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Methemoglobinemia, Emergency Medicine


Basics


Description


  • Iron molecule in hemoglobin is oxidized from ferrous (Fe2+) to ferric (Fe3+) state resulting in a form of hemoglobin that cannot transport oxygen.
  • Oxygen-carrying capacity of blood is reduced and cyanosis is generally present with significant levels.
  • Normal methemoglobin levels are ≤1%; symptoms usually occur with levels >20%.
  • More serious with coexisting anemia
  • Methemoglobin:
    • Decreases total oxygen-carrying capacity (functional anemia)
    • Shifts hemoglobin oxygen-dissociation curve to the left, impairing O2 release to tissues
    • Maintained at physiologic level (1 " “2%) by nicotinamide adenine dinucleotide (NADH)-methemoglobin (cytochrome B5) reductase in red blood cells (RBCs)
  • Congenital methemoglobinemia:
    • NADH-methemoglobin (cytochrome B5) reductase deficiency (homozygous or heterozygous)
    • Heterozygous hemoglobin M and other abnormal hemoglobins
  • Acquired methemoglobinemia results from oxidant stress on RBCs:
    • Some methemoglobin-inducing agents are direct oxidants (e.g., nitrites)
    • Many substances produce oxidant injury via N-hydroxylamine metabolites.
    • Methemoglobinemia may be delayed relative to initial substance exposure.
  • Many methemoglobin-inducing agents also cause Heinz body hemolytic anemia (HA):
    • Caused by oxidant injury of RBC proteins
    • Glucose-6-phosphate dehydrogenase (G6PD) " “deficient patients have higher risk.
    • Patients with methemoglobinemia should be worked up for HA.
  • Methemoglobinemia may serve as marker for genetic abnormalities:
    • Heterozygous NADH-methemoglobin (cytochrome B5) reductase deficiency

Etiology


  • Cyanide (CN) antidote kit:
    • Induces methemoglobinemia via amyl and sodium nitrite
    • CN will preferentially complex with methemoglobin, which can then be chelated by sodium thiosulfate.
  • Nitrates/nitrites:
    • Nitrites (NO2)
    • Nitrates (NO3) (e.g., nitroglycerine, via metabolic conversion to nitrites)
    • Nitric oxide (NO)
  • Dyes:
    • Aniline dyes
    • Methylene blue (excessive)
  • Antiparasitic drugs (high potential for MetHb formation):
    • Dapsone
    • Primaquine
    • Chloroquine
  • Local anesthetics (high potential for MetHb formation):
    • Benzocaine
    • Lidocaine
    • Prilocaine
  • Analgesics:
    • Phenazopyridine (Pyridium)
    • Phenacetin
  • Antibiotics:
    • Nitrofurantoin
    • Sulfones
    • Sulfonamides
  • Others:
    • Metoclopramide
    • Naphthalene (mothballs)
    • Paraquat (herbicide)
    • Arsine gas (AsH3)
    • Chlorates (ClO4)
    • Phenols (e.g., dinitrophenol, hydroquinone)

Diagnosis


Signs and Symptoms


  • Central cyanosis, refractory to oxygen administration:
    • Cyanosis evident at methemoglobin (MetHb) of 10 " “15% of total hemoglobin in nonanemic patient (or 1.5 g of MetHb/dL blood)
  • Dyspnea/tachypnea
  • Chest pain/dysrhythmias
  • Syncope
  • Altered mental status with levels >50%

History
  • Exposure to methemoglobin-inducing agent
  • All substances ingested and time(s) of ingestion
  • G6PD deficiency
  • Medical conditions vulnerable to impaired oxygen delivery (e.g., coronary artery disease)

Physical Exam
  • Cyanosis
  • Emphasis on mental status and cardiovascular findings
  • Icterus or dark-colored urine with accompanying HA

Essential Workup


  • Pulse oximetry is inaccurate in methemoglobinemia:
    • MetHb interferes with pulse oximetry measurement of hemoglobin oxygen saturation.
    • Saturation decreases to ¢ ˆ ¼85% with increasingly more severe methemoglobinemia.
    • Pulse oximetry cannot be used to guide management.
  • ABG for:
    • Methemoglobin level
    • Carboxyhemoglobin level
    • PaO2 and PaCO2
  • ECG

Diagnosis Tests & Interpretation


Lab
  • Blood classically described as chocolate colored
  • CBC with manual differential count and smear analysis for evidence of HA

Imaging
CXR to rule out other pulmonary pathology ‚  

Differential Diagnosis


  • Hypoxia:
    • CHF
    • COPD
    • Pulmonary embolism
  • Irritant gas exposure
  • Blue discoloration:
    • Hypoxia
    • Sulfhemoglobinemia
    • CN poisoning
    • Hydrogen sulfide poisoning
    • Excess methylene blue administration
    • Tellurium toxicity
    • Skin contact/staining with blue dye

Treatment


Pre-Hospital


  • Bring to hospital all substances patient may have ingested.
  • Question witnesses and observe scene for household products and other potential coingestants:
    • Document and relay findings to emergency medical staff.
  • Commercial or industrial sites:
    • Obtain relevant material safety data sheets (MSDSs) if available to identify commercial or chemical products.
    • Avoid dermal exposures.

Initial Stabilization/Therapy


  • ABCs:
    • Cardiac monitor
    • Isotonic crystalloids as needed for hypotension
  • Naloxone, thiamine, and dextrose (D50W) as indicated for altered mental status
  • Supplemental oxygen

Ed Treatment/Procedures


  • Decontamination:
    • If owing to acute ingestion/overdose within previous 1 " “2 hr, and protective airway reflexes are intact, administer 50 " “100 g of activated charcoal PO.
  • Remove source of oxidant stress.
  • Methylene blue:
    • Indications:
      • Asymptomatic with levels >30%
      • Symptomatic patients with levels >10 " “20%, especially if comorbid diseases are present
    • Expect transient worsening of saturations on pulse oximetry after methylene blue is administered:
      • Interferes with pulse oximetry measurement and no specific intervention required
    • Use with caution in patients with glucose-6 pyruvate decarboxylase deficiency:
      • May cause hemolysis
  • If no improvement with methylene blue, consider that source of oxidant stress is not eliminated, or that sulfhemoglobinemia is present:
    • Sulfhemoglobin is sulfur molecule bound to hemoglobin. Presents similar to methemoglobin, but is self-limited and not responsive to methylene blue.
  • RBC transfusion:
    • May be necessary to increase blood oxygen-carrying capacity
    • Consider in the presence of HA.
  • Exchange transfusion:
    • Especially with neonates/infants
  • Hyperbaric oxygen therapy:
    • Increases oxygen delivery to tissues by allowing more oxygen to be dissolved in the blood, independent of hemoglobin.
    • Use in life-threatening methemoglobinemia if immediately available.

  • Children may develop significant methemoglobinemia from apparently minor ingestions.
  • Symptoms delayed several hours after ingestion, so prolonged observation necessary
  • Neonates are also at higher risk of methemoglobinemia (owing to decreased stores of NADH methemoglobin reductase).

Medication


  • Dextrose 50%: 25 g (50 mL) (peds: 0.5 " “1 g/kg of dextrose) IV for hypoglycemia
  • Methylene blue: 0.1 " “0.2 mL/kg 1% solution IV over 5 min (adults and peds)
    • May repeat if no improvement in 1 hr
    • Doses of 0.3 to 1 mg/kg IV have been effective in neonates. Has been used IO over 3 " “5 min.
  • Naloxone: 0.4 " “2 mg (peds: 0.1 mg/kg) IV, may repeat up to 10 mg for suspected opioid intoxication
  • Thiamine: 100 mg (peds: 1 mg/kg) IM or IV

Follow-Up


Disposition


Admission Criteria
  • Severely symptomatic patients
  • Patients requiring multiple doses of methylene blue
  • Dapsone may cause prolonged recurrent methemoglobinemia

Discharge Criteria
Methemoglobin levels <20% and falling with no symptoms or comorbid disease ‚  
Issues for Referral
Toxicology consult for significant exposures ‚  

Followup Recommendations


Occupational medicine referral for work-related exposures ‚  

Pearls and Pitfalls


  • Pulse oximetry is inaccurate in methemoglobinemia.
  • Obtain an ABG.
  • Administer methylene blue for significant levels/symptoms.

A special thanks to Dr. Gerald Maloney who contributed to the previous edition. ‚  

Additional Reading


  • Barclay ‚  JA, Ziemba ‚  SE, Ibrahim ‚  RB. Dapsone-induced methemoglobinemia: A primer for clinicians. Ann Pharmacother.  2011;45:1103 " “1115.
  • Bradberry ‚  SM, Aw ‚  TC, Williams ‚  NR, et al. Occupational methaemoglobinaemia. Occup Environ Med.  2001;58:611 " “615.
  • Guay ‚  J. Methemoglobinemia related to local anesthetics: A summary of 242 episodes. Anesth Analg.  2009;108:837 " “845.
  • Price ‚  D. Methemoglobin inducers. In: Flomenbaum ‚  NE, Goldfrank ‚  LR, Hoffman ‚  RS, et al., eds. Goldfranks Toxicologic Emergencies. 9th ed. New York, NY: McGraw-Hill; 2011.
  • Wright ‚  RO, Lewander ‚  WJ, Woolf ‚  AD. Methemoglobinemia: Etiology, pharmacology, and clinical management. Ann Emerg Med.  1999;34:646 " “656.

Codes


ICD9


289.7 Methemoglobinemia ‚  

ICD10


  • D74.0 Congenital methemoglobinemia
  • D74.8 Other methemoglobinemias
  • D74.9 Methemoglobinemia, unspecified
  • D74 Methemoglobinemia

SNOMED


  • 38959009 Methemoglobinemia (disorder)
  • 267550008 Congenital methemoglobinemia (disorder)
  • 295315008 Acquired methemoglobinemia
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