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Ludwig Angina, Emergency Medicine


Basics


Description


  • Named for German physician Wilhelm Friedrich von Ludwig, who 1st described this in 1836 as a rapidly progressive, gangrenous cellulitis and edema of soft tissues of the neck, floor of the mouth
  • Gangrene is serosanguineous infiltration with little or no frank pus or primary abscesses
    • Contiguous spread may encircle the airway or involve the mediastinum
    • Emergent interventions rarely include surgical or aspiration techniques
  • Most deaths are due to airway compromise, occlusion, and resultant asphyxia
    • Mortality exceeded 50% in preantibiotic era, currently <8%

Etiology


  • Odontogenic in 90% of adult cases, usually from 2nd, 3rd mandibular molars
  • Less commonly: Mandibular fractures, oral lacerations, contiguous infections, errant drug injections, tongue piercings
  • Polymicrobial: Ž ²-hemolytic strep commonly associated with anaerobes such as peptostreptococcus, pigmented bacteroides
    • Microbiologic analyses may guide therapy

  • Frequently no clear etiology or site of origin
  • Ideally, a destination facility will have specialty expertise available (surgery and subspecialties, anesthesia) and be properly equipped to provide emergent intervention

Diagnosis


Signs and Symptoms


History
  • Nonspecific constitutional symptoms: Fever, malaise, anxiety
  • Pain: Tongue, throat, jaw, chest, neck stiffness
  • Dysphagia, dysphonia
  • Dentition, dental care suboptimal

Physical Exam
  • Febrile, toxic, tripod "sniffing "  posture
  • Stridor, "hot potato "  voice
  • HEENT:
    • Tongue progressively displaced upward in both posterior, anterior directions at unpredictable rate
    • Airway increasingly compromised
    • Drooling, salivary incontinence
    • Trismus impedes diagnosis and complicates treatment measures
  • Physical exam findings beyond those of the head and neck area are often noncontributory or unrelated

Essential Workup


  • The diagnosis is usually clinically evident
    • No study or procedure needed to confirm the diagnosis
  • Loss of airway patency can be unexpected, precipitous, and calamitous
  • Securing airway patency and initiating treatment take precedent over workup considerations

Diagnosis Tests & Interpretation


Lab
No test will establish the diagnosis; assess severity or direct therapy ‚  
Imaging
Contrast-enhanced CT: ‚  
  • CT of the neck with IV contrast enhancement is the study of choice:
    • Standard cross-sectional imaging extends from skull base to aortic arch
    • Best for evaluating the mediastinum, deep space infection location and extent, degree of airway involvement.
    • Findings include streaky or "dirty "  fat in areas of inflammation; adenopathy (submandibular, submental, anterior and posterior cervical chains); perhaps pus or gas formation
    • Potential limitations: Patient must remain supine for the study duration. Scanning location often away from optimal resuscitation, intervention capability.

Plain Radiographs: ‚  
  • Soft tissue lateral neck x-ray may demonstrate altered anatomy, especially in the upper airway
  • Chest x-ray of little utility, including detecting presence and extent of mediastinal involvement
  • Panorex may detect odontogenic or mandibular pathology, but of no use imaging soft tissue

Contrast-enhanced MRI: ‚  
  • Information obtained is the same, of no greater value than contrast-enhanced CT:
    • Potential limitations: patient must remain supine, motionless for the study duration. Scanning location often away from optimal resuscitation, intervention capability.

Ultrasound: ‚  
  • Detects gas in tissues, abscesses, reactive lymphadenopathy
  • May locate, outline the airway amongst edematous, distorted tissues of the anterior neck
  • A guide for abscess or fluid aspiration

Diagnostic Procedures/Surgery
No surgery or invasive procedure will establish the diagnosis, assess severity, or direct therapy ‚  

Differential Diagnosis


  • Infectious: Cellulitis, epiglottitis, tracheitis, peritonsillar abscess
  • Traumatic: Penetrating injury, sublingual hematoma from fracture, soft tissue injury
  • Angioneurotic edema
  • Neoplasia

Treatment


Pre-Hospital


  • Transport in position of comfort
    • Allow adult tripod "sniffing "  position, to suction themselves
    • Allow pediatric transport, simple interventions (blow-by O2, nebulizer treatments) on mothers lap
  • Maximize oxygenation:
    • FIO2 of 100%
    • Consider concurrent O2 delivery systems, such as facemask and nasal cannula
  • Jet insufflation: An infrequently used temporizing rescue device for oxygenation
    • Potential limitations: Few experienced with device assembly or use.
    • Newer rescue devices easier to place and use.

  • Minimize patient upset, agitation
    • Allow transport, simple interventions (blow-by O2, nebulizer treatments) in parent's embrace.
    • Question the necessity for any interventions: IV access, blood draws, O2 mask, monitor leads.
    • Transport to facility best able to care for this complex patient if possible.

Initial Stabilization/Therapy


Airway Measures ‚  
  • Maximize oxygenation
  • Maintain in position of comfort
  • Gather supplies/personnel for back-up airway techniques
  • See "Airway Management "  below.

Vascular Access
  • Vascular access: Provides rapid, titratable, predictable medication delivery
    • Intraosseus (IO) access useful with poor peripheral access, resuscitations, pediatric access, adverse prehospital conditions
    • Commercially available device provides IO access rapidly, effectively
  • 2nd access recommended: Rescue backup, concurrent polypharmacy.

Ed Treatment/Procedures


  • Immediate priorities are to secure the airway and to institute medical treatment. Diminishing consensus on need for acute surgical intervention other than airway related.
  • Infrequently see treatable abscess formation, fluid collections on initial presentation.

Airway management: ‚  
  • Rescue airway devices may be difficult to place, altered effectiveness due to anatomy distortion, trismus, excessive secretions
  • Avoid blind intubation techniques to reduce laryngospasm, iatrogenic injury, bleeding, further tissue distortion
  • Equipment considerations:
    • Smaller ET tubes
    • Prelubricate with gel or viscous lidocaine
    • Use stylet or bougie for tube support
    • Bend distal tube into "hockey stick "  shape
  • Rapid-sequence intubation (RSI) agents may cause abrupt loss of muscle tone, airway architecture, or precipitate airway compromise
  • Concern for impending respiratory failure increases with stridor, voice change, trismus, tripod posture, sialorrhea

Definitive management: ‚  
  • Traditional surgical gold standard: Tracheostomy using local anesthesia:
    • Potential difficulties: Surgeon, specialist availability, facility capabilities not uniform
  • Traditional nonsurgical gold standard intubation using fiberoptic guidance:
    • Potential difficulties: Fiberoptic scopes expensive, fragile, require specific cleaning regimens. Short scopes often lack suction or irrigation ports, visualization easily impaired. Their use is not intuitive to the infrequent operator
  • Best management option "double setup " 
    • Patient in an operating theater equipped, prepared to establish surgical airway
    • Nonsurgical intervention attempted
    • Immediate surgical intervention if unsuccessful or clinical deterioration
  • Intubation: Anticipate distorted anatomy:
    • Sitting, awake a preferred option
    • Sequential topical applications

Medication


  • IV administration: Preferred route of administration as previously outlined
  • IO considerations:
    • Lidocaine flush reduces infusion pain
    • Flow rates same as IV for routine fluids, medication administration
    • Avoid hyperosmolar agents, potential marrow injury
  • Antibiotics: Empiric use of broad-spectrum antibiotics justifiable, for use until return of culture and antibiogram results, which should direct further therapy:
    • Ampicillin/sulbactam: 1.5 " “3 g IM/IV q6h (peds: 300 mg/kg/d div. q6 if <1yr, <40 kg; 1.5 " “3 g IV q6h if >1 yr, >40 kg); max. 12 g/d
    • Cefoxitin: 1 " “2 g IV q6 " “8h (peds: 80 " “160 mg/kg/d div. q4 " “6h); max. 12g/d
    • Clindamycin: 600 " “900 mg IM/IV q8h (peds: 15 " “25 mg/kg/d div. q6 " “8h)
    • Piperacillin/tazobactam: 3.375 g IV q6h (peds: If >9 mo, <40 kg; 300 mg/kg/d IV div. q8h)
    • Ticarcillin/clavulanate: 3.1 g IV q4 " “6h (peds: If >3 mo, <60 kg; 200 " “300 mg/kg/d div. q4 " “6h)
  • Analgesia: Pain control should be a primary concern
  • Antiemetics: Proactive, prophylactic use for medication-related or condition-induced symptoms
  • Steroids: Recommend empiric use of longer acting steroids to reduce:
    • swelling
    • inflammation
    • systemic stress dose replenishment
  • Hyperbaric oxygen: Consider if mediastinitis or necrotizing fasciitis

Follow-Up


Disposition


Admission Criteria
  • All are admitted:
    • Airway encroachment and obstruction can be progressive and unpredictable
  • ICU or closely monitored setting due to unpredictable progression of symptoms

Issues for Referral
  • This is a clinical diagnosis with unpredictable progression:
    • Early specialty consultation is necessary for possible assistance with airway management or drainage
  • Early transfer to higher level of care if the illness acuity exceeds the clinicians level of expertise or if the facility is not adequately equipped for such management

  • Mother is susceptible to all aspects and complications as nongravid patients
  • Focus: Airway management, oxygenation, treatment of sepsis if present

Chronic comorbid conditions, chronic medications, less physiologic reserve can all complicate the presentation and treatment ‚  

Complications


  • Asphyxia
  • Spread into thoracic cavity:
    • Empyema
    • Mediastinitis
    • Lung abscess
  • Pericarditis
  • Internal jugular vein thrombosis
  • Carotid artery erosion and/or infection
  • Sepsis/bacteremia
  • Subphrenic abscess

Pearls and Pitfalls


Pearls: ‚  
  • Prepare to manage airway immediately
  • Consult appropriate medical specialists as soon as possible, whether for transfer to a higher level of care, or to the operating suite for "double setup "  management
  • Video laryngoscopy is intuitive and easy to use, provides rapid, safe, high probability intubation success

Pitfalls: ‚  
  • Failure to appreciate the progressive nature, unpredictable rate, extent of advancement
  • Diagnostic testing and/or imaging should not delay definitive airway management or other therapy

Additional Reading


  • Candamourty ‚  R, Venkatachalam ‚  S, Babu ‚  MR, et al. Ludwigs Angina " ”An emergency: A case report with literature review. J Nat Sci Biol Med.  2012;3(2):206 " “208.
  • Ludwig ‚  BJ, Foster ‚  BR, Saito ‚  N, et al. Diagnostic imaging in nontraumatic pediatric head and neck emergencies. Radiographics.  2010;30(3):781 " “799.
  • Mckellop ‚  JA, Bou-Assaly ‚  W, Mukherji ‚  SK. Emergency head & neck imaging: Infection and inflammatory processes. Neuroimaging Clin N Am.  2010;20:651 " “661.
  • Tobias ‚  JD, Ross ‚  AK. Intraosseous infusions: A review for the anesthesiologist with a focus on pediatric use. Anesth Analg.  2010;110:391 " “401
  • Weingart ‚  SD, Levitan ‚  RM. Preoxygenation and prevention of desaturation during emergency airway management. Ann Emerg Med.  2012;59:165 " “175.e1.
  • Wolfe ‚  MM, Davis ‚  JW, Parks ‚  SN. Is surgical airway necessary for airway management in deep neck infections and Ludwig angina? J Crit Care.  2011:26:11 " “14.

Codes


ICD9


528.3 Cellulitis and abscess of oral soft tissues ‚  

ICD10


K12.2 Cellulitis and abscess of mouth ‚  

SNOMED


  • 196542004 Ludwigs angina (disorder)
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