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Osteogenesis Imperfecta, Emergency Medicine


Basics


Description


  • Inherited abnormality ofprocollagen amino acid sequence
  • Bone hypomineralization and incomplete ossification result in brittle bones.
  • Abnormal collagen affects all connective tissue to varying degrees.
  • Time course is variable:
    • Most cases involve fractures during childhood followed by quiescence during adolescence and early adulthood.

Etiology


  • Procollagen defects result in abnormalities of bone and connective tissue matrix.
  • Defects in different sites on procollagen protein chain result in more severe forms.
  • Defects are inherited, either autosomal recessive (generally milder) or autosomal dominant (more severe).
  • Lethal cases involve sporadic or new mutations.
  • Ehlers " “Danlos syndrome involves mutations of the same procollagen protein in different locations.

  • Most cases involve pathologic fractures during childhood.
  • Multiple fractures often initiate evaluation for abuse, but the possibility of pathologic fractures also should be considered.

Diagnosis


Signs and Symptoms


  • Multiple heritable defects that lead to brittle bones:
    • Often associated with other connective tissue abnormalities
  • A suspected fracture with a relatively minor mechanism or a history of multiple fractures in a child suggests the diagnosis.
  • Careful social history with consideration for the possibility of nonaccidental trauma
  • Bones:
    • Multiple recurrent fractures (especially in long bones) are the hallmark of this disease.
    • Fractures may be present at birth or may recur in the elderly.
    • Shortened or bowed limbs, pectus excavatum, curving of long bones, vertebral compression fractures, scoliosis, kyphosis, and abnormal skull shape
    • All bones are affected to some extent (see Imaging/Special Tests).
  • Eyes:
    • Blue sclerae are another hallmark of this disease.
    • No visual changes are reported.
  • Ears:
    • Hearing loss usually begins in adolescence; >90% of patients have some deficit by age 30 yr.
    • Hearing loss is generally sensorineural, although some middle ear abnormalities have been demonstrated.
    • Academic difficulties should raise suspicion of possible hearing deficits.
  • Other:
    • Discolored, fragile, and abnormal shape of teeth
    • Shares several features with Ehlers " “Danlos syndrome:
      • Loose joints
      • Valve problems
      • Vascular abnormalities
  • Thyroid abnormalities may be seen.
  • Extreme cases may result in perinatal death.

Essential Workup


  • Diagnosis is usually made as combination of clinical and radiographic findings.
  • History of repeated fractures or fractures with unimpressive mechanism
  • Thorough search for other tender areas and evaluation of eyes, teeth, and joints is important for diagnosis.
  • Careful exam of neurovascular status distal to fracture

Diagnosis Tests & Interpretation


Lab
  • Evaluate for metabolic derangements such as hyperparathyroidism, vitamin C or D deficiencies, and calcium/phosphate abnormalities.
  • DNA studies may be indicated for familial analysis, prenatal testing, and genetic counseling.
  • Tissue biopsy is controversial but may help differentiate from tumors.

Imaging
  • Radiographs of fracture sites:
    • May reveal osteopenia (usually mild)
    • Crumpled long bones ( "accordion femora " ť)
    • Incomplete ossification at physes
  • Skeletal survey is mandatory, especially in children.
  • Skull films may show wormian appearance of irregular ossification.
  • Popcorn-like deposits on long-bone ends are poor prognostic finding.
  • Formal audiologic testing as outpatient is required in older patients.

Differential Diagnosis


  • Nonaccidental trauma in children
  • Ehlers " “Danlos syndrome
  • Hypophosphatasia
  • Achondroplasia
  • Scurvy
  • Congenital syphilis
  • Celiac disease

Treatment


Pre-Hospital


Personnel should obtain information about mechanism or social factors that point toward pathologic fracture vs. nonaccidental trauma. ‚  

Initial Stabilization/Therapy


  • Airway management and resuscitation as indicated
  • Fracture immobilization/splinting

Ed Treatment/Procedures


  • Specific fracture management dictated by type and location of injury
  • Orthopedic consultation regarding need for traction or operative fixation
  • No specific treatment for osteogenesis imperfecta exists at present.

Medication


  • Pain medications as indicated
  • Elderly women may benefit from calcium (1 " “1.5 g/d) and estrogen replacement (0.625 mg/d).

Follow-Up


Disposition


Admission Criteria
  • Admission is determined by multiple trauma or operative needs for fracture repair.
  • Pediatric patients may need admission to investigate possibility of nonaccidental trauma.

Discharge Criteria
  • Patients may be considered for outpatient management if isolated fracture is present and appropriate home resources are available.
  • Most patients should be discharged with orthopedic and primary physician follow-up.

Issues for Referral
  • Orthopedic referral is driven by the acute injury.
  • The presence of fractures in multiple locations or at different times also suggests nonaccidental trauma, which should prompt acute consultation and/or referral per local protocol.

Follow-Up Recommendations


  • Follow-up is generally driven by the acute injuries.
  • Follow-up with the primary physician should be instituted to encourage treatment and monitoring of the disease.

Pearls and Pitfalls


  • The most challenging aspect of caring for these patients is differentiating between pathologic fractures associated with osteogenesis imperfecta and nonaccidental trauma. With any questions, acute consultation and/or referral should be initiated per local protocol.
  • It is a myth that children with osteogenesis imperfecta feel less pain than other patients.
  • Predisposition to respiratory infections

Additional Reading


  • Bishop ‚  N. Osteogenesis imperfecta. Medicine.  2005;33(12):67 " “69.
  • Prockop ‚  DJ. Heritable disorders of connective tissue. In: Wilson ‚  JD, et al., eds. Harrisons Principles of Internal Medicine. 12th ed. New York, NY: McGraw-Hill; 1991:1860.
  • Rauch ‚  F, Glorieux ‚  FH. Osteogenesis imperfecta. Lancet.  2004;363(9418):1377 " “1385.
  • Shapiro ‚  JR, Sponsellor ‚  PD. Osteogenesis imperfecta: Questions and answers. Curr Opin Pediatr.  2009;21(6):709 " “716. www.oif.org

See Also (Topic, Algorithm, Electronic Media Element)


Specific Orthopedic Injuries ‚  

Codes


ICD9


756.51 Osteogenesis imperfecta ‚  

ICD10


Q78.0 Osteogenesis imperfecta ‚  

SNOMED


  • 78314001 Osteogenesis imperfecta (disorder)
  • 385482004 osteogenesis imperfecta type I (disorder)
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