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Neuroretinitis

para>If a bilateral presentation is found, the differential diagnosis should expand to include increased intracranial pressure or mass effect, although there have been case reports of bilateral presentation associated with cat-scratch disease (CSD). ‚  

DIAGNOSTIC TESTS & INTERPRETATION


Directed to suspected underlying etiology ‚  
Initial Tests (lab, imaging)
Evaluation might include the following: ‚  
  • CBC
  • Angiotensin-converting enzyme
  • Antinuclear antibody
  • Antidouble-stranded DNA
  • Complement (C3)
  • Erythrocyte sedimentation rate
  • Fluorescent treponemal antibody absorption
  • Serologies for viral, fungal, and bacterial etiologies including B. henselae
  • Enzyme-linked immunosorbent assay for T. canis
  • Tuberculin skin test
  • MRI of the head and orbits

Follow-Up Tests & Special Considerations
  • The stellate macular appearance may develop subsequently to the optic nerve involvement and should be monitored in the 1st weeks after initial presentation.
  • Rarely, a CSF evaluation may be indicated.

Diagnostic Procedures/Other
  • Visual field testing may show a central scotoma affecting vision in the central field or a cecocentral scotoma, which includes the blind spot and extends into the area of central fixation due to involvement of the papillomacular bundle.
  • Color vision testing
  • Fluorescein angiography
  • Optical coherence tomography
  • Diagnostic vitrectomy, when appropriate

TREATMENT


  • Treatment is directed at the underlying etiology.
    • High-dose oral steroids are commonly used in idiopathic and recurrent neuroretinitis with occasional high-dose IV steroids used in recurrent neuroretinitis, although evidence for altering visual outcomes is lacking (3)[B].
  • Consider antibiotics for CSD while serologies are pending (4)[C].
    • Adults: rifampin plus ciprofloxacin or azithromycin
    • Children: rifampin plus azithromycin or sulfamethoxazole-trimethoprim

MEDICATION


Direct to underlying etiology ‚  

ISSUES FOR REFERRAL


Refer for neurologic and ophthalmologic consultation upon initial suspicion. ‚  

ADDITIONAL THERAPIES


Laser treatment has been used for T. canis (5)[C]. ‚  

SURGERY/OTHER PROCEDURES


Laser treatment can be directed at the invading organism in helminthic infections (5)[C]. ‚  

INPATIENT CONSIDERATIONS


As necessary for underlying systemic/neurologic complications ‚  

ONGOING CARE


FOLLOW-UP RECOMMENDATIONS


  • Systemic monitoring will depend on the symptoms and underlying etiology.
  • Ophthalmologic monitoring initially may be weekly to monitor for development of macular star and depending on underlying etiology and severity, followed by less-frequent monitoring until resolution of optic nerve and macular involvement is seen. Macular involvement may take longer to subside.

Patient Monitoring
Directed to underlying etiology ‚  

DIET


No specific diet ‚  

PROGNOSIS


  • Generally favorable outlook with typical reported recovery of vision to ≥20/40 within 2 months (6).
  • Initial visual acuity and lack of systemic symptoms appear to be the best predictors of good visual outcome in neuroretinitis caused by B. henselae (4).
  • Optic disc edema resolves >8 to 12 weeks (3).

COMPLICATIONS


  • Permanent vision loss
  • Retinal detachment
  • Optic atrophy
  • Retinal pigment epithelial defects

REFERENCES


11 Kitamei ‚  H, Suzuki ‚  Y, Takahashi ‚  M, et al. Retinal angiography and optical coherence tomography disclose focal optic disc vascular leakage and lipid-rich fluid accumulation within the retina in a patient with Leber idiopathic stellate neuroretinitis. J Neuroophthalmol.  2009;29(3):203 " “207.22 Ray ‚  S, Gragoudas ‚  E. Neuroretinitis. Int Ophthalmol Clin.  2001;41(1):83 " “102.33 Purvin ‚  V, Sundaram ‚  S, Kawasaki ‚  A. Neuroretinitis: review of the literature and new observations. J Neuroophthalmol.  2011;31(1):58 " “68.44 Chi ‚  SL, Stinnett ‚  S, Eggenberger ‚  E, et al. Clinical characteristics in 53 patients with cat scratch optic neuropathy. Ophthalmology.  2012;119(1):183 " “187.55 Narayan ‚  SK, Kaliaperumal ‚  S, Srinivasan ‚  R. Neuroretinitis, a great mimicker. Ann Indian Acad Neurol.  2008;11(2):109 " “113.66 Casson ‚  RJ, O 'Day ‚  J, Crompton ‚  JL. Leber 's stellate neuroretinitis: differential diagnosis and approach to management. Aust N Z J Ophthalmol.  1999;27(1):65 " “69.

ADDITIONAL READING


Spencer ‚  BRJr, Digre ‚  KB. Treatments for neuro-ophthalmologic conditions. Neurol Clin.  2010;28(4):1005 " “1035. ‚  

CODES


ICD10


  • H30.90 Unspecified chorioretinal inflammation, unspecified eye
  • H30.009 Unsp focal chorioretinal inflammation, unspecified eye
  • H30.899 Other chorioretinal inflammations, unspecified eye
  • H30.019 Focal chorioretinal inflammation, juxtapapillary, unsp eye
  • H35.069 Retinal vasculitis, unspecified eye
  • H30.92 Unspecified chorioretinal inflammation, left eye
  • H35.061 Retinal vasculitis, right eye
  • H35.063 Retinal vasculitis, bilateral
  • H30.91 Unspecified chorioretinal inflammation, right eye
  • H35.062 Retinal vasculitis, left eye
  • H30.001 Unspecified focal chorioretinal inflammation, right eye
  • H30.893 Other chorioretinal inflammations, bilateral
  • H30.93 Unspecified chorioretinal inflammation, bilateral
  • H30.002 Unspecified focal chorioretinal inflammation, left eye
  • H30.003 Unspecified focal chorioretinal inflammation, bilateral
  • H30.011 Focal chorioretinal inflammation, juxtapapillary, right eye
  • H30.012 Focal chorioretinal inflammation, juxtapapillary, left eye
  • H30.013 Focal chorioretinal inflammation, juxtapapillary, bilateral
  • H30.891 Other chorioretinal inflammations, right eye
  • H30.892 Other chorioretinal inflammations, left eye

ICD9


  • 363.05 Focal retinitis and retinochoroiditis, juxtapapillary
  • 363.00 Focal chorioretinitis, unspecified
  • 362.12 Exudative retinopathy
  • 362.18 Retinal vasculitis

SNOMED


  • neuroretinitis (disorder)
  • Focal retinitis (disorder)
  • Optic neuroretinitis
  • retinal vasculitis (disorder)
  • Juxtapapillary focal retinitis AND retinochoroiditis
  • Bartonella henselae neuroretinitis

CLINICAL PEARLS


  • For a patient with painless unilateral vision loss after a viral-like prodrome, consider neuroretinitis and refer immediately to an ophthalmologist for further evaluation including a complete eye exam with dilated funduscopy.
  • Many of the underlying etiologies can present with various eye manifestations in addition to neuroretinitis, including anterior uveitis, retinitis, chorioretinitis, posterior uveitis, optic neuritis, and endophthalmitis.
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