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Oculomotor Nerve Palsy, Emergency Medicine


Basics


Description


  • Typical presentation of a 3rd cranial nerve (CN) palsy:
    • Eyelid drooping
    • Blurred or double vision
    • Light sensitivity
    • May also have other neurologic signs/symptoms:
      • Hemiplegia
      • Ataxia
      • Tremor
  • CN III controls elevation, adduction and depression of the eye. This nerve also raises the lid and mediates pupillary constriction and lens accommodation:
    • Medial rectus:
      • Moves eye medially toward nose (adduction)
    • Superior rectus:
      • Moves eye upward
      • Rotates top of eye toward nose
      • Slight adduction
    • Inferior rectus:
      • Moves eye inferiorly
      • Rotates top of eye away from nose
      • Slight adduction
    • Inferior oblique:
      • Rotates top of eye away from nose
      • Slight elevation and abduction
    • Levator palpebrae superioris:
      • Raises eyelid
  • CN IV innervates the superior oblique:
    • Moves eye down when looking medially
    • Rotates eye internally
  • CN VI innervates the lateral rectus:
    • Moves eye laterally (abduction)
    • Lesions categorized as:
    • Complete vs. incomplete
    • Pupil involving vs. pupil sparing
  • Complete: Total loss of CN III function ( "down and out " ):
    • Compressive lesions:
      • Aneurysms
      • Tumors
      • Brainstem herniation with compression
      • Increased intracranial pressure
  • Incomplete: Partial loss of CN III function:
    • Vascular infarction of vasa vasorum
  • Pupil involving:
    • 95 " “97% of compressive lesions (aneurysm, tumor, etc.) involve the pupil
    • Parasympathetic fibers sit peripherally in CN III
  • Pupil sparing:
    • Ischemic injury to nerve
    • Diabetics, uncontrolled hypertension

Etiology


  • Intracranial or orbital tumor
  • Aneurysm (particularly posterior communicating artery)
  • Trauma
  • Intracranial hemorrhage
  • Diabetes mellitus
  • Migraine headache
  • Infection, meningitis
  • Arteriovenous malformation or fistula
  • Cavernous sinus thrombosis
  • Neuropathy (e.g., myasthenia gravis, Guillain " “Barre)
  • Collagen vascular diseases (e.g., sarcoidosis)
  • Idiopathic

Trauma is the most common cause of acquired oculomotor nerve palsies ‚  

Diagnosis


Signs and Symptoms


A careful history and physical exam are vital to narrow down the differential diagnosis ‚  
History
History is of utmost importance in determining cause: ‚  
  • Headache
  • Pupillary dilation
  • Eye pain
  • Diplopia
  • Blurry vision
  • History of long-standing diabetes mellitus
  • Head trauma, either recent or distant
  • Unintentional weight loss
  • Signs and symptoms of infection
  • Sudden onset of severe headache, meningeal signs, photophobia
  • Proptosis
  • Lid swelling

Physical Exam
  • Ophthalmologic exam:
    • Extraocular movements
    • Fundoscopic exam for papilledema
    • Ipsilateral and contralateral pupillary reaction
    • Ptosis
    • Diplopia
    • Chemosis or conjunctival injection
    • Tenderness
    • Visual acuity
    • Exophthalmos
  • Pupil sparing lesion:
    • Ptosis
    • Globe is "down and out " 
    • No elevation, depression, or adduction
    • Normal pupil exam
    • CN IV, V, VI intact
    • Usually no other neurologic signs/symptoms
    • Most commonly caused by ischemia in adults
    • Also consider giant cell arteritis and trauma
  • Pupil-involving lesion:
    • Anisocria is present with a dilated pupil on affected side
    • Need to rule out compressive aneurysm
  • Incomplete, 3rd CN palsy:
    • May have involvement of 1 or more extraocular muscle and may or may not involve pupil
  • Look for associated symptoms:
    • Extremity weakness
    • Changes in speech
    • Dysfunction of other CNs
    • Gait or coordination

Essential Workup


CT/MRI of brain, orbit, sinuses ‚  

Diagnosis Tests & Interpretation


Lab
When indicated based on history and physical exam: ‚  
  • CBC with differential
  • ESR
  • Antinuclear antibodies, rheumatoid factor to evaluate for vasculitis
  • Lumbar puncture

Imaging
  • MRI/MRA of brain and cerebral vessels particularly when pupil is involved
  • CT angiogram
  • Cerebral arteriogram: Has associated risk of neurologic morbidity and mortality
  • Doppler imaging for arteriovenous malformations, dural sinus thrombosis

Diagnostic Procedures/Surgery
  • Intraocular pressure to exclude glaucoma
  • Slit-lamp exam:
    • Observe structural abnormalities of iris or anterior chamber

Differential Diagnosis


  • Intracranial infections
  • Malignancy
  • Vasculitis
  • Aneurysms
  • Myasthenia gravis
  • Botulism
  • Orbital infections
  • Trauma
  • Lens pathology
  • Retinal pathology
  • Glaucoma
  • MS

Consider congenital oculomotor nerve palsy ‚  

Treatment


Pre-Hospital


Without associated trauma, no specific pre-hospital care issues exist ‚  

Initial Stabilization/Therapy


  • Initial stabilization of trauma patient should concentrate on underlying injuries
  • Any patient with evidence of herniation should have the following measures to control intracranial pressure:
    • Intubation using rapid-sequence induction and controlled ventilation to a PCO2 level of 35 " “40 mm Hg
    • Elevate head of bed 30 ‚ °
    • Mannitol

Ed Treatment/Procedures


  • Differentiation between incomplete and complete oculomotor or pupil-involving vs. pupil-sparing nerve palsy guides focus of ED treatment
  • All patients younger than 50 yr with any extent of 3rd nerve palsy should be evaluated for a compressive lesion
  • If pupil is involved, neuroimaging is indicated as well as consultation to determine cause
  • If pupil is spared and the patient has diabetes or other risk for an ischemic 3rd nerve, discharge is likely reasonable with outpatient follow-up:
    • If partial sparing or patient does not have these risk factors, consultation and neuroimaging is indicated
  • Medication regimen determined by cause:
    • Aneurysm:
      • Control severe HTN.
      • Decrease intracranial pressure
      • Controlled ventilation
      • Elevation of head
      • Mannitol
    • Intracranial tumor: Control increasing intracranial pressure
    • Inflammation and edema: Decrease with IV steroids.
    • Meningitis:
      • Rapid administration of IV antibiotics
      • IV steroids may be useful to decrease inflammatory response and edema
    • Vasculitis and collagen vascular diseases: Decrease inflammatory cell infiltration with IV steroids
    • Neuropathy: Myasthenia gravis " ”edrophonium chloride test
  • Neurosurgical consultation as appropriate

MRI/MRA is indicated for all children with a 3rd nerve palsy ‚  

Medication


  • Ceftriaxone: 1 " “2 g (peds: 50 " “100 mg/kg) IV
  • Dexamethasone: 10 mg IV (peds: 0.15 " “0.5 mg/kg IV single dose in ED)
  • Edrophonium chloride: 5 " “8 mg IV (peds: 0.15 mg/kg IV; 1/10 test dose given 1st)
  • Mannitol: 1 g/kg IV (peds: Not routinely recommended)
  • Methylprednisolone: Adults/peds: 1 " “2 mg/kg IV single dose in ED

Follow-Up


Disposition


Admission Criteria
  • Complete oculomotor nerve palsy of any cause requires admission and emergency neurosurgical evaluation
  • Incomplete oculomotor nerve palsy with abnormal CT or MRI, abnormal lab studies, or other focal neurologic or constitutional symptoms should receive prompt neurologic consultation and imaging

Discharge Criteria
  • Incomplete oculomotor nerve palsy with negative CT or MRI, normal lab studies, and no other symptoms can be referred for urgent outpatient neurologic evaluation
  • Complete pupil-sparing oculomotor palsy in patients with risk factors for microvascular disease (i.e., diabetic) can receive outpatient neurologic workup

Followup Recommendations


If the patient is being discharged, prompt neurologic follow-up is required ‚  

Pearls and Pitfalls


  • Complete lesions must be assessed rapidly
  • Patients <50 yr old with any extent of CN III palsy should be evaluated for compressive lesions
  • If the pupil is involved, compressive lesions are often the cause and immediate MRI/MRA is indicated

Additional Reading


  • Bruce ‚  BB, Biousse ‚  V, Newman ‚  NJ. Third nerve palsies. Semin Neurol.  2007;27:257 " “268.
  • Chen ‚  CC, Pai ‚  YM, Wang ‚  RF, et al. Isolated oculomotor nerve palsy from minor head trauma. Br J Sports Med.  2005;39:e34.
  • Woodruff ‚  MM, Edlow ‚  JA. Evaluation of third nerve palsy in the emergency department. J Emerg Med.  2008;35:239 " “246.
  • Yanovitch ‚  T, Buckley ‚  E. Diagnosis and management of third nerve palsy. Curr Opin Ophthalmol.  2007;18:373 " “378.

Codes


ICD9


  • 378.51 Third or oculomotor nerve palsy, partial
  • 378.52 Third or oculomotor nerve palsy, total

ICD10


  • H49.00 Third [oculomotor] nerve palsy, unspecified eye
  • H49.01 Third [oculomotor] nerve palsy, right eye
  • H49.02 Third [oculomotor] nerve palsy, left eye
  • H49.03 Third [oculomotor] nerve palsy, bilateral
  • H49.0 Third [oculomotor] nerve palsy

SNOMED


  • 388980004 third cranial nerve weakness (disorder)
  • 3171005 Partial third nerve palsy (disorder)
  • 194119004 Total oculomotor nerve palsy (disorder)
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