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Anticholinergic Poisoning, Emergency Medicine


Basics


Description


  • Central and peripheral cholinergic blockade
  • Depending on the drug involved, antagonism occurs at muscarinic (most common), nicotinic, or both receptors.
  • Onset of activity: 15-30 min after ingestion
  • Duration of effect: 2-24 hr

Etiology


  • Many drugs contain anticholinergic properties:
    • Mild at therapeutic doses
    • Life threatening in overdose
  • Anticholinergic substances:
    • Antihistamines
    • Belladonna alkaloids and synthetic congeners
    • Antiparkinsonian drugs
    • Cyclic antidepressants
    • Antipsychotics (neuroleptics)
    • Mydriatics
    • Skeletal muscle relaxants (orphenadrine, cyclobenzaprine)
    • Antispasmodics
    • Mushrooms-Amanita muscaria, Amanita pantherina
    • Plants-deadly nightshade, mandrake, henbane
    • Jimson weed-smoked or ingested

Diagnosis


Signs and Symptoms


History
  • Onset and duration of symptoms
  • Type and extent of ingestion/exposure

Physical Exam
  • Classic toxidrome:
    • "Mad as a hatter"-altered mental status
    • "Hot as a hare"-hyperthermia
    • "Red as a beet"-flushed skin
    • "Dry as a bone"-dry skin and mucous membranes
    • "Blind as a bat"-blurred vision secondary to mydriasis
  • General:
    • Hyperthermia
    • Altered mental status
  • Ocular:
    • Unreactive mydriasis
    • Inability to accommodate
  • Cardiovascular:
    • Sinus tachycardia
    • Dysrhythmias (rare except in massive ingestions)
    • Hypotension/HTN
    • Cardiogenic pulmonary edema
  • Pulmonary:
    • Tachypnea
    • Respiratory failure
  • GI:
    • Decreased/absent bowel sounds
    • Dysphagia
    • Decreased GI motility
    • Decreased salivation
  • Genitourinary (GU):
    • Urinary retention
  • Integument:
    • Decreased sweating
    • Flushed skin
    • Dry skin and mucous membranes
  • CNS:
    • Altered mental status
    • Auditory or visual hallucinations
    • Coma
    • Seizures

Essential Workup


Diagnosis based on clinical presentation and an accurate history  

Diagnosis Tests & Interpretation


Lab
  • Urine toxicologic screen if clinically indicated
  • Electrolytes, BUN, creatinine, and glucose
  • CBC
  • Creatine phosphokinase (CPK) if suspected rhabdomyolysis
  • Urinalysis
  • Acetaminophen and salicylate levels:
    • Detects occult ingestion (e.g., Tylenol PM)

Imaging
ECG:  
  • Sinus tachycardia most common
  • QRS prolongation
  • AV blockade
  • Bundle branch block pattern
  • Dysrhythmias

Differential Diagnosis


  • Sympathomimetic intoxication
  • Withdrawal syndrome
  • Acute psychiatric disorders
  • Sepsis
  • Thyroid disorder

Treatment


Pre-Hospital


Transport all pills/pill bottles involved in overdose for identification in ED.  

Initial Stabilization/Therapy


  • Airway, breathing, and circulation (ABCs):
    • Airway control essential
    • Administer supplemental oxygen.
    • IV access
    • Cardiac monitor and pulse oximetry
  • Naloxone, thiamine, D50 (or Accu-Chek) if altered mental status

Ed Treatment/Procedures


  • Supportive care:
    • IV rehydration with 0.9% NS
    • Standard aggressive cooling measures for hyperthermia
    • Use benzodiazepines for treatment of agitation:
      • Avoid phenothiazines owing to anticholinergic effects.
    • Treat seizures with benzodiazepines and barbiturates.
    • Dysrhythmias:
      • Use standard antidysrhythmics.
      • Avoid class Ia antidysrhythmic owing to the quinidine-like effect of many anticholinergic drugs.
      • Sodium bicarbonate boluses may reverse the quinidine-like effects.
  • Decontamination:
    • Administer activated charcoal for oral ingestions if within 1 hr.
    • Ocular lavage for eyedrop exposure
  • Physostigmine (Antilirium):
    • Reversible acetylcholinesterase inhibitor that crosses the blood-brain barrier
    • Short-term reversal of both central and peripheral anticholinergic effects
    • Indicated in the presence of peripheral anticholinergic signs and the following:
      • Seizures unresponsive to conventional therapy
      • Uncontrollable agitation
    • Use with caution if prolonged QRS is present on ECG owing to risk of dysrhythmias (especially asystole), seizures, and cholinergic crises:
      • Place on cardiac monitor.
      • Observe for cholinergic symptoms.
    • Contraindications:
      • Cyclic antidepressant overdose (potentiates toxicity)
      • Cardiovascular disease
      • Asthma/bronchospasm
      • Intestinal obstruction
      • Heart block
      • Peripheral vascular disease
      • Bladder obstruction

Medication


  • Activated charcoal: 1 g/kg PO
  • Dextrose: 50-100 mL D50 (peds: 2 mL/kg of D25 over 1 min) IV; repeat if necessary
  • Diazepam: 5-10 mg (peds: 0.2-0.5 mg/kg) IV every 10-15 min
  • Dopamine: 2-20 μg/kg/min IV with titration to effect
  • Lorazepam: 2-4 mg (peds: 0.03-0.05 mg/kg) IV every 10-15 min
  • Physostigmine: 0.5-2.0 mg (peds: 0.02 mg/kg) IV over 5 min; repeat if necessary in 30-60 min
  • Phenobarbital: 10-20 mg/kg IV (loading dose); monitor for respiratory depression
  • Thiamine (vitamin B1): 100 mg (peds: 50 mg) IV or IM

First Line
Lorazepam or Diazepam  
Second Line
Physostigmine (use with caution and consult with medical toxicologist)  

Follow-Up


Disposition


Admission Criteria
  • ICU admission for moderate to severe anticholinergic symptoms (agitation control, temperature control, and observation for seizures or dysrhythmias)
  • Any patient receiving physostigmine

Discharge Criteria
Mild and improving symptoms of anticholinergic toxicity after 6-8 hr of ED observation  
Issues for Referral
  • Substance abuse referral for patients with recreational anticholinergic abuse
  • Patients with unintentional (accidental) poisoning require poison prevention counseling.
  • Patients with intentional (e.g., suicide) poisoning require psychiatric evaluation.

Followup Recommendations


Appropriate psychiatric referral for intentional ingestions  

Pearls and Pitfalls


  • Aggressively treat hyperthermia.
  • Antipyretic medications are not effective in toxic hyperthermia.
  • Use physostigmine cautiously and consult with medical toxicologist when available.

Additional Reading


  • Burns  MJ, Linden  CH, Graudins  A, et al. A comparison of physostigmine and benzodiazepines for the treatment of anticholinergic poisoning. Ann Emerg Med.  2000;35:374-381.
  • Ceha  LJ, Presperin  C, Young  E, et al. Anticholinergic toxicity from nightshade berry poisoning responsive to physostigmine. J Emerg Med.  1997;15:65-69.
  • Delaney  KA. Anticholinergics and antihistamines (H1 antagonists). In: Ford  MD, Delaney  KA, Ling  LJ, et al., eds. Clinical Toxicology. Philadelphia, PA: WB Saunders; 2001;472-477.
  • Hidalgo  HA, Mowers  RM. Anticholinergic drug abuse. Ann Pharmacother.  1990;24:40.
  • Patel  RJ, Saylor  T, Williams  SR, et al. Prevalence of autonomic signs and symptoms in antimuscarinic drug poisonings. J Emerg Med.  2004;26(1):89-94.
  • Reilly  KM, Chan  L, Mehta  NJ, et al. Systemic toxicity from ocular homatropine. Acad Emerg Med.  1996;3:868-871.

Codes


ICD9


971.1 Poisoning by parasympatholytics (anticholinergics and antimuscarinics) and spasmolytics  

ICD10


T44.3X1A Poisoning by oth parasympath and spasmolytics, acc, init  

SNOMED


  • 216593002 Accidental poisoning by anticholinergics (disorder)
  • 296393006 Anticholinergic drug overdose (disorder)
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