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Pancreatic Pseudocyst, Pediatric


Basics


  • A pancreatic pseudocyst is a peripancreatic (or intrapancreatic) fluid collection associated with a history of pancreatitis, that is surrounded by a well-defined inflammatory wall, and that has no solid component.
    • The term "pseudocyst "  is often incorrectly used to define various types of fluid collections associated with pancreatitis. As a result, medical literature on pseudocysts is not consistent in its descriptions or its findings.
    • An important distinction between "fluid collections "  associated with pancreatitis is that some consist of fluid alone, whereas others arise from necrosis of pancreatic parenchyma and/or peripancreatic tissues. The latter type of fluid collection involves a solid component (with variable amounts of fluid), which distinguishes them from pseudocysts.
  • Types of fluid collections:
    • Acute peripancreatic fluid collection (APFC)
      • A fluid collection that develops in the early phase of interstitial edematous acute (typically mild) pancreatitis
      • Lack a well-defined wall on CT scan
      • NOT associated with necrotizing pancreatitis
      • Remain sterile, and usually resolve without intervention
      • If APFC persists beyond 4 weeks, likely to develop into a pancreatic pseudocyst; although this is considered a rare outcome.
    • Pancreatic pseudocyst
      • Refers specifically to a peripancreatic (or less commonly, an intrapancreatic) fluid collection
      • Surrounded by a well-defined inflammatory wall and containing NO solid material
      • Pancreatic pseudocysts develop more than 4 weeks after the onset of interstitial pancreatitis.
    • Acute necrotic collection (ANC)
      • A collection of BOTH variable amounts of fluid and solid (necrotic) material related to pancreatic and/or peripancreatic necrosis
      • Occur within the first 4 weeks of disease, and can resemble an APFC in the first few days of acute pancreatitis
      • As necrotizing pancreatitis develops and necrosis evolves, solid component become evident.
      • May be multiple and may involve the pancreatic parenchyma alone, the peripancreatic tissue alone, or most commonly both
      • May be infected or sterile
      • Generally associated with more severe sequelae of AP
    • Walled-off necrosis (WON)
      • Collection of varying amounts of liquid and solid material surrounded by a mature, enhancing wall of reactive tissue
      • Represents a mature, encapsulated ANC
      • Develops no earlier than 4 weeks after episode of necrotizing pancreatitis
      • May be multiple and present at sites distant from the pancreas
      • May be sterile or infected

Pathophysiology


  • Pseudocysts occur when there is disruption in the pancreatic ductular system, or its intrapancreatic branches, without any evidence of pancreatic or peripancreatic necrosis.
  • This results in the extravasation of pancreatic enzymes evoking an inflammatory response.
  • The inflammatory reaction leads to a fluid collection that is rich in pancreatic enzymes (APFC).
  • If the duration of the fluid collection is >4 weeks, becomes localized (intrapancreatic or extrapancreatic), and develops a fibrin capsule, it becomes a pseudocyst.
  • A pseudocyst does not have a true epithelial lining.
  • If there is communication between the pseudocyst and the pancreatic duct, the enzyme level in the fluid remain elevated; if there is no communication, the enzyme level falls with time.

Diagnosis


History


Acute or chronic pancreatitis ‚  
  • Suspect pancreatic pseudocyst in patients recovering from acute pancreatitis, or in the patient with chronic pancreatitis, who has recurrent/persistent abdominal pain, a palpable abdominal mass, or persistently elevated serum pancreatic enzymes.

Physical Exam


  • Abdominal tenderness
  • Abdominal mass
  • Nausea and vomiting
  • Weight loss
  • Jaundice
  • Abdominal distention
    • Mass/ascites
  • In many situations, no clinical signs are seen.
  • Clinical signs may be secondary to complications:
    • Jaundice in hepatobiliary obstruction
    • Lower limb edema in compression of inferior vena cava
    • Ascites in peritonitis
    • Pleural effusion

Diagnostic Tests & Interpretation


Lab
Serum pancreatic enzyme levels: ‚  
  • Persistently elevated enzymes in blood can be a clue, but is not an absolute indicator.
  • Elevated enzymes in fluid drained from a peripancreatic or intrapancreatic fluid collection with no solid component is consistent with a pancreatic pseudocyst.

Imaging
  • CT scan
    • Reveals pseudopancreatic cyst; can also be used to gauge size of pseudocyst and its relationship to adjacent organs
  • Ultrasonography
    • Visualizes pancreatic pseudocysts
    • Can be used to follow cyst size over time
  • Endoscopic ultrasonography (EUS)
    • Common modality in adult patients and increasingly used in pediatrics
    • Can be used to diagnose presence and size of pseudocyst; can also be used to guide peroral fluid aspiration and drainage
  • Endoscopic retrograde cholangiopancreatography (ERCP)
    • Used in some cases to delineate the pancreatic ductular system before drainage to distinguish ductal stenosis, disruption, stones, and other obstructions

Differential Diagnosis


  • Congenital/genetic
    • Congenital cysts
    • Polycystic disease
    • Von Hippel-Lindau disease
    • Cystic fibrosis
  • Infections
    • Pancreatic abscess
    • Echinococcal (hydatid) cyst
    • Taenia solium cyst
  • Tumor
    • Serous cystadenoma
    • Mucinous cystadenoma
    • Cystic islet cell tumors
    • Teratoma
    • Pancreatoblastoma
    • Cystadenocarcinoma
    • Franz tumor
    • Angiomatous cystic neoplasms
    • Lymphangiomas
    • Hemangioendothelioma
  • Miscellaneous:
    • Splenic cyst
    • Adrenal cyst
    • Enterogenous cyst
    • Duplication cysts
    • Endometriosis

Treatment


General Measures


  • Medical management:
    • Most cases resolve with supportive care.
    • If eating precipitates pain, short-term nasojejunal feedings or parenteral nutrition may be warranted.
    • Follow up with ultrasound or CT scan to make sure there are no complications.
    • >60% have complete resolution by the end of 1 year.
    • Usually, no medications are used for managing pseudocysts.
    • Somatostatin analogue (octreotide) has been reported to be used to decrease fluid collection along with drainage.
    • Antibiotics are used in situations of infected pseudocyst.
  • Drainage
    • Most often used in the setting of WON
    • Indications: infection, rupture with cardiopulmonary compromise, biliary and gastric outlet obstruction, persistent symptoms, rapid enlargement, failure of large pseudocysts (>6 cm) to shrink after 6 weeks
    • Modalities:
      • Percutaneous drainage (aspiration or catheter drainage) is done in cases in which the pseudocyst has a less mature wall.
      • Percutaneous aspiration has a high recurrence rate of 63% and failure rate of 54%.
      • Continuous drainage has a recurrence rate of 8% and a failure rate of 19%.
      • Endoscopic procedures are becoming the 1st-line drainage modality, as they are less invasive than surgery.
      • Endoscopic procedures include transmural cystoenterostomies and transpapillary route procedures such as stent placement for pseudocysts that communicate with the main pancreatic duct.
      • Endoscopic procedures in experienced hands report success rates of 82 " “89%, complication rates of 10 " “20%, and recurrence rates of 6 " “18%.

Surgery/Other Procedures


  • Reserved for failed endoscopic procedures, difficult to access areas of WON and multiple WONs
  • Includes internal drainage (cystogastrostomy, cystoduodenostomy, and Roux-en-Y cystojejunostomy), resection, and external drainage
  • Success rate is 85 " “90%.
  • Recurrence rate is 0 " “17%.
  • Mortality rate is between 3 and 5%.

Ongoing Care


Prognosis


Majority of pseudocysts resolve without intervention. ‚  

Complications


  • Perforation/rupture
    • Cardiopulmonary compromise secondary to pleural effusion and ascites
    • Peritonitis and ascites, which can be fatal
  • Hemorrhage
    • Erosions of vessels lining the cyst cause intracystic bleeding and rapid increase in the cyst size.
    • Bleeding may occur directly into stomach, duodenum (clinically manifesting as GI bleeding), or peritoneal cavity.
  • Obstruction
    • Biliary obstruction: jaundice
    • Portal obstruction: portal hypertension
    • Gastric outlet obstruction
    • Inferior vena cava obstruction: peripheral edema
    • Urinary obstruction
    • Colonic obstruction
  • Infection of pseudocysts is rare in children compared to adults:
    • Associated with high mortality rate for children and adults
    • Management usually requires surgical drainage.

Additional Reading


  • Law ‚  NM, Freeman ‚  ML. Emergency complications of acute and chronic pancreatitis. Gastroenterol Clin.  2002;32(4):1169 " “1194. ‚  [View Abstract]
  • Reber ‚  HA. Surgery for acute and chronic pancreatitis. Gastrointest Endosc.  2002;56(6)(Suppl):S246 " “S248. ‚  [View Abstract]
  • Sarr ‚  MG, Banks ‚  PA, Bollen ‚  TL, et al. The new revised classification of acute pancreatitis 2012. Surg Clin North Am,  2013;93(3):549 " “562. ‚  [View Abstract]
  • Vidyarthi ‚  G, Steinberg ‚  SE. Endoscopic management of pancreatic pseudocysts. Surg Clin North Am.  2001;81(2):405 " “410. ‚  [View Abstract]
  • Weckman ‚  L, Kylanpaa ‚  ML, Poulakkainen ‚  P, et al. Endoscopic treatment of pancreatic pseudocysts. Surg Endosc.  2006;20(4):603 " “607. ‚  [View Abstract]

Codes


ICD09


  • 577.2 Cyst and pseudocyst of pancreas

ICD10


  • K86.3 Pseudocyst of pancreas

SNOMED


  • 111374002 pseudocyst of pancreas (disorder)
  • 405563007 Infected pancreatic pseudocyst (disorder)
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