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Ankylosing Spondylitis

para>Infants exposed to NSAIDs in 1st trimester may have a higher incidence of cardiac malformations (5)[A]. á
Second Line
  • Biologic agents: tumor necrosis factor (TNF)-╬▒ antagonists
    • Recommended for high disease activity or when a trial of two NSAIDs over 4 weeks have failed (3)[C]
    • FDA-approved agents for AS include etanercept (recombinant TNF receptor fusion protein), infliximab (chimeric monoclonal IgG1 antibody to TNF-╬▒), adalimumab (fully humanized IgG1 monoclonal antibody to TNF-╬▒), and golimumab (human IgG1 kappa monoclonal antibody to TNF-╬▒).
    • Approved agents improve pain, function, and symptoms of AS as compared to placebo (6)[A].
    • No definitive evidence for TNF-╬▒ blockers with regards to disease remission, prevention of radiologic progression, or prevention of extra-articular manifestations (7).
    • Monoclonal TNF-╬▒ blockers are preferred when IBD is involved (3)[C].
    • Further investigation as to the effectiveness of TNF blocker therapy with NSAIDs is needed (8).
  • Precautions with TNF-╬▒ blockers
    • Anti-TNFs increase the risk of serious bacterial, mycobacterial, fungal, opportunistic, and viral infections. Screen for tuberculosis and hepatitis B.
    • Monitor for reactivation of tuberculosis and invasive fungal infections, such as histoplasmosis, in all patients, especially those who travel to (or residents in) endemic areas.
    • Lymphomas, nonmelanoma skin cancers, and other malignancies have been reported in patients receiving anti-TNFs.
    • Immunizations (especially live vaccines) should be updated before initiating anti-TNFs; live vaccines are contraindicated once patients receive anti-TNFs.
  • Disease-modifying antirheumatic drugs (DMARDs), such as methotrexate and sulfasalazine, are ineffective for axial disease; sulfasalazine may be effective for peripheral arthritis (3)[C].

ISSUES FOR REFERRAL


  • Physical therapy can assist with treatment plan (including home regimens).
  • Coordinate care with a rheumatologist for diagnosis, monitoring, and management (anti-TNF therapy).
  • Management of aortic regurgitation, uveitis, spinal fractures, pulmonary fibrosis, hip joint involvement, renal amyloidosis, and cauda equina syndrome may require referral to appropriate specialty.

ADDITIONAL THERAPIES


  • Bisphosphonate medications if osteopenia or osteoporosis is present
  • Monitoring and management of CVD risk factors and comorbidities

SURGERY/OTHER PROCEDURES


  • Evaluate for C-spine ankylosis/instability before intubation in patients with AS undergoing surgery.
  • Total hip replacement if necessary to restore mobility and to control pain.
  • Vertebral osteotomy can improve posture for patients with severe cervical or thoracolumbar flexion.

ONGOING CARE


FOLLOW-UP RECOMMENDATIONS


Patient Monitoring
  • Symptom control and maintenance of mobility and function are primary treatment goals.
  • Monitor posture and range of motion with 6- to 12-month visits; increase frequency if higher disease activity.
  • Bath Ankylosing Spondylitis Disease Activity Index (BASDAI) or Ankylosing Spondylitis Disease Activity Score (ASDAS) can be used to measure disease activity.

PATIENT EDUCATION


  • Maintain physical activity and posture.
  • Swimming, tai chi, and walking are excellent activities.
  • Avoid trauma/contact sports.
  • Appropriate ergonomic modification of workplace
  • Counsel about risk of spinal fracture.
  • MedicAlert bracelet (helpful if intubation required)
  • Arthritis Foundation: http://www.arthritis.org
  • Spondylitis Association of America: http://www.spondylitis.org

PROGNOSIS


  • Extent and rapidity of progression of ankylosis are highly variable.
  • Progressive limitation of spinal mobility necessitates lifestyle modification.

COMPLICATIONS


  • Spine
    • Spinal fusion causing kyphosis
    • Cervical spine fracture or subluxation carries high mortality rate; fracture can occur at any level of ankylosed spine.
    • Cauda equina syndrome (rare)
  • Pulmonary: restrictive lung disease, upper lobe fibrosis (rare)
  • Cardiac: conduction defects at atrioventricular (AV) node, aortic insufficiency, aortitis, pericarditis (extremely rare)
  • Eye: uveitis and cataracts
  • Renal: IgA nephropathy, amyloidosis (<1%)
  • GI: microscopic, subclinical ileal, and colonic mucosal ulcerations in up to 50% of patients, mostly asymptomatic

REFERENCES


11 Reveille áJD, Weisman áMH. The epidemiology of back pain, axial spondyloarthritis and HLA-B27 in the United States. Am J Med Sci.  2013;345(6):431-436.22 Dougados áM, Baeten áD. Spondyloarthritis. Lancet.  2011;377(9783):2127-2137.33 Braun áJ, van den Berg áR, Baraliakos áX, et al. 2010 update of the ASAS/EULAR recommendations for the management of ankylosing spondylitis. Ann Rheum Dis.  2011;70(6):896-904.44 Guellec áD, Nocturne áG, Tatar áZ, et al. Should non-steroidal anti-inflammatory drugs be used continuously in ankylosing spondylitis? Joint Bone Spine.  2014;81(4):308-312.55 Adams áK, Bombardier áC, van der Heijde áDM. Safety of pain therapy during pregnancy and lactation in patients with inflammatory arthritis: a systematic literature review. J Rheumatol Suppl.  2012;90:59-61.66 Maxwell áLJ, Zochling áJ, Boonen áA, et al. TNF-alpha inhibitors for ankylosing spondylitis. Cochrane Database of Syst Rev.  2015;(4):CD005468. doi:10.1002/14651858.CD005468.pub2.77 Gensler áL, Inman áR, Deodhar áA. The "knowns"Ł and "unknowns"Ł of biologic therapy in ankylosing spondylitis. Am J Med Sci.  2012;343(5):360-363.88 Sieper áJ. Treatment challenges in axial spondyloarthritis and future directions. Curr Rheumatol Rep.  2013;15(9):356. doi:10.1007/s11926-013-0356-9.

ADDITIONAL READING


  • Baraliakos áX, van den Berg áR, Braun áJ, et al. Update of the literature review on treatment with biologics as a basis for the first update of the ASAS/EULAR management recommendations of ankylosing spondylitis. Rheumatology (Oxford).  2012;51(8):1378-1387.
  • Garg áN, van den Bosch áF, Deodhar áA. The concept of spondyloarthritis: where are we now? Best Pract Res Clin Rheumatol.  2014;28(5):663-672.
  • Sieper áJ. Developments in therapies for spondyloarthritis. Nat Rev Rheumatol.  2012;8(5):280-287.
  • Sieper áJ, Rudwaleit áM, Baraliakos áX, et al. The Assessment of Spondyloarthritis International Society (ASAS) handbook: a guide to assess spondyloarthritis. Ann Rheum Dis. 2009;68(Suppl 2): ii1-ii44.
  • van den Berg áR, Baraliakos áX, Braun áJ, et al. First update of the current evidence for the management of ankylosing spondylitis with non-pharmacological treatment and non-biologic drugs: a systematic literature review for the ASAS/EULAR management recommendations in ankylosing spondylitis. Rheumatology (Oxford).  2012;51(8):1388-1396.

SEE ALSO


Arthritis, Psoriatic; Arthritis, Rheumatoid (RA); Crohn Disease; Reactive Arthritis (Reiter Syndrome); Ulcerative Colitis á

CODES


ICD10


  • M45.9 Ankylosing spondylitis of unspecified sites in spine
  • M08.1 Juvenile ankylosing spondylitis
  • M45.8 Ankylosing spondylitis sacral and sacrococcygeal region
  • M45.6Ankylosing spondylitis lumbar region
  • M45.0Ankylosing spondylitis of multiple sites in spine
  • M45.7Ankylosing spondylitis of lumbosacral region
  • M45.1Ankylosing spondylitis of occipito-atlanto-axial region
  • M45.4Ankylosing spondylitis of thoracic region
  • M45.3Ankylosing spondylitis of cervicothoracic region
  • M45.5Ankylosing spondylitis of thoracolumbar region
  • M45.2Ankylosing spondylitis of cervical region

ICD9


  • 720.0Ankylosing spondylitis

SNOMED


  • 9631008Ankylosing spondylitis (disorder)
  • 239805001Juvenile ankylosing spondylitis (disorder)
  • 239810002ankylosing spondylitis with organ / system involvement (disorder)

CLINICAL PEARLS


  • Diagnosis of AS is suggested by a history of inflammatory back pain, evidence of limited chest wall expansion, restricted spinal movements in all planes, radiographic evidence of sacroiliitis, and a therapeutic response to NSAIDs.
  • HLA-B27 testing supports the diagnosis if clinical features are not definitive.
  • MRI is more sensitive at detecting SI joint inflammation than plain radiography.
  • Physical therapy is important in helping to maintain posture and mobility.
  • NSAIDs and TNF-╬▒ blockers are the mainstays of pharmacologic treatment of AS.
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