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Meningococcemia, Pediatric


Basics


Description


  • A systemic infection with the bacterium Neisseria meningitidis, a gram-negative diplococcus that is relatively fastidious. Despite treatment with appropriate antibiotics, this disease may have a fulminant course (i.e., significant complications within hours of presentation) with a high likelihood of mortality.
  • 13 serogroups have been described on the basis of capsular polysaccharide antigens; serotypes B, C, and Y account for most of the cases in the United States. Serogroup Y accounted for 30% of cases between 1996 and 1998.

General Prevention


  • Isolation of the hospitalized patient; hospitalized patients require respiratory isolation until 24 hours after initiation of appropriate antibiotic therapy.
  • Exposed contacts, including household, day care, and nursery school, should receive the following:
    • Rifampin, 10 mg/kg (maximum 600 mg) PO q12h for 4 doses
    • Contacts <1 month of age should receive rifampin 5 mg/kg PO q12h for 4 doses.
    • Alternatively, ceftriaxone is also effective prophylaxis for contacts ≤15 years of age; a single dose of 125 mg IM is recommended.
    • For contacts >15 years old, ceftriaxone 250 mg IM is recommended. Its safety profile is preferred for pregnant women.
  • Medical personnel should receive prophylaxis only if they had close contact with respiratory secretions.
  • Vaccines for types A, C, Y, and W-135 are available and produce an immune response in 10 " “14 days.
  • A tetravalent conjugate meningococcal vaccine, MCV4, is licensed for use in people in the age range of 2 " “55 years. It is recommended in all unimmunized 11 " “12-year-old adolescents, with a booster dose at age 16 years.
  • Serotype B vaccine was recently approved by the FDA and is licensed for use in people 10 " “25 years of age.
  • The Centers for Disease Control and Prevention (CDC) continues to recommend routine adolescent immunization with the exception of persons with a history of Guillain-Barre syndrome (GBS) who are not in a high-risk group for invasive meningococcal disease. An updated fact sheet on GBS and MCV4 is available at http://www.cdc.gov/vaccinesafety/Concerns/gbsfactsheet.html. A study published in 2012 did not support an association between GBS and MCV4 vaccination.

Epidemiology


  • The rates of meningococcal disease in the United States have remained stable at 0.9 " “1.5 cases per 100,000 population per year.
  • Children <5 years of age are most often affected, with peak incidence between 3 and 5 months of age.
  • During epidemics, more school-aged children may be affected.
  • The disease occurs most commonly in winter and spring months.
  • Increased disease activity may follow an influenza A outbreak.

Risk Factors


  • Patients with asplenia, deficiencies of properdin C3, or a terminal complement component (C5 " “C9), and HIV are at increased risk for invasive and recurrent disease.
  • Organism virulence factors, such as differences in the bacterial cell wall lipopolysaccharide, play a role in disease severity. Less virulent organisms are more likely in chronic meningococcemia, which has a favorable prognosis.

Genetics
  • Inherited deficiency of terminal complement may be found in 5 " “10% of patients during epidemics. The frequency increases to 30% in patients with recurrent disease.
  • A number of other immune function " “related genes associated with either susceptibility or protection from infection have been identified.

Pathophysiology


  • Fulminant disease is signified by diffuse microvascular damage and disseminated intravascular coagulation (DIC); see "Diagnosis " ť section.
  • Death results from effects of endotoxic shock, including circulatory collapse and myocardial dysfunction.

Etiology


  • Colonization and infection of the upper respiratory tract occurs after inhalation of, or direct contact with, the organism, usually in oral secretions.
  • Disseminated disease occurs when the organism penetrates the nasal mucosa and enters the bloodstream, where it replicates.

Diagnosis


Signs and Symptoms


  • Fever
  • Malaise
  • Rash
  • Petechiae
  • Tachycardia
  • Delayed capillary fill
  • Abnormal mental status
  • Bacteremia without sepsis presents with fever, malaise, myalgias, and headache. Patients may clear the infection spontaneously or it may invade meninges, joints, lungs, and so forth.
  • Meningococcemia without meningitis occurs after initial bacteremia with systemic sepsis. A rash erupts, which may be nonspecific maculopapular, morbilliform, or urticarial. Progression to petechiae or purpura signifies evolution of disease.
  • Fulminant disease can manifest within 1 " “2 hours of onset of signs or symptoms and is signified by hypotension, oliguria, DIC, myocardial dysfunction, and vascular collapse. Death occurs in 15 " “20% of these patients.

History


Time of onset of fever, malaise, and rash ‚  

Physical Exam


  • Physical examination of a child with fever should include careful evaluation of the skin for petechiae and signs of early shock (tachycardia, delayed capillary refill, abnormal mental status, etc.).
  • Recognition of abnormal vital signs and lethargy is necessary.
  • Nuchal rigidity, lethargy, and irritability should be carefully but expeditiously evaluated.

Diagnostic Tests & Interpretation


The organism can be cultured from blood, CSF, and skin lesions. ‚  
Lab
  • Gram stain of CSF or scraped petechial lesion (pressed against a glass slide) revealing gram-negative diplococci will give a presumptive diagnosis.
  • Rapid test for antigen detection
    • Supports diagnosis if found in CSF but not sensitive for serogroup B
  • CBC
    • One study showed that 94% of children show abnormalities in 1 or more of the following parameters: abnormalities in absolute neutrophil count ( ≤1,000/mm3 or ≥10,000/mm3), immature neutrophil count ( ≥500/mm3), and/or immature-to-total neutrophil ratio ( ≥0.20).

Diagnostic Procedures/Other
Lumbar puncture: antigen detection although culture remains the gold standard ‚  

Differential Diagnosis


  • Meningitis due to N. meningitidis is indistinguishable from that of other causes, except for 1/3 of children who have a petechial rash.
  • Sepsis from other microbial causes (e.g., Streptococcus, Rocky Mountain spotted fever, viruses) may have a very similar clinical presentation, including the petechiae or purpuric rash.

Treatment


General Measures


  • Because of the rapidly progressing nature of meningococcemia in some, patients with acute onset of petechial rash and fever should receive a prompt initial dose of antibiotics (if possible and practical, after blood culture or lumbar puncture).
  • Close monitoring of vital signs and clinical status should follow, preferably in an ICU setting.

Medication


  • Cefotaxime or ceftriaxone can be initiated as presumptive therapy. After sensitivity is confirmed, penicillin is preferred.
  • After isolate is proven sensitive to penicillin, treatment of choice is aqueous penicillin G IV at a dose of 300,000 IU/kg/24 h q4 " “6h (max, 12 million U/24 h) for 5 " “7 days.
  • In penicillin-allergic patients, 3rd-generation cephalosporins, or chloramphenicol are acceptable alternatives.

Issues for Referral


Public health officials should be notified of N. meningitidis cases. ‚  

Ongoing Care


Follow-up Recommendations


Patient Monitoring
Patients with bacterial meningitis should have a hearing test as a follow-up. ‚  

Prognosis


  • Fatality rate of meningococcemia is 15 " “20%, even when recognized and treated.
  • Fatality rate of meningococcal meningitis is 5%. The most severe cases often have a rapid progression from onset of symptoms to death over a matter of hours. At the time of hospital admission, the following signs predict poor survival:
    • Lack of meningitis
    • Shock
    • Coma
    • Purpura
    • Neutropenia
    • Thrombocytopenia
    • DIC
    • Myocarditis

Complications


  • Complications may result directly from the infection or be classified as allergic immune complex mediated.
  • Meningococcemia may be complicated by myocarditis, arthritis, hemorrhage, and pneumonia; digit or limb amputation, and skin scarring.
  • Meningococcal meningitis is most commonly complicated by deafness in 5 " “10% of survivors.
  • Other complications of meningitis include seizures, subdural effusions, and cranial nerve palsies.
  • Immunologic complications include arthritis, vasculitis, pericarditis, and episcleritis.

Additional Reading


  • Brouwer ‚  MC, Spanjaard ‚  L, Prins ‚  JM, et al. Association of chronic meningococcemia with infection by meningococci with underacylated lipopolysaccharide. J Infect.  2011;62(6):479 " “483.
  • Centers for Disease Control and Prevention. Update: Guillain-Barre syndrome among recipients of Menactra meningococcal conjugate vaccine " ”United States, June 2005 " “September 2006. MMWR Morb Mortal Wkly Rep.  2006;55(43):1120 " “1124.
  • Demissie ‚  DE, Kaplan ‚  SL, Romero ‚  JR, et al. Altered neutrophil counts at diagnosis of invasive meningococcal infection in children. Pediatr Infect Dis J.  2013;32(10):1070 " “1072. ‚  [View Abstract]
  • Pathan ‚  N, Faust ‚  SN, Levin ‚  M. Pathophysiology of meningococcal meningitis and septicaemia. Arch Dis Child.  2003;88(7):601 " “607. ‚  [View Abstract]
  • Rosenstein ‚  NE, Perkins ‚  BA, Stephens ‚  DS, et al. Medical progress: meningococcal disease. N Engl J Med.  2001;344(18):1378 " “1388. ‚  [View Abstract]
  • Velentgas ‚  P, Amato ‚  AA, Bohn ‚  RL, et al. Risk of Guillain-Barre syndrome after meningococcal conjugate vaccination. Pharmacoepidemiol Drug Saf.  2012;21(12):1350 " “1358. ‚  [View Abstract]
  • Welch ‚  SB, Nadel ‚  S. Treatment of meningococcal infection. Arch Dis Child.  2003;88(7):608 " “614. ‚  [View Abstract]

Codes


ICD09


  • 036.2 Meningococcemia

ICD10


  • A39.4 Meningococcemia, unspecified
  • A39.3 Chronic meningococcemia
  • A39.2 Acute meningococcemia

SNOMED


  • 4089001 Meningococcemia (disorder)
  • 240426001 chronic meningococcemia (disorder)
  • 186365005 Acute meningococcemia

FAQ


  • Q: How long should antibiotic therapy be given to a patient with septic shock?
  • A: 7 days
  • Q: Is MCV4 meningococcal vaccine indicated for all adolescents?
  • A: Yes, MCV4 is now recommended in all previously unimmunized adolescents at the doctor visit from 11 to 12 years or at high school entry, whichever comes first. A booster dose is recommended at age 16 years.
  • Q: How does 1 approach MCV4 immunization of adolescents who previously received MPSV4?
  • A: If 3 " “5 years have elapsed since their MPSV4 vaccination, then MCV4 immunization is indicated.
  • Q: When should 1 test for complement deficiency?
  • A: In patients with recurrent disease
  • Q: Which hospital personnel should receive prophylaxis?
  • A: Only those with direct exposure to index patient 's secretions.
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