dL, Hct >48%
  • Symptoms are related to blood viscosity, which increases exponentially at Hct >60%.

Etiology


  • Relative (apparent) polycythemia:
    • Resulting from decrease in plasma volume
    • Acute: Dehydration
    • Chronic: Gaisbock syndrome (stress polycythemia): Obese, hypertensive, middle-aged smokers
  • Primary erythrocytosis:
    • Polycythemia vera (PV): A stem cell disorder characterized by panhyperplasia of all bone marrow elements leading to increased production of RBCs, WBCs, and platelets. Erythrocytosis is the most prominent feature:
      • Mutation in the tyrosine kinase (JAK2), which acts in signaling pathways of the EPO-receptor, rendering those cells hypersensitive to erythropoietin
      • Median age 60, 5% <40, peak in 70s
      • Higher in Ashkenazi Jews and lower in Asians and African Americans.
      • May progress to myelofibrosis or acute leukemia
  • Secondary polycythemia:
    • Central hypoxia increasing erythropoietin:
      • Chronic pulmonary disease
      • Sleep apnea (5 " “10% have high Hgb)
      • Obesity hypoventilation syndrome (Pickwickian syndrome)
      • Congenital heart disease (right-to-left shunt)
      • High altitude (chronic)
      • Smokers erythrocytosis
      • Carbon monoxide poisoning (chronic)
      • Chronic methemoglobinemia
    • Renal-mediated causes of increased erythropoietin production:
      • Renal cell carcinoma
      • Renal artery atherosclerotic narrowing
      • Focal glomerulonephritis
      • Postrenal transplant with or without rejection
      • Chronic hydronephrosis
      • Polycystic kidney disease and renal cysts
    • Inappropriate autonomous erythropoietin production:
      • Hepatomas
      • Cerebellar hemangioblastoma
      • Wilms tumor
      • Parathyroid carcinoma and adenoma
      • Ovarian tumors
      • Adrenal adenomas and carcinomas (pheochromocytoma, Cushing)
      • Uterine leiomyomata
    • Blood doping:
      • Recombinant erythropoietin abuse
      • Autologous transfusions
    • Drug abuse:
      • Chronic cocaine abuse
      • Androgenic steroids
  • Genetic disorders with polycythemia:
    • High-affinity Hgb variants
    • Bisphosphoglycerate deficiency
    • von Hippel " “Lindau syndrome
    • Chuvash polycythemia
    • Erythropoietin-receptor mutations
    • Congenital methemoglobinemia
  • Infections:
    • Viral hepatitis, AIDS

Diagnostic Criteria for Polycythemia Vera
  • Major criteria:
    • Hgb >18.5 g/dL in men, >16.5 g/dL in women
    • Presence of JAK2 mutation by polymerase chain reaction (PCR) " “ clinches the dx
    • Oxygen saturation >92% and no other cause for secondary erythrocytosis
  • Minor criteria:
    • Low serum erythropoietin level
    • Bone marrow aspirate and biopsy revealing panhyperplasia
  • Adjuncts to diagnosis:
    • Platelets >400,000/mm3
    • ANC >10,000 (WBC >12,000/mm3)
    • Splenomegaly on exam or by CT
    • Leukocyte alkaline phosphatase elevation
    • B12 >900 pg/mL; unbound vitamin B12-binding capacity >2,200 pg/mL

Diagnosis


Signs and Symptoms


History
  • General:
    • Dyspnea
    • Weakness/fatigue
    • Excessive sweating
    • Epistaxis/gingival bleeding
    • Pruritus (40% of PV):
      • Generalized
      • Exacerbated by warm bath or shower
      • Excoriations common in PV
    • Gouty arthritis and tophi
  • Neurologic (hyperviscosity):
    • Headache
    • Vertigo/dizziness/tinnitus
    • Lethargy/confusion
    • Paresthesias
    • Cerebrovascular accident/TIAs
  • Visual (hyperviscosity):
    • Amaurosis fugax
    • Scotoma/blurred vision
    • Ophthalmic migraine
  • Cardiovascular:
    • CHF
    • Angina/myocardial infarction
    • Deep vein thrombosis (DVT)
    • Hypertension
  • Extremities:
    • Erythromelalgia:
      • Secondary to capillary sludging
      • Burning pain in the feet or hands
      • Warmth, erythema/cyanosis and puffiness of hand and feet
      • Acral paresthesias
      • Worse at night
      • Relief with cooling and aspirin
      • Pulses intact
    • Painful ulcers of fingers and toes (digital ischemia)
  • GI (unique to PV):
    • Hepatomegaly/splenomegaly
      • Sudden spleen enlargement in known PV suggests development of myelofibrosis
    • Epigastric discomfort/early satiety
    • Peptic ulcer disease/GI bleed
    • Budd " “Chiari syndrome (hepatic vein thrombosis): Ascites and peripheral edema

Physical Exam
  • Hypertension
  • Conjunctival suffusion
  • Fundus: Venous engorgement
  • Ruddy complexion/plethora
  • Erythema/rubor of hands, feet, nail beds
  • Skin excoriations from severe pruritus
  • Splenomegaly (75% in PV)
  • Hepatomegaly (30% in PV)
  • Thrombotic complications:
    • 2/3 arterial, 1/3 venous
    • Cumulative risk of 2 " “5% per year
    • TIAs, stroke, MI, digital infarcts
    • Unusual venous thrombosis:
      • Splenic or mesenteric veins
      • Hepatic vein and IVC clot with subsequent cirrhosis/ascites (Budd " “Chiari syndrome)
      • Cerebral sinus thrombosis
    • DVT, PE
  • Complications of hyperviscosity:
    • Lethargy/confusion
    • Headaches, dizziness, vision changes
    • Crackles/findings of CHF
  • Hemorrhagic complications:
    • Ecchymosis
    • Epistaxis
    • Gingival bleeding

Essential Workup


CBC with platelets ‚  

Diagnosis Tests & Interpretation


Lab
  • 1st priority: Distinguish relative from true erythrocytosis:
    • Volume repletion IV or PO, then repeat CBC
  • 2nd priority: Evaluate for secondary causes:
    • Pulse oximetry with pO2 <92%
    • Carboxyhemoglobin level
    • Erythropoietin level (normal or elevated if secondary)
    • CXR, chest CT, pulmonary function tests
    • Sleep study
    • Hgb electrophoresis
  • RBC mass:
    • Cr-51 " “labeled RBCs by nuclear medicine
    • Concomitant plasma volume with I-131 " “labeled albumin
    • Not necessary if Hgb >18.5 in men, or >16.5 in women
    • Red blood cell mass <35 mg/kg (males) or <31 mg/kg (females) is normal.
    • Decreased plasma volume with normal RBC mass verifies relative erythrocytosis.
    • Elevated RBC mass suggests PV or secondary polycythemia.
    • Falsely low if iron deficient or obese
  • PV suspected if:
    • Hgb >18.5 g/dL (men), 16.5 g/dL (women)
    • Absolute neutrophil count >10,000
    • Platelet count >400,000
    • Pulse oximetry >92%
    • Low erythropoietin level " “ a major clue
    • Vitamin B12 level elevated in 30% (unbound vitamin B12-binding capacity elevated in 75%)
    • Uric acid elevated in 40%
    • Leukocyte alkaline phosphatase elevated in 70%
    • PCR for JAK2 gene mutation diagnostic of PV (seen in 97%)

Imaging
Abdominal US or CT can detect a splenomegaly ‚  

Differential Diagnosis


See Etiology. ‚  

Treatment


Initial Stabilization/Therapy


ABCs with emphasis on fluid resuscitation if no evidence of CHF ‚  

Ed Treatment/Procedures


Emergency Management of Hyperviscosity Syndrome or Hct >60%
  • Fluid resuscitation to achieve hemodilution:
    • Withhold if evidence of CHF
  • Emergency phlebotomy of 250 " “500 mL of blood over 1 " “2 hr replacing with an equal amount of 0.9% normal saline (NS)
  • Removal of 1,000 " “1,500 mL of blood over 24 hr with a goal of Hct <60 or relief of symptoms:
    • Keep Hct >45.
    • Replace with an equal amount of 0.9% NS.
  • Phlebotomize the elderly and those with cardiovascular disease more slowly:
    • Every-other-day phlebotomy
  • Emergent surgery with polycythemia:
    • Phlebotomize to Hct of 45 to avoid thrombotic complications postoperatively.
  • Thrombocytosis therapy:
    • Administer aspirin if platelet count is 500,000 " “1,500,000/mm3 and there are no hemorrhagic complications.
  • Treat pruritus with diphenhydramine.

Long Term Management
  • Phlebotomy: Maintain Hct at 45% for men and 42% for women.
  • Aspirin 81 mg daily if thrombocytosis
  • Interferon-α (normalizes CBC in 80%):
    • Especially helpful for refractory pruritus and painful splenomegaly
    • Suggested in symptomatic patients <60 yr
  • Anagrelide:
    • Specific for thrombocytosis
    • No risk of leukemia, ideal for younger patients with postphlebotomy thrombocytosis
    • Effective alone and can decrease need for or frequency of chemotherapy
  • Hydroxyurea:
    • Mainstay of therapy, especially for patients >60 yr, with frequent phlebotomy requirements, thrombotic episodes, or refractory thrombocytosis
  • Aldylating agents: Busulfan:
    • Severe refractory disease in the elderly
    • High risk of leukemic transformation

Temporary remission during pregnancy, no treatment usually needed ‚  
  • In the neonate, defined as a peripheral venous Hct >65%, Hgb >22 g/dL:
    • Sample must be obtained >6 hr post delivery.
    • Capillary Hgb and Hct are 10% higher than venous (always rely on venous)
    • 1 " “5% of neonates
    • Up to 50% of neonates with intrauterine growth retardation
  • Etiology:
    • Maternal " “fetal hypoxemia secondary to maternal heart or lung disease, diabetes, preeclampsia, hypertension, or smoking
    • Delayed clamping of the umbilical cord with increase cord transfusion
  • Symptoms and signs (most asymptomatic):
    • Acrocyanosis/plethoric
    • Tachypnea/respiratory distress
    • Irritable, lethargic, jittery, poor feeding
  • Hypoglycemia and hyperbilirubinemia common
  • Treatment:
    • Observation and serial CBCs
    • 0.9 NS 100 mL/kg per day (symptomatic)
    • Partial exchange transfusion: Remove 20 mL/kg blood and infuse equal amount of saline (persistent or severe symptoms)
    • Dextrose solutions if hypoglycemia

Caution with speed of phlebotomy and fluid resuscitation as noted ‚  

Follow-Up


Disposition


Admission Criteria
  • New diagnosis of polycythemia
  • Hct >60% without symptoms
  • Symptoms of hyperviscosity
  • Unstable vital signs/significant comorbidities

Discharge Criteria
  • Previous diagnosis of polycythemia, Hct <60, and asymptomatic
  • Stable vital signs

Issues for Referral
All patients should be referred to a hematologist or primary care physician. ‚  

Pearls and Pitfalls


  • Criteria for phlebotomy in polycythemia secondary to hypoxemia is not clear. While phlebotomy will decrease viscosity, it may decrease oxygen-carrying capacity.
  • It is critical to distinguish PV from secondary causes of erythrocytosis since PV carries a high risk of thrombotic complications.
  • Pruritus with water contact and erythromelalgia (pain, paresthesia and rubor in hands/feet) are unique features of PV.

Additional Reading


  • Adams ‚  BD, Baker ‚  R, Lopez ‚  JA, et al. Myeloproliferative disorders and hyperviscosity syndrome. Emerg Med Clin North Am.  2009;27:459 " “476.
  • Kremyanskaya ‚  M, Mascarenhas ‚  J, Hoffman ‚  R. Why does my patient have erythrocytosis? Hematol Oncol Clin North Am.  2012;26(2):267 " “283.
  • Landolfi ‚  R, Nicolazzi ‚  MA, Porfidia ‚  A, et al. Polycythemia vera. Intern Emerg Med.  2010;5(5):375 " “384.
  • McMullin ‚  MF. The classification and diagnosis of erythrocytosis. Int J Lab Hematol.  2008;30:447 " “459.
  • Patnaik ‚  MM, Tefferi ‚  A. The complete evaluation of erythrocytosis: Congenital and acquired. Leukemia.  2009;23:834 " “844.
  • Tefferi ‚  A. Polycythemia vera and essential thrombocythemia: 2012 update on diagnosis, risk stratification and management. Am J Hematol.  2012;87(3):285 " “293.

Codes


ICD9


  • 238.4 Polycythemia vera
  • 289.0 Polycythemia, secondary

ICD10


  • D45 Polycythemia vera
  • D75.1 Secondary polycythemia

SNOMED


  • 127062003 Erythrocytosis (disorder)
  • 109992005 Polycythemia vera (disorder)
  • 44865000 Secondary polycythemia (disorder)
  • 389147003 Relative polycythemia (disorder)
  • 36874002 Stress polycythemia (disorder)
  • 48250002 Gaisbocks syndrome (disorder)
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